In vivo HIV-1 infection of CD45RA(+)CD4(+) T cells is established primarily by syncytium-inducing variants and correlates with the rate of CD4(+) T cell decline
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HIV envelope induces a cascade of cell signals in non-proliferating target cells that favor virus replicationCCR5, GPR15, and CXCR6 are major coreceptors of human immunodeficiency virus type 2 variants isolated from individuals with and without plasma viremiaDendritic Cells in HIV-1 and HCV Infection: Can They Help Win the Battle?A novel CCR5 mutation common in sooty mangabeys reveals SIVsmm infection of CCR5-null natural hosts and efficient alternative coreceptor use in vivoClinical significance of HIV-1 coreceptor usage.Bioinformatic analysis of HIV-1 entry and pathogenesis.HIV-1 escape from a small molecule, CCR5-specific entry inhibitor does not involve CXCR4 useHuman immunodeficiency virus 1 envelope glycoprotein complex-induced apoptosis involves mammalian target of rapamycin/FKBP12-rapamycin-associated protein-mediated p53 phosphorylationHIV and Tuberculosis: a Deadly Human SyndemicDefining the fitness of HIV-1 isolates with dual/mixed co-receptor usageThe effect of Trim5 polymorphisms on the clinical course of HIV-1 infection.Critical involvement of the ATM-dependent DNA damage response in the apoptotic demise of HIV-1-elicited syncytia.HIV-1 with multiple CCR5/CXCR4 chimeric receptor use is predictive of immunological failure in infected children.HIV-1 residual viremia correlates with persistent T-cell activation in poor immunological responders to combination antiretroviral therapy.HIV-1 clade B Tat, but not clade C Tat, increases X4 HIV-1 entry into resting but not activated CD4+ T cells.Diversity of HIV-1 subtype B: implications to the origin of BF recombinantsEvolution of the HIV-1 env gene in the Rag2-/- gammaC-/- humanized mouse modelHIV-1 gp41 and TCRalpha trans-membrane domains share a motif exploited by the HIV virus to modulate T-cell proliferationDifferences in the fitness of two diverse wild-type human immunodeficiency virus type 1 isolates are related to the efficiency of cell binding and entry.Resting naive CD4+ T cells are massively infected and eliminated by X4-tropic simian-human immunodeficiency viruses in macaquesThe replicative fitness of primary human immunodeficiency virus type 1 (HIV-1) group M, HIV-1 group O, and HIV-2 isolates.X4 and R5 HIV-1 have distinct post-entry requirements for uracil DNA glycosylase during infection of primary cells.Differential pathogenesis of primary CCR5-using human immunodeficiency virus type 1 isolates in ex vivo human lymphoid tissue.Adoption of an "open" envelope conformation facilitating CD4 binding and structural remodeling precedes coreceptor switch in R5 SHIV-infected macaques.Switching of inferred tropism caused by HIV during interruption of antiretroviral therapy.T cell depletion in HIV-1 infection: how CD4+ T cells go out of stock.Sequential involvement of Cdk1, mTOR and p53 in apoptosis induced by the HIV-1 envelope.Variation in the biological properties of HIV-1 R5 envelopes: implications of envelope structure, transmission and pathogenesis.Human immunodeficiency virus type 1 fitness is a determining factor in viral rebound and set point in chronic infection.Naïve and memory cell turnover as drivers of CCR5-to-CXCR4 tropism switch in human immunodeficiency virus type 1: implications for therapy.Relaxation of adaptive evolution during the HIV-1 infection owing to reduction of CD4+ T cell countsBoth R5 and X4 human immunodeficiency virus type 1 variants persist during prolonged therapy with five antiretroviral drugs.HIV-1 tropism determination using a phenotypic Env recombinant viral assay highlights overestimation of CXCR4-usage by genotypic prediction algorithms for CRF01_AE and CRF02_AG [corrected].Non-macrophage-tropic human immunodeficiency virus type 1 R5 envelopes predominate in blood, lymph nodes, and semen: implications for transmission and pathogenesis.Distribution of HIV-1 infection in different T lymphocyte subsets: antiretroviral therapy-naïve vs. experienced patients.A single HIV-1 cluster and a skewed immune homeostasis drive the early spread of HIV among resting CD4+ cell subsets within one month post-infectionEffect of B-cell depletion on coreceptor switching in R5 simian-human immunodeficiency virus infection of rhesus macaques.Identification of a subset of human immunodeficiency virus type 1 (HIV-1), HIV-2, and simian immunodeficiency virus strains able to exploit an alternative coreceptor on untransformed human brain and lymphoid cells.The presence of CXCR4-using HIV-1 prior to start of antiretroviral therapy is an independent predictor of delayed viral suppressionHIV-1 co-receptor usage: influence on mother-to-child transmission and pediatric infection
P2860
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P2860
In vivo HIV-1 infection of CD45RA(+)CD4(+) T cells is established primarily by syncytium-inducing variants and correlates with the rate of CD4(+) T cell decline
description
2000 nî lūn-bûn
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2000 թուականի Փետրուարին հրատարակուած գիտական յօդուած
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2000 թվականի փետրվարին հրատարակված գիտական հոդված
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2000年の論文
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2000年論文
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2000年論文
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2000年論文
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2000年論文
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2000年论文
@wuu
name
In vivo HIV-1 infection of CD4 ...... rate of CD4(+) T cell decline
@ast
In vivo HIV-1 infection of CD4 ...... rate of CD4(+) T cell decline
@en
type
label
In vivo HIV-1 infection of CD4 ...... rate of CD4(+) T cell decline
@ast
In vivo HIV-1 infection of CD4 ...... rate of CD4(+) T cell decline
@en
prefLabel
In vivo HIV-1 infection of CD4 ...... rate of CD4(+) T cell decline
@ast
In vivo HIV-1 infection of CD4 ...... rate of CD4(+) T cell decline
@en
P2093
P2860
P356
P1476
In vivo HIV-1 infection of CD4 ...... rate of CD4(+) T cell decline
@en
P2093
Hooibrink B
Hovenkamp E
Schuitemaker H
van't Wout AB
P2860
P304
P356
10.1073/PNAS.97.3.1269
P407
P577
2000-02-01T00:00:00Z