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PI3Kδ inhibition reduces TNF secretion and neuroinflammation in a mouse cerebral stroke model.The Complement Receptor C5aR Controls Acute Inflammation and Astrogliosis following Spinal Cord Injury.Targeting acute inflammation to complement spinal cord repair.The receptor for complement component C3a mediates protection from intestinal ischemia-reperfusion injuries by inhibiting neutrophil mobilization.IVIg attenuates complement and improves spinal cord injury outcomes in mice.Complement activation in the injured central nervous system: another dual-edged sword?Therapeutic targeting of complement to modify disease course and improve outcomes in neurological conditions.Longitudinal assessment of white matter pathology in the injured mouse spinal cord through ultra-high field (16.4 T) in vivo diffusion tensor imaging.Mobilisation of the splenic monocyte reservoir and peripheral CX₃CR1 deficiency adversely affects recovery from spinal cord injury.The Alternative Receptor for Complement Component 5a, C5aR2, Conveys Neuroprotection in Traumatic Spinal Cord Injury.
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description
researcher, ORCID id # 0000-0002-9201-2476
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wetenschapper
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name
Faith H Brennan
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Faith H Brennan
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Faith H Brennan
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type
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Faith H Brennan
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Faith H Brennan
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Faith H Brennan
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prefLabel
Faith H Brennan
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Faith H Brennan
@en
Faith H Brennan
@nl
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0000-0002-9201-2476