Oral glycotoxins are a modifiable cause of dementia and the metabolic syndrome in mice and humans - Wikidata - awgldk - TriplyDB

Oral glycotoxins are a modifiable cause of dementia and the metabolic syndrome in mice and humans

Oral glycotoxins are a modifiable cause of dementia and the metabolic syndrome in mice and humans

http://www.wikidata.org/entity/Q30575530

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Current perspectives on the health risks associated with the consumption of advanced glycation end products: recommendations for dietary management Food Processing and the Mediterranean Diet Advanced glycation end-products: modifiable environmental factors profoundly mediate insulin resistance Dietary advanced glycation end products and their role in health and disease Using Multicountry Ecological and Observational Studies to Determine Dietary Risk Factors for Alzheimer's Disease.Dietary interventions to contrast the onset and progression of diabetic nephropathy: A critical survey of new data.Dietary Sugars and Endogenous Formation of Advanced Glycation Endproducts: Emerging Mechanisms of Disease.On the Relationship between Energy Metabolism, Proteostasis, Aging and Parkinson's Disease: Possible Causative Role of Methylglyoxal and Alleviative Potential of Carnosine.Dietary advanced glycation end products are associated with decline in memory in young elderly.Dietary fat quantity and quality modifies advanced glycation end products metabolism in patients with metabolic syndrome.Dietary consumption of advanced glycation end products and pancreatic cancer in the prospective NIH-AARP Diet and Health Study.Glycation of human cortical and cancellous bone captures differences in the formation of Maillard reaction products between glucose and ribose Effects of sevelamer carbonate on advanced glycation end products and antioxidant/pro-oxidant status in patients with diabetic kidney disease.Let Food Be Thy Medicine: Diet, Nutrition, and Biomarkers' Risk of Alzheimer's Disease.Dietary glycemia as a determinant of health and longevity Production of butyrate from lysine and the Amadori product fructoselysine by a human gut commensal.Uremic Toxicity of Advanced Glycation End Products in CKD.High dietary advanced glycation end products are associated with poorer spatial learning and accelerated Aβ deposition in an Alzheimer mouse model.Cerebral Ketone Body Oxidation Is Facilitated by a High Fat Diet Enriched with Advanced Glycation End Products in Normal and Diabetic Rats.Dietary glycotoxins and infant formulas.Nutrition and AGE-ing: Focusing on Alzheimer's Disease.Methylglyoxal-Glyoxalase 1 Balance: The Root of Vascular Damage.Advanced glycation end products, dementia, and diabetes The role of advanced glycation end products in various types of neurodegenerative disease: a therapeutic approach.Post-ischemic inflammation regulates neural damage and protection.Effect of diet-derived advanced glycation end products on inflammation.Carboxymethyl-lysine: thirty years of investigation in the field of AGE formation.Common neurodegenerative pathways in obesity, diabetes, and Alzheimer's disease.Calycosin Rebalances Advanced Glycation End Products-Induced Glucose Uptake Dysfunction of Hepatocyte In Vitro.Cellular mechanisms and consequences of glycation in atherosclerosis and obesity.Preserving Brain Function in Aging: The Anti-glycative Potential of Berry Fruit.Implication of advanced glycation end products (Ages) and their receptor (Rage) on myocardial contractile and mitochondrial functions.Methylglyoxal in Metabolic Disorders: Facts, Myths, and Promises.Kinetic Study on Peptide-Bound Pyrraline Formation and Elimination in the Maillard Reaction Using Single- and Multiple-Response Models.Oral AGE restriction ameliorates insulin resistance in obese individuals with the metabolic syndrome: a randomised controlled trial.Evaluation of individual aging degree by standard-free, label-free LC-MS/MS quantification of formaldehyde-modified peptides.Association of advanced glycation end products, evaluated by skin autofluorescence, with lifestyle habits in a general Japanese population.Dietary CML-enriched protein induces functional arterial aging in a RAGE-dependent manner in mice.Mediterranean Diet Supplemented With Coenzyme Q10 Modulates the Postprandial Metabolism of Advanced Glycation End Products in Elderly Men and Women.Immunomodulation by Processed Animal Feed: The Role of Maillard Reaction Products and Advanced Glycation End-Products (AGEs)

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Oral glycotoxins are a modifiable cause of dementia and the metabolic syndrome in mice and humans

http://www.wikidata.org/entity/Q30575530

description

2014 nî lūn-bûn

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2014 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2014 թվականի փետրվարին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Oral glycotoxins are a modifia ...... ic syndrome in mice and humans

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Oral glycotoxins are a modifia ...... ic syndrome in mice and humans

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Oral glycotoxins are a modifia ...... ic syndrome in mice and humans

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Oral glycotoxins are a modifia ...... ic syndrome in mice and humans

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Oral glycotoxins are a modifia ...... ic syndrome in mice and humans

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PNAS.1316013111

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Proceedings of the National Academy of Sciences of the United States of America

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Oral glycotoxins are a modifia ...... ic syndrome in mice and humans

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P2093

Calogera Simonaro

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Fabrizio Grosjean

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Gary E Striker

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Helen Vlassara

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Jaime Uribarri

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Li Zhu

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Mark Woodward

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Michal Schnaider-Beeri

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Shobha Swamy

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Weijing Cai

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P2860

RAGE-mediated signaling contributes to intraneuronal transport of amyloid-beta and neuronal dysfunction

SIRT1 suppresses beta-amyloid production by activating the alpha-secretase gene ADAM10.

Advanced glycation end products in foods and a practical guide to their reduction in the diet

Orally absorbed reactive glycation products (glycotoxins): an environmental risk factor in diabetic nephropathy

A multimodal RAGE-specific inhibitor reduces amyloid β-mediated brain disorder in a mouse model of Alzheimer disease

SIRT1 is essential for normal cognitive function and synaptic plasticity

Receptors for advanced glycosylation endproducts in human brain: role in brain homeostasis

Neurotoxicity of advanced glycation endproducts during focal stroke and neuroprotective effects of aminoguanidine

Insulin-resistant brain state: the culprit in sporadic Alzheimer's disease?

Restriction of advanced glycation end products improves insulin resistance in human type 2 diabetes: potential role of AGER1 and SIRT1

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4940-4945

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111

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2014-02-24T00:00:00Z

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260379422

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24567379

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metabolic syndrome

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3977256

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