Impaired social interactions and motor learning skills in tuberous sclerosis complex model mice expressing a dominant/negative form of tuberin
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Clinical and Neurobiological Relevance of Current Animal Models of Autism Spectrum Disorders.Behavioral phenotypes of genetic mouse models of autism.Dendritic spine dysgenesis in autism related disordersSeizure-dependent mTOR activation in 5-HT neurons promotes autism-like behaviors in miceLoss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficitsThe BTBR T+ tf/J mouse model for autism spectrum disorders-in search of biomarkers.Manual versus Automated Rodent Behavioral Assessment: Comparing Efficacy and Ease of Bederson and Garcia Neurological Deficit Scores to an Open Field Video-Tracking System.Selective suppression of excessive GluN2C expression rescues early epilepsy in a tuberous sclerosis murine model.A circuitry and biochemical basis for tuberous sclerosis symptoms: from epilepsy to neurocognitive deficitsMechanism-based treatment in tuberous sclerosis complex.The interaction between early life epilepsy and autistic-like behavioral consequences: a role for the mammalian target of rapamycin (mTOR) pathway.Exaggerated translation causes synaptic and behavioural aberrations associated with autism.Increasing our understanding of human cognition through the study of Fragile X Syndrome.Genetic activation of mTORC1 signaling worsens neurocognitive outcome after traumatic brain injury.Metabotropic glutamate receptor-dependent long-term depression is impaired due to elevated ERK signaling in the ΔRG mouse model of tuberous sclerosis complex.Synaptic plasticity in mouse models of autism spectrum disorders.Ethanol and Caffeine Effects on Social Interaction and Recognition in Mice: Involvement of Adenosine A2A and A1 Receptors.Synaptic plasticity and learning in animal models of tuberous sclerosis complex.Dysregulated mTORC1-Dependent Translational Control: From Brain Disorders to Psychoactive Drugs.Tsc1 haploinsufficiency is sufficient to increase dendritic patterning and Filamin A levels.Conditional and domain-specific inactivation of the Tsc2 gene in neural progenitor cells.Tuber-Less Models of Tuberous Sclerosis Still Provide Insights Into Epilepsy.
P2860
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P2860
Impaired social interactions and motor learning skills in tuberous sclerosis complex model mice expressing a dominant/negative form of tuberin
description
2011 nî lūn-bûn
@nan
2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2011年の論文
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2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Impaired social interactions a ...... inant/negative form of tuberin
@ast
Impaired social interactions a ...... inant/negative form of tuberin
@en
type
label
Impaired social interactions a ...... inant/negative form of tuberin
@ast
Impaired social interactions a ...... inant/negative form of tuberin
@en
prefLabel
Impaired social interactions a ...... inant/negative form of tuberin
@ast
Impaired social interactions a ...... inant/negative form of tuberin
@en
P2093
P2860
P1476
Impaired social interactions a ...... inant/negative form of tuberin
@en
P2093
Emanuela Santini
Eric Klann
Jordan M Maki
P2860
P304
P356
10.1016/J.NBD.2011.07.018
P577
2011-07-30T00:00:00Z