Neurodegeneration and neuroinflammation in cdk5/p25-inducible mice: a model for hippocampal sclerosis and neocortical degeneration.
about
Rodent models of neuroinflammation for Alzheimer's diseaseDirect and indirect roles of cyclin-dependent kinase 5 as an upstream regulator in the c-Jun NH2-terminal kinase cascade: relevance to neurotoxic insults in Alzheimer's disease.Mice lacking phosphatase PP2A subunit PR61/B'delta (Ppp2r5d) develop spatially restricted tauopathy by deregulation of CDK5 and GSK3beta.AAV-tau mediates pyramidal neurodegeneration by cell-cycle re-entry without neurofibrillary tangle formation in wild-type mice.Molecular and immunocytochemical characterization of primary neuronal cultures from adult rat brain: Differential expression of neuronal and glial protein markersTFP5, a Peptide Inhibitor of Aberrant and Hyperactive Cdk5/p25, Attenuates Pathological Phenotypes and Restores Synaptic Function in CK-p25Tg Mice.Dendritic degeneration, neurovascular defects, and inflammation precede neuronal loss in a mouse model for tau-mediated neurodegeneration.TFP5, a peptide derived from p35, a Cdk5 neuronal activator, rescues cortical neurons from glucose toxicity.Strain background influences neurotoxicity and behavioral abnormalities in mice expressing the tetracycline transactivator.Neurological characterization of mice deficient in GSK3α highlight pleiotropic physiological functions in cognition and pathological activity as Tau kinase.Activated cyclin-dependent kinase 5 promotes microglial phagocytosis of fibrillar β-amyloid by up-regulating lipoprotein lipase expressionCrosstalk between Cdk5 and GSK3beta: Implications for Alzheimer's DiseaseLinking Alzheimer's disease to insulin resistance: the FoxO response to oxidative stress.Modulation of p25 and inflammatory pathways by fisetin maintains cognitive function in Alzheimer's disease transgenic miceModeling of tau-mediated synaptic and neuronal degeneration in Alzheimer's disease.Neurodegeneration-Like Pathological and Behavioral Changes in an AAV9-Mediated p25 Overexpression Mouse Model.Curcumin Ameliorates Neuroinflammation, Neurodegeneration, and Memory Deficits in p25 Transgenic Mouse Model that Bears Hallmarks of Alzheimer's Disease.Fisetin Reduces the Impact of Aging on Behavior and Physiology in the Rapidly Aging SAMP8 Mouse.Inhibition of cyclin-dependent kinase 5 affects early neuroinflammatory signalling in murine model of amyloid beta toxicity.Differential Neurotoxicity Related to Tetracycline Transactivator and TDP-43 Expression in Conditional TDP-43 Mouse Model of Frontotemporal Lobar Degeneration
P2860
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P2860
Neurodegeneration and neuroinflammation in cdk5/p25-inducible mice: a model for hippocampal sclerosis and neocortical degeneration.
description
2008 nî lūn-bûn
@nan
2008 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Neurodegeneration and neuroinf ...... and neocortical degeneration.
@ast
Neurodegeneration and neuroinf ...... and neocortical degeneration.
@en
Neurodegeneration and neuroinf ...... and neocortical degeneration.
@nl
type
label
Neurodegeneration and neuroinf ...... and neocortical degeneration.
@ast
Neurodegeneration and neuroinf ...... and neocortical degeneration.
@en
Neurodegeneration and neuroinf ...... and neocortical degeneration.
@nl
prefLabel
Neurodegeneration and neuroinf ...... and neocortical degeneration.
@ast
Neurodegeneration and neuroinf ...... and neocortical degeneration.
@en
Neurodegeneration and neuroinf ...... and neocortical degeneration.
@nl
P2093
P2860
P1476
Neurodegeneration and neuroinf ...... and neocortical degeneration.
@en
P2093
Anna Kremer
David Muyllaert
Dick Terwel
Herman Devijver
Kristina Sennvik
Peter Borghgraef
P2860
P304
P356
10.2353/AJPATH.2008.070693
P407
P577
2008-01-17T00:00:00Z