Modulation of insulin-like growth factor (IGF)-I and IGF-binding protein interactions enhances skeletal muscle regeneration and ameliorates the dystrophic pathology in mdx mice.
about
Multifaceted role of insulin-like growth factors and mammalian target of rapamycin in skeletal muscleDeletion of skeletal muscle SOCS3 prevents insulin resistance in obesityPolycomb EZH2 controls self-renewal and safeguards the transcriptional identity of skeletal muscle stem cellsThe histone- and PRMT5-associated protein COPR5 is required for myogenic differentiationHsp72 preserves muscle function and slows progression of severe muscular dystrophyVitamin D and human skeletal muscle.Antibody-directed myostatin inhibition improves diaphragm pathology in young but not adult dystrophic mdx mice.Targeting the activin type IIB receptor to improve muscle mass and function in the mdx mouse model of Duchenne muscular dystrophy.Vitamin d and athletic performance: the potential role of muscle.Anabolic agents for improving muscle regeneration and function after injury.Attenuated muscle regeneration is a key factor in dysferlin-deficient muscular dystrophy.Emerging drugs for treating skeletal muscle injury and promoting muscle repair.Prospect for pharmacological therapies to treat skeletal muscle dysfunction.Trends in the Design and Development of Specific Aptamers Against Peptides and Proteins.High-Methionine Diet Attenuates Severity of Arthritis and Modulates IGF-I Related Gene Expressions in an Adjuvant Arthritis Rats Model.Growth factor and cytokine interactions in myogenesis. Part II. Expression of IGF binding proteins and protein kinases essential for myogenesis in mouse C2C12 myogenic cells exposed to TNF-alpha and IFN-gamma.Contraction-related stimuli regulate GLUT4 traffic in C2C12-GLUT4myc skeletal muscle cells.Silencing Nfix rescues muscular dystrophy by delaying muscle regeneration.Downstream mechanisms of nitric oxide-mediated skeletal muscle glucose uptake during contraction.Skeletal muscle glucose uptake during contraction is regulated by nitric oxide and ROS independently of AMPK.Systemic IGF-I administration attenuates the inhibitory effect of chronic arthritis on gastrocnemius mass and decreases atrogin-1 and IGFBP-3.Systemic cell cycle activation is induced following complex tissue injury in axolotl.Insulin-like growth factor-I analogue protects muscles of dystrophic mdx mice from contraction-mediated damage.Citrulline does not prevent skeletal muscle wasting or weakness in limb-casted mice.
P2860
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P2860
Modulation of insulin-like growth factor (IGF)-I and IGF-binding protein interactions enhances skeletal muscle regeneration and ameliorates the dystrophic pathology in mdx mice.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Modulation of insulin-like gro ...... trophic pathology in mdx mice.
@ast
Modulation of insulin-like gro ...... trophic pathology in mdx mice.
@en
type
label
Modulation of insulin-like gro ...... trophic pathology in mdx mice.
@ast
Modulation of insulin-like gro ...... trophic pathology in mdx mice.
@en
prefLabel
Modulation of insulin-like gro ...... trophic pathology in mdx mice.
@ast
Modulation of insulin-like gro ...... trophic pathology in mdx mice.
@en
P2860
P1476
Modulation of insulin-like gro ...... trophic pathology in mdx mice.
@en
P2093
Jonathan D Schertzer
Stefan M Gehrig
P2860
P304
P356
10.2353/AJPATH.2007.070292
P407
P577
2007-09-06T00:00:00Z