Midkine is regulated by hypoxia and causes pulmonary vascular remodeling. - Wikidata - wikimedia - TriplyDB

Midkine is regulated by hypoxia and causes pulmonary vascular remodeling.

Midkine is regulated by hypoxia and causes pulmonary vascular remodeling.

http://www.wikidata.org/entity/Q40545303

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Serum response factor and myocardin mediate arterial hypercontractility and cerebral blood flow dysregulation in Alzheimer's phenotype.Midkine and the kidney: health and diseases Midkine in nephrogenesis, hypertension and kidney diseases Midkine in host defence Structure and function of midkine as the basis of its pharmacological effects Hypoxia-inducible factor 1 and cardiovascular disease Therapeutic vulnerability of an in vivo model of alveolar soft part sarcoma (ASPS) to antiangiogenic therapy The transcription factor gene Nfib is essential for both lung maturation and brain development The forkhead box m1 transcription factor is essential for embryonic development of pulmonary vasculature Diesel particulate matter induces receptor for advanced glycation end-products (RAGE) expression in pulmonary epithelial cells, and RAGE signaling influences NF-κB-mediated inflammation Correlating global gene regulation to angiogenesis in the developing chick extra-embryonic vascular system.Midkine is a NF-kappaB-inducible gene that supports prostate cancer cell survival.Glucocorticoid regulation of lung development: lessons learned from conditional GR knockout mice Neurovascular mechanisms and blood-brain barrier disorder in Alzheimer's disease.RNA-Seq quantification of the human small airway epithelium transcriptome Antenatal exposure of maternal secondhand smoke (SHS) increases fetal lung expression of RAGE and induces RAGE-mediated pulmonary inflammation.TTF-1 regulates α5 nicotinic acetylcholine receptor (nAChR) subunits in proximal and distal lung epithelium.Midkine is expressed and differentially processed during chronic obstructive pulmonary disease exacerbations and ventilator-associated pneumonia associated with Staphylococcus aureus infection.Midkine: a promising molecule for drug development to treat diseases of the central nervous system Genomic profile of matrix and vasculature remodeling in TGF-alpha induced pulmonary fibrosis Control mechanisms of lung alveolar development and their disorders in bronchopulmonary dysplasia.Midkine, a heparin-binding cytokine with multiple roles in development, repair and diseases.Neurovascular dysfunction and faulty amyloid β-peptide clearance in Alzheimer disease.Follistatin like-1 (Fstl1) is required for the normal formation of lung airway and vascular smooth muscle at birth.Smooth muscle alpha-actin expression and myofibroblast differentiation by TGFbeta are dependent upon MK2.Embryological origin of airway smooth muscle Midkine: A Novel Biomarker to Predict Malignancy in Patients with Nodular Thyroid Disease.Modulation of pulmonary vascular smooth muscle cell phenotype in hypoxia: role of cGMP-dependent protein kinase and myocardin Midkine: a novel prognostic biomarker for cancer.The angiogenic factor midkine is regulated by dexamethasone and retinoic acid during alveolarization and in alveolar epithelial cells.Therapeutic potential of midkine in cardiovascular disease.PKCδ/midkine pathway drives hypoxia-induced proliferation and differentiation of human lung epithelial cells.Regulation of CLL survival by hypoxia-inducible factor and its target genes.Lessons from the heart and ischemic limbs: midkine as anti-inflammatory mediator for kidney diseases?Myocardin in biology and disease.Midkine, a heparin-binding growth factor, and its roles in atherogenesis and inflammatory kidney diseases.Primary alveolar macrophages exposed to diesel particulate matter increase RAGE expression and activate RAGE signaling.From top to bottom: midkine and pleiotrophin as emerging players in immune regulation.Up-Regulation of Claudin-6 in the Distal Lung Impacts Secondhand Smoke-Induced Inflammation Receptor for advanced glycation end-products signals through Ras during tobacco smoke-induced pulmonary inflammation.

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P2860

Midkine is regulated by hypoxia and causes pulmonary vascular remodeling.

http://www.wikidata.org/entity/Q40545303

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2004 nî lūn-bûn

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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2004年论文

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2004年论文

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name

Midkine is regulated by hypoxia and causes pulmonary vascular remodeling.

@en

type

label

Midkine is regulated by hypoxia and causes pulmonary vascular remodeling.

@en

prefLabel

Midkine is regulated by hypoxia and causes pulmonary vascular remodeling.

@en

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Journal of Biological Chemistry

P1476

Midkine is regulated by hypoxia and causes pulmonary vascular remodeling

@en

P2093

Jeffrey A Whitsett

http://www.w3.org/2001/XMLSchema#string

Michael L Mucenski

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William C Nichols

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P2860

A nuclear factor induced by hypoxia via de novo protein synthesis binds to the human erythropoietin gene enhancer at a site required for transcriptional activation

Potentiation of serum response factor activity by a family of myocardin-related transcription factors.

General involvement of hypoxia-inducible factor 1 in transcriptional response to hypoxia

Regulation of hypoxia-inducible factor 1alpha is mediated by an O2-dependent degradation domain via the ubiquitin-proteasome pathway

Myocardin is a master regulator of smooth muscle gene expression

Increased metalloproteinase activity, oxidant production, and emphysema in surfactant protein D gene-inactivated mice

Wnt/Wingless signaling through beta-catenin requires the function of both LRP/Arrow and frizzled classes of receptors

LDL receptor-related protein as a component of the midkine receptor

HIF-1, O(2), and the 3 PHDs: how animal cells signal hypoxia to the nucleus

beta-Catenin is required for specification of proximal/distal cell fate during lung morphogenesis

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37124-37132

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10.1074/JBC.M405254200

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35

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279

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Paul R Reynolds

Timothy D Le Cras

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2004-06-14T00:00:00Z

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8510766

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