Cdc42 in oncogenic transformation, invasion, and tumorigenesis - Wikidata - awgldk - TriplyDB

Cdc42 in oncogenic transformation, invasion, and tumorigenesis

Cdc42 in oncogenic transformation, invasion, and tumorigenesis

http://www.wikidata.org/entity/Q28236231

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RhoT1 and Smad4 are correlated with lymph node metastasis and overall survival in pancreatic cancer SPARC Promotes Cell Invasion In Vivo by Decreasing Type IV Collagen Levels in the Basement Membrane GDI-mediated cell polarization in yeast provides precise spatial and temporal control of Cdc42 signaling A new genetically encoded single-chain biosensor for Cdc42 based on FRET, useful for live-cell imaging The dual regulatory role of miR-204 in cancer Targeting Cdc42 with the small molecule drug AZA197 suppresses primary colon cancer growth and prolongs survival in a preclinical mouse xenograft model by downregulation of PAK1 activity Pax6 exerts regional control of cortical progenitor proliferation via direct repression of Cdk6 and hypophosphorylation of pRb A Rac1/Cdc42 GTPase-specific small molecule inhibitor suppresses growth of primary human prostate cancer xenografts and prolongs survival in mice A Pan-GTPase Inhibitor as a Molecular Probe Novel Activities of Select NSAID R-Enantiomers against Rac1 and Cdc42 GTPases The cation channel Trpv2 is a new suppressor of arthritis severity, joint damage, and synovial fibroblast invasion Cdc42 promotes transendothelial migration of cancer cells through β1 integrin.Small molecule targeting Cdc42-intersectin interaction disrupts Golgi organization and suppresses cell motility.Identification of DPPA4 and DPPA2 as a novel family of pluripotency-related oncogenes.Identifying subtype-specific associations between gene expression and DNA methylation profiles in breast cancer Expression and significance of Rac1, Pak1 and Rock1 in gastric carcinoma.Essential role of Cdc42 in Ras-induced transformation revealed by gene targeting The Rac Inhibitor EHop-016 Inhibits Mammary Tumor Growth and Metastasis in a Nude Mouse Model.Transcriptomic changes during regeneration of the central nervous system in an echinoderm.Targeting Rho-GTPases in immune cell migration and inflammation.Silencing of CDC42 inhibits neuroblastoma cell proliferation and transformation Cis-eQTL analysis and functional validation of candidate susceptibility genes for high-grade serous ovarian cancer microRNA-195 inhibits cell proliferation in bladder cancer via inhibition of cell division control protein 42 homolog/signal transducer and activator of transcription-3 signaling.Evolutionary trends and functional anatomy of the human expanded autophagy network.Myotonic dystrophy kinase-related Cdc42-binding kinases (MRCK), the ROCK-like effectors of Cdc42 and Rac1.A Novel Pharmacologic Activity of Ketorolac for Therapeutic Benefit in Ovarian Cancer Patients.The isolation and characterization of CTC subsets related to breast cancer dormancy Regulation and functional significance of CDC42 alternative splicing in ovarian cancer Redundant and nonredundant roles for Cdc42 and Rac1 in lymphomas developed in NPM-ALK transgenic mice.Characterization of a Cdc42 protein inhibitor and its use as a molecular probe.ARHGAP10, downregulated in ovarian cancer, suppresses tumorigenicity of ovarian cancer cells.Roles of rho GTPases in intracellular transport and cellular transformation.FMNL2/FMNL3 formins are linked with oncogenic pathways and predict melanoma outcome.Systems-wide analysis of K-Ras, Cdc42, and PAK4 signaling by quantitative phosphoproteomics.CD47 Promotes Tumor Invasion and Metastasis in Non-small Cell Lung Cancer.Deletion of Cdc42 enhances ADAM17-mediated vascular endothelial growth factor receptor 2 shedding and impairs vascular endothelial cell survival and vasculogenesis.Variation at 2q35 (PNKD and TMBIM1) influences colorectal cancer risk and identifies a pleiotropic effect with inflammatory bowel disease.Expression of CDC42 in cervical squamous cell carcinoma and its correlation with clinicopathologic characteristics.Dihydroartemisinin suppresses pancreatic cancer cells via a microRNA-mRNA regulatory network.Cdc42 overexpression induces hyperbranching in the developing mammary gland by enhancing cell migration.

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Cdc42 in oncogenic transformation, invasion, and tumorigenesis

http://www.wikidata.org/entity/Q28236231

description

2011 nî lūn-bûn

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2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Cdc42 in oncogenic transformation, invasion, and tumorigenesis

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Cdc42 in oncogenic transformation, invasion, and tumorigenesis

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Cdc42 in oncogenic transformation, invasion, and tumorigenesis

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type

label

Cdc42 in oncogenic transformation, invasion, and tumorigenesis

@ast

Cdc42 in oncogenic transformation, invasion, and tumorigenesis

@en

Cdc42 in oncogenic transformation, invasion, and tumorigenesis

@nl

prefLabel

Cdc42 in oncogenic transformation, invasion, and tumorigenesis

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Cdc42 in oncogenic transformation, invasion, and tumorigenesis

@en

Cdc42 in oncogenic transformation, invasion, and tumorigenesis

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J.CELLSIG.2011.04.001

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Cellular Signalling

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Cdc42 in oncogenic transformation, invasion, and tumorigenesis

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Kristy Stengel

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Yi Zheng

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P2860

Molecular cloning and characterization of the human dbl proto-oncogene: evidence that its overexpression is sufficient to transform NIH/3T3 cells

DLC-1: a Rho GTPase-activating protein and tumour suppressor

The faciogenital dysplasia gene product FGD1 functions as a Cdc42Hs-specific guanine-nucleotide exchange factor

The adenomatous polyposis coli-associated exchange factors Asef and Asef2 are required for adenoma formation in Apc(Min/+)mice

Identification and characterization of Asef2, a guanine-nucleotide exchange factor specific for Rac1 and Cdc42

DOCK10-mediated Cdc42 activation is necessary for amoeboid invasion of melanoma cells

Asef2 functions as a Cdc42 exchange factor and is stimulated by the release of an autoinhibitory module from a concealed C-terminal activation element

Rho GDP dissociation inhibitor 2 suppresses metastasis via unconventional regulation of RhoGTPases.

Molecular mechanisms of invadopodium formation: the role of the N-WASP-Arp2/3 complex pathway and cofilin

Cdc42: An essential Rho-type GTPase controlling eukaryotic cell polarity.

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10.1016/J.CELLSIG.2011.04.001

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21515363

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