Isolation and characterization of fra-2, an additional member of the fos gene family.
about
Multiple mitogen-activated protein kinase signaling pathways connect the cot oncoprotein to the c-jun promoter and to cellular transformationRedox activation of Fos-Jun DNA binding activity is mediated by a DNA repair enzymeCross-family dimerization of transcription factors Fos/Jun and ATF/CREB alters DNA binding specificityAlteration of a cyclic AMP-dependent protein kinase phosphorylation site in the c-Fos protein augments its transforming potentialIdentification of SWI.SNF complex subunit BAF60a as a determinant of the transactivation potential of Fos/Jun dimersDistinct roles of Jun : Fos and Jun : ATF dimers in oncogenesisThe human involucrin gene contains spatially distinct regulatory elements that regulate expression during early versus late epidermal differentiationSteroid receptor coactivator-1 coactivates activating protein-1-mediated transactivations through interaction with the c-Jun and c-Fos subunitsFos-related antigen (Fra-1), junB, and junD activate human involucrin promoter transcription by binding to proximal and distal AP1 sites to mediate phorbol ester effects on promoter activityTissue-specific transgenic knockdown of Fos-related antigen 2 (Fra-2) expression mediated by dominant negative Fra-2Restraint-induced fra-2 and c-fos expression in the rat forebrain: relationship to stress durationThe ras-related gene rhoB is an immediate-early gene inducible by v-Fps, epidermal growth factor, and platelet-derived growth factor in rat fibroblastsThe Mos/MAP kinase pathway stabilizes c-Fos by phosphorylation and augments its transforming activity in NIH 3T3 cellsLigand-specific sequential regulation of transcription factors for differentiation of MCF-7 cellsMicroarray analysis of Shigella flexneri-infected epithelial cells identifies host factors important for apoptosis inhibitionActivation of junB by PKC and PKA signal transduction through a novel cis-acting elementFra-2 mediates oxygen-sensitive induction of transforming growth factor beta in cardiac fibroblasts.A network of mitogen-activated protein kinases links G protein-coupled receptors to the c-jun promoter: a role for c-Jun NH2-terminal kinase, p38s, and extracellular signal-regulated kinase 5.Cot, a novel kinase of histone H3, induces cellular transformation through up-regulation of c-fos transcriptional activity.miR-597 inhibits breast cancer cell proliferation, migration and invasion through FOSL2.AP1 regulation of proliferation and initiation of apoptosis in erythropoietin-dependent erythroid cellsFra-1 induces morphological transformation and increases in vitro invasiveness and motility of epithelioid adenocarcinoma cells.AP-1 factors play an important role in transformation induced by the v-rel oncogene.Molecular determinants of NOTCH4 transcription in vascular endothelium.AP-1 is a component of the transcriptional network regulated by GSK-3 in quiescent cells.An AP-1 binding site is the predominant cis-acting regulatory element in the 1.2-kilobase early RNA promoter of human cytomegalovirus.JAC, a direct target of oncogenic transcription factor Jun, is involved in cell transformation and tumorigenesisInhibitory effect of AP-1 complex on 5-aminolevulinate synthase gene expression through sequestration of cAMP-response element protein (CRE)-binding protein (CBP) coactivator.Interleukin-12: an update on its immunological activities, signaling and regulation of gene expressionDeterminants of half-site spacing preferences that distinguish AP-1 and ATF/CREB bZIP domainsStructure and mapping of the fosB gene. FosB downregulates the activity of the fosB promoterFRA2 is a STAT5 target gene regulated by IL-2 in human CD4 T cells.Autocrine growth and anchorage independence: two complementing Jun-controlled genetic programs of cellular transformation.Transcription factors activated in mammalian cells after clinically relevant doses of ionizing radiation.Regulation of genetic expression in shear stress-stimulated endothelial cells.A GSK-3-mediated transcriptional network maintains repression of immediate early genes in quiescent cells.Promoter-level expression clustering identifies time development of transcriptional regulatory cascades initiated by ErbB receptors in breast cancer cells.Chondrocytes as a specific target of ectopic Fos expression in early developmentThe cAMP-response-element-binding protein interacts, but Fos protein does not interact, with the proenkephalin enhancer in rat striatum.Induction of apoptosis by c-Fos protein.
P2860
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P2860
Isolation and characterization of fra-2, an additional member of the fos gene family.
description
1990 nî lūn-bûn
@nan
1990 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1990 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
name
Isolation and characterization of fra-2, an additional member of the fos gene family.
@ast
Isolation and characterization of fra-2, an additional member of the fos gene family.
@en
Isolation and characterization of fra-2, an additional member of the fos gene family.
@nl
type
label
Isolation and characterization of fra-2, an additional member of the fos gene family.
@ast
Isolation and characterization of fra-2, an additional member of the fos gene family.
@en
Isolation and characterization of fra-2, an additional member of the fos gene family.
@nl
prefLabel
Isolation and characterization of fra-2, an additional member of the fos gene family.
@ast
Isolation and characterization of fra-2, an additional member of the fos gene family.
@en
Isolation and characterization of fra-2, an additional member of the fos gene family.
@nl
P2093
P2860
P356
P1476
Isolation and characterization of fra-2, an additional member of the fos gene family.
@en
P2093
P2860
P304
P356
10.1073/PNAS.87.9.3619
P407
P577
1990-05-01T00:00:00Z