The c-terminus of GRK3 indicates rapid dissociation of G protein heterotrimers.
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GPR158/179 regulate G protein signaling by controlling localization and activity of the RGS7 complexesEssential role of the m2R-RGS6-IKACh pathway in controlling intrinsic heart rate variabilityG protein beta 5 is targeted to D2-dopamine receptor-containing biochemical compartments and blocks dopamine-dependent receptor internalizationGNB5 mutation causes a novel neuropsychiatric disorder featuring attention deficit hyperactivity disorder, severely impaired language development and normal cognitionStable G protein-effector complexes in striatal neurons: mechanism of assembly and role in neurotransmitter signaling.Förster resonance energy transfer as a tool to study photoreceptor biologySubcellular optogenetic inhibition of G proteins generates signaling gradients and cell migration.Third-party bioluminescence resonance energy transfer indicates constitutive association of membrane proteins: application to class a g-protein-coupled receptors and g-proteins.Regulators of G-protein signaling accelerate GPCR signaling kinetics and govern sensitivity solely by accelerating GTPase activity.Mechanisms underlying the activation of G-protein-gated inwardly rectifying K+ (GIRK) channels by the novel anxiolytic drug, ML297.Making structural sense of dimerization interfaces of delta opioid receptor homodimers.Expression and iron-dependent regulation of succinate receptor GPR91 in retinal pigment epitheliumInteraction of transducin with uncoordinated 119 protein (UNC119): implications for the model of transducin trafficking in rod photoreceptors.Effects of chlorpyrifos and chlorpyrifos-oxon on the dynamics and movement of mitochondria in rat cortical neurons.G protein-coupled receptors and resistance to inhibitors of cholinesterase-8A (Ric-8A) both regulate the regulator of g protein signaling 14 RGS14·Gαi1 complex in live cellsIntegrins activate trimeric G proteins via the nonreceptor protein GIV/GirdinGIRK channel modulation by assembly with allosterically regulated RGS proteinsMutations in GNAL cause primary torsion dystonia.The complex of G protein regulator RGS9-2 and Gβ(5) controls sensitization and signaling kinetics of type 5 adenylyl cyclase in the striatumAssociation with the Plasma Membrane Is Sufficient for Potentiating Catalytic Activity of Regulators of G Protein Signaling (RGS) Proteins of the R7 Subfamily.Dominant-negative Gα subunits are a mechanism of dysregulated heterotrimeric G protein signaling in human disease.Distinct profiles of functional discrimination among G proteins determine the actions of G protein-coupled receptors.Macromolecular composition dictates receptor and G protein selectivity of regulator of G protein signaling (RGS) 7 and 9-2 protein complexes in living cells.Synergistically acting agonists and antagonists of G protein-coupled receptors prevent photoreceptor cell degeneration.L-2-oxothiazolidine-4-carboxylic acid attenuates oxidative stress and inflammation in retinal pigment epitheliumRGS7/Gβ5/R7BP complex regulates synaptic plasticity and memory by modulating hippocampal GABABR-GIRK signaling.Optogenetic reporters.Monitoring G Protein Activation in Cells with BRET.RGS14 regulates the lifetime of Gα-GTP signaling but does not prolong Gβγ signaling following receptor activation in live cells.LGR5 receptor promotes cell-cell adhesion in stem cells and colon cancer cells via the IQGAP1-Rac1 pathway.Dissociated GαGTP and Gβγ protein subunits are the major activated form of heterotrimeric Gi/o proteins.All G protein βγ complexes are capable of translocation on receptor activation.Blockade of dendritic cell development by bacterial fermentation products butyrate and propionate through a transporter (Slc5a8)-dependent inhibition of histone deacetylasesDifferential regulation of phospholipase C-beta2 activity and membrane interaction by Galphaq, Gbeta1gamma2, and Rac2.Pharmacogenomics of GPCR Drug Targets.Specific inhibition of GPCR-independent G protein signaling by a rationally engineered protein.Rapid kinetic BRET measurements to monitor G protein activation by GPCR and non-GPCR proteins.Overexpression of GRK3, Promoting Tumor Proliferation, Is Predictive of Poor Prognosis in Colon Cancer.Gi;Go Heterotrimeric G-protein complex dissociatesMembrane Recruitment of the Non-receptor Protein GIV/Girdin (Gα-interacting, Vesicle-associated Protein/Girdin) Is Sufficient for Activating Heterotrimeric G Protein Signaling.
P2860
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P2860
The c-terminus of GRK3 indicates rapid dissociation of G protein heterotrimers.
description
2009 nî lūn-bûn
@nan
2009 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
The c-terminus of GRK3 indicates rapid dissociation of G protein heterotrimers.
@ast
The c-terminus of GRK3 indicates rapid dissociation of G protein heterotrimers.
@en
The c-terminus of GRK3 indicates rapid dissociation of G protein heterotrimers.
@nl
type
label
The c-terminus of GRK3 indicates rapid dissociation of G protein heterotrimers.
@ast
The c-terminus of GRK3 indicates rapid dissociation of G protein heterotrimers.
@en
The c-terminus of GRK3 indicates rapid dissociation of G protein heterotrimers.
@nl
altLabel
The c-terminus of GRK3 indicates rapid dissociation of G protein heterotrimers
@en
prefLabel
The c-terminus of GRK3 indicates rapid dissociation of G protein heterotrimers.
@ast
The c-terminus of GRK3 indicates rapid dissociation of G protein heterotrimers.
@en
The c-terminus of GRK3 indicates rapid dissociation of G protein heterotrimers.
@nl
P2093
P2860
P3181
P1433
P1476
The c-terminus of GRK3 indicates rapid dissociation of G protein heterotrimers.
@en
P2093
Bettye Hollins
Gregory J Digby
Nevin A Lambert
Sudhakiranmayi Kuravi
P2860
P304
P3181
P356
10.1016/J.CELLSIG.2009.02.017
P407
P577
2009-06-01T00:00:00Z