NG2: a component of the glial scar that inhibits axon growth. - Wikidata - awgldk - TriplyDB

NG2: a component of the glial scar that inhibits axon growth.

NG2: a component of the glial scar that inhibits axon growth.

http://www.wikidata.org/entity/Q36347824

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Chondroitin sulfate proteoglycans in the nervous system: inhibitors to repair Biomaterial Approaches to Enhancing Neurorestoration after Spinal Cord Injury: Strategies for Overcoming Inherent Biological Obstacles Role of the lesion scar in the response to damage and repair of the central nervous system Androgens and stroke: good, bad or indifferent?Spatio-temporal expression pattern of frizzled receptors after contusive spinal cord injury in adult rats Stem cell therapy for central nerve system injuries: glial cells hold the key Adult oligodendrocyte progenitor cells - Multifaceted regulators of the CNS in health and disease.Adult NG2+ cells are permissive to neurite outgrowth and stabilize sensory axons during macrophage-induced axonal dieback after spinal cord injury.Temporal patterns of cortical proliferation of glial cell populations after traumatic brain injury in mice The morphology and spatial arrangement of astrocytes in the optic nerve head of the mouse.Contributions of chondroitin sulfate proteoglycans to neurodevelopment, injury, and cancer.Entrapment via synaptic-like connections between NG2 proteoglycan+ cells and dystrophic axons in the lesion plays a role in regeneration failure after spinal cord injury.NG2+ progenitors derived from embryonic stem cells penetrate glial scar and promote axonal outgrowth into white matter after spinal cord injury NMDA receptor signaling in oligodendrocyte progenitors is not required for oligodendrogenesis and myelination NG2-glia and their functions in the central nervous system.NG2, a common denominator for neuroinflammation, blood-brain barrier alteration, and oligodendrocyte precursor response in EAE, plays a role in dendritic cell activation Delayed cell cycle pathway modulation facilitates recovery after spinal cord injury Synantocytes: the fifth element.Increased chondroitin sulfate proteoglycan expression in denervated brainstem targets following spinal cord injury creates a barrier to axonal regeneration overcome by chondroitinase ABC and neurotrophin-3.Expression of Tgfβ1 and Inflammatory Markers in the 6-hydroxydopamine Mouse Model of Parkinson's Disease.STAT3 and SOCS3 regulate NG2 cell proliferation and differentiation after contusive spinal cord injury.Polydendrocytes: NG2 cells with many roles in development and repair of the CNS.Recognition molecules and neural repair.Permissive Schwann cell graft/spinal cord interfaces for axon regeneration Regeneration and plasticity in the brain and spinal cord.Morphological and physiological interactions of NG2-glia with astrocytes and neurons.Genetic and Pharmacological Inhibition of p38α Improves Locomotor Recovery after Spinal Cord Injury.NG2 cells: Properties, progeny and origin.Roles of NG2 glial cells in diseases of the central nervous system.Glycogen synthase kinase 3 beta (GSK3β) at the tip of neuronal development and regeneration.Manipulating the extracellular matrix and its role in brain and spinal cord plasticity and repair.Glial cells as progenitors and stem cells: new roles in the healthy and diseased brain.Immunoablation of cells expressing the NG2 chondroitin sulphate proteoglycan.Deletion of the Fractalkine Receptor, CX3CR1, Improves Endogenous Repair, Axon Sprouting, and Synaptogenesis after Spinal Cord Injury in Mice.The Glia Response after Peripheral Nerve Injury: A Comparison between Schwann Cells and Olfactory Ensheathing Cells and Their Uses for Neural Regenerative Therapies.The regulatory mechanisms of NG2/CSPG4 expression.Inhibitors of myelination: ECM changes, CSPGs and PTPs.P2X7 receptors and Fyn kinase mediate ATP-induced oligodendrocyte progenitor cell migration.Effects of glutamate receptor activation on NG2-glia in the rat optic nerve.Proliferating NG2 cell-dependent angiogenesis and scar formation alter axon growth and functional recovery after spinal cord injury in mice.

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P2860

NG2: a component of the glial scar that inhibits axon growth.

http://www.wikidata.org/entity/Q36347824

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2005 nî lūn-bûn

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NG2: a component of the glial scar that inhibits axon growth.

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NG2: a component of the glial scar that inhibits axon growth.

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NG2: a component of the glial scar that inhibits axon growth.

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NG2: a component of the glial scar that inhibits axon growth.

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NG2: a component of the glial scar that inhibits axon growth.

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NG2: a component of the glial scar that inhibits axon growth.

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J.1469-7580.2005.00452.X

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Journal of Anatomy

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NG2: a component of the glial scar that inhibits axon growth.

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Andrew M Tan

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Joel M Levine

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Weibing Zhang

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P2860

A novel repeat in the melanoma-associated chondroitin sulfate proteoglycan defines a new protein family

Multiple regions of the NG2 proteoglycan inhibit neurite growth and induce growth cone collapse

Chondroitinase ABC promotes functional recovery after spinal cord injury

The oligodendrocyte precursor cell in health and disease.

Cells expressing the NG2 antigen contact nodes of Ranvier in adult CNS white matter.

A light and electron microscopic study of NG2 chondroitin sulfate proteoglycan-positive oligodendrocyte precursor cells in the normal and kainate-lesioned rat hippocampus.

NG2-expressing glial progenitor cells: an abundant and widespread population of cycling cells in the adult rat CNS.

NG2-expressing cells in the central nervous system: are they oligodendroglial progenitors?

Antisera specific for cell lines with mixed neuronal and glial properties.

Generation of truncated forms of the NG2 proteoglycan by cell surface proteolysis.

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