Both memory and CD45RA+/CD62L+ naive CD4(+) T cells are infected in human immunodeficiency virus type 1-infected individuals
about
HIV envelope induces a cascade of cell signals in non-proliferating target cells that favor virus replicationCD226 (DNAM-1) is involved in lymphocyte function-associated antigen 1 costimulatory signal for naive T cell differentiation and proliferationDendritic Cells in HIV-1 and HCV Infection: Can They Help Win the Battle?Recent developments in human immunodeficiency virus-1 latency researchClinical significance of HIV-1 coreceptor usage.Dynamic correlation between intrahost HIV-1 quasispecies evolution and disease progression.HIV-1 residual viremia correlates with persistent T-cell activation in poor immunological responders to combination antiretroviral therapy.RNA interference screen in primary human T cells reveals FLT3 as a modulator of IL-10 levels.HIV-1 gp41 and TCRalpha trans-membrane domains share a motif exploited by the HIV virus to modulate T-cell proliferationPreferential feline immunodeficiency virus (FIV) infection of CD4+ CD25+ T-regulatory cells correlates both with surface expression of CXCR4 and activation of FIV long terminal repeat binding cellular transcriptional factorsRNAi-mediated CCR5 silencing by LFA-1-targeted nanoparticles prevents HIV infection in BLT miceInfection of the CD45RA+ (naive) subset of peripheral CD8+ lymphocytes by human immunodeficiency virus type 1 in vivo.Induction of cell death in human immunodeficiency virus-infected macrophages and resting memory CD4 T cells by TRAIL/Apo2lActivated peripheral CD8 lymphocytes express CD4 in vivo and are targets for infection by human immunodeficiency virus type 1.Coreceptor and cytokine concentrations may not explain differences in disease progression observed in HIV-1 clade A and D infected Ugandans.Differential pathogenesis of primary CCR5-using human immunodeficiency virus type 1 isolates in ex vivo human lymphoid tissue.IL-7 differentially regulates cell cycle progression and HIV-1-based vector infection in neonatal and adult CD4+ T cells.Human immunodeficiency virus type 1 can establish latent infection in resting CD4+ T cells in the absence of activating stimuli.Variation in the biological properties of HIV-1 R5 envelopes: implications of envelope structure, transmission and pathogenesis.Higher levels of Zidovudine resistant HIV in the colon compared to blood and other gastrointestinal compartments in HIV infection.R5 variants of human immunodeficiency virus type 1 preferentially infect CD62L- CD4+ T cells and are potentially resistant to nucleoside reverse transcriptase inhibitorsVaried sensitivity to therapy of HIV-1 strains in CD4+ lymphocyte sub-populations upon ART initiation.Non-macrophage-tropic human immunodeficiency virus type 1 R5 envelopes predominate in blood, lymph nodes, and semen: implications for transmission and pathogenesis.Single cell analysis of lymph node tissue from HIV-1 infected patients reveals that the majority of CD4+ T-cells contain one HIV-1 DNA molecule.Identification of a subset of human immunodeficiency virus type 1 (HIV-1), HIV-2, and simian immunodeficiency virus strains able to exploit an alternative coreceptor on untransformed human brain and lymphoid cells.In vivo HIV-1 infection of CD45RA(+)CD4(+) T cells is established primarily by syncytium-inducing variants and correlates with the rate of CD4(+) T cell declineDifferential susceptibility of naïve, central memory and effector memory T cells to dendritic cell-mediated HIV-1 transmission.Differential coreceptor expression allows for independent evolution of non-syncytium-inducing and syncytium-inducing HIV-1.Phenotypic and immunologic comparison of clade B transmitted/founder and chronic HIV-1 envelope glycoproteins.CD4+ T cells from elite suppressors are more susceptible to HIV-1 but produce fewer virions than cells from chronic progressors.Suppression of Foxo1 activity and down-modulation of CD62L (L-selectin) in HIV-1 infected resting CD4 T cells.HIV Latency Is Established Directly and Early in Both Resting and Activated Primary CD4 T Cells.HIV persistence in the setting of antiretroviral therapy: when, where and how does HIV hide?Porphyromonas gingivalis-mediated signaling through TLR4 mediates persistent HIV infection of primary macrophages.Endogenous factors enhance HIV infection of tissue naive CD4 T cells by stimulating high molecular mass APOBEC3G complex formationHIV-1 Vpr enhances viral burden by facilitating infection of tissue macrophages but not nondividing CD4+ T cells.CD1d-restricted human natural killer T cells are highly susceptible to human immunodeficiency virus 1 infection.Compartmentalization of the gut viral reservoir in HIV-1 infected patients.Loss of naïve (CD45RA+) CD4+ lymphocytes during pediatric infection with feline immunodeficiency virus.A Subset of CD4/CD8 Double-Negative T Cells Expresses HIV Proteins in Patients on Antiretroviral Therapy.
P2860
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P2860
Both memory and CD45RA+/CD62L+ naive CD4(+) T cells are infected in human immunodeficiency virus type 1-infected individuals
description
1999 nî lūn-bûn
@nan
1999 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Both memory and CD45RA+/CD62L+ ...... us type 1-infected individuals
@ast
Both memory and CD45RA+/CD62L+ ...... us type 1-infected individuals
@en
type
label
Both memory and CD45RA+/CD62L+ ...... us type 1-infected individuals
@ast
Both memory and CD45RA+/CD62L+ ...... us type 1-infected individuals
@en
prefLabel
Both memory and CD45RA+/CD62L+ ...... us type 1-infected individuals
@ast
Both memory and CD45RA+/CD62L+ ...... us type 1-infected individuals
@en
P2093
P2860
P1433
P1476
Both memory and CD45RA+/CD62L+ ...... us type 1-infected individuals
@en
P2093
Adelsberger J
Hallahan CW
Justement SJ
Ostrowski MA
Spinelli MA
P2860
P304
P577
1999-08-01T00:00:00Z