JunB Inhibits ER Stress and Apoptosis in Pancreatic Beta Cells - Wikidata - awgldk - TriplyDB

JunB Inhibits ER Stress and Apoptosis in Pancreatic Beta Cells

JunB Inhibits ER Stress and Apoptosis in Pancreatic Beta Cells

http://www.wikidata.org/entity/Q28473492

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p53 up-regulated modulator of apoptosis (PUMA) activation contributes to pancreatic beta-cell apoptosis induced by proinflammatory cytokines and endoplasmic reticulum stress JunB protects β-cells from lipotoxicity via the XBP1-AKT pathway.Regulation of CCAAT/enhancer-binding protein homologous protein (CHOP) expression by interleukin-1 beta in pancreatic beta cells The transcription factor C/EBP delta has anti-apoptotic and anti-inflammatory roles in pancreatic beta cells The duration of gastrin treatment affects global gene expression and molecular responses involved in ER stress and anti-apoptosis.HuR Contributes to TRAIL Resistance by Restricting Death Receptor 4 Expression in Pancreatic Cancer Cells Glucagon-like peptide-1 agonists protect pancreatic beta-cells from lipotoxic endoplasmic reticulum stress through upregulation of BiP and JunB.MDA5 and PTPN2, two candidate genes for type 1 diabetes, modify pancreatic beta-cell responses to the viral by-product double-stranded RNA.The proapoptotic BH3-only proteins Bim and Puma are downstream of endoplasmic reticulum and mitochondrial oxidative stress in pancreatic islets in response to glucotoxicity Functional and morphological impact of ER stress on mitochondria.Sumoylation Modulates the Susceptibility to Type 1 Diabetes.4-Deoxypyridoxine improves the viability of isolated pancreatic islets ex vivo.Cross-talk between the unfolded protein response and nuclear factor-κB signalling pathways regulates cytokine-mediated beta cell death in MIN6 cells and isolated mouse islets.Differential usage of NF-κB activating signals by IL-1β and TNF-α in pancreatic beta cells.The transcription factor c-Jun protects against sustained hepatic endoplasmic reticulum stress thereby promoting hepatocyte survival.Quantitative proteomic analysis of host epithelial cells infected by Salmonella enterica serovar Typhimurium.Induction of ATF3 gene network by triglyceride-rich lipoprotein lipolysis products increases vascular apoptosis and inflammation SRp55 Regulates a Splicing Network that Controls Human Pancreatic Beta Cell Function and Survival.MicroRNA 199a-5p induces apoptosis by targeting JunB.Pancreatic β-cells activate a JunB/ATF3-dependent survival pathway during inflammation

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JunB Inhibits ER Stress and Apoptosis in Pancreatic Beta Cells

http://www.wikidata.org/entity/Q28473492

description

2008 nî lūn-bûn

@nan

2008 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2008年の論文

@ja

2008年論文

@yue

2008年論文

@zh-hant

2008年論文

@zh-hk

2008年論文

@zh-mo

2008年論文

@zh-tw

2008年论文

@wuu

name

JunB Inhibits ER Stress and Apoptosis in Pancreatic Beta Cells

@ast

JunB Inhibits ER Stress and Apoptosis in Pancreatic Beta Cells

@en

JunB Inhibits ER Stress and Apoptosis in Pancreatic Beta Cells

@nl

type

label

JunB Inhibits ER Stress and Apoptosis in Pancreatic Beta Cells

@ast

JunB Inhibits ER Stress and Apoptosis in Pancreatic Beta Cells

@en

JunB Inhibits ER Stress and Apoptosis in Pancreatic Beta Cells

@nl

prefLabel

JunB Inhibits ER Stress and Apoptosis in Pancreatic Beta Cells

@ast

JunB Inhibits ER Stress and Apoptosis in Pancreatic Beta Cells

@en

JunB Inhibits ER Stress and Apoptosis in Pancreatic Beta Cells

@nl

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JOURNAL.PONE.0003030

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JunB Inhibits ER Stress and Apoptosis in Pancreatic Beta Cells

@en

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Decio L Eizirik

http://www.w3.org/2001/XMLSchema#string

Latifa Bakiri

http://www.w3.org/2001/XMLSchema#string

P2860

Histone modifications defining active genes persist after transcriptional and mitotic inactivation

Efficient selection for high-expression transfectants with a novel eukaryotic vector

Targeting c-Jun and JunB proteins as potential anticancer cell therapy

Targeted disruption of the Chop gene delays endoplasmic reticulum stress-mediated diabetes

Toll-like receptor 3 and STAT-1 contribute to double-stranded RNA+ interferon-gamma-induced apoptosis in primary pancreatic beta-cells

JunB can substitute for Jun in mouse development and cell proliferation

AP-1 as a regulator of cell life and death

Nitric oxide synthases: roles, tolls, and controls

Nitric oxide-induced apoptosis in pancreatic beta cells is mediated by the endoplasmic reticulum stress pathway.

Suppressor of cytokine signaling 3 (SOCS-3) protects beta -cells against interleukin-1beta - and interferon-gamma -mediated toxicity

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e3030

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scholarly article

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10.1371/JOURNAL.PONE.0003030

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8

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3

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Esteban N. Gurzov

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2008-08-21T00:00:00Z

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23183799

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18716665

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2516602

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