Inhibition of p38 mitogen-activated protein kinase decreases cardiomyocyte apoptosis and improves cardiac function after myocardial ischemia and reperfusion.
about
Apoptosis in myocardial ischaemia and infarctionEmerging treatment options to improve cardiovascular outcomes in patients with acute coronary syndrome: focus on losmapimodp38 MAPK in cardioprotection - are we there yet?The case for inhibiting p38 mitogen-activated protein kinase in heart failureStress-responsive JNK mitogen-activated protein kinase mediates aspirin-induced suppression of B16 melanoma cellular proliferationMitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale.Angiotensin II reduces infarct size and has no effect on post-ischaemic contractile dysfunction in isolated rat heartsCocaine and apoptosis in myocardial cellsMagnesium Lithospermate B Protects Cardiomyocytes from Ischemic Injury Via Inhibition of TAB1-p38 Apoptosis SignalingBax translocates to mitochondria of heart cells during simulated ischaemia: involvement of AMP-activated and p38 mitogen-activated protein kinasescGMP-dependent protein kinase type I inhibits TAB1-p38 mitogen-activated protein kinase apoptosis signaling in cardiac myocytesp38 MAP kinases in the heartInhibition of p38 MAPK alpha/beta reduces ischemic injury and does not block protective effects of preconditioning.Adenosine-induced late preconditioning in mouse hearts: role of p38 MAP kinase and mitochondrial K(ATP) channels.Inhibition of p38 mitogen-activated protein kinase may decrease intestinal epithelial cell apoptosis and improve intestinal epithelial barrier function after ischemia- reperfusion injury.p38 mitogen-activated protein kinase inhibition decreases TNFalpha secretion and protects against left ventricular remodeling in rats with myocardial ischemia.MAPKAPK-2 modulates p38-MAPK localization and small heat shock protein phosphorylation but does not mediate the injury associated with p38-MAPK activation during myocardial ischemia.Protease-activated receptor 2-mediated protection of myocardial ischemia-reperfusion injury: role of transient receptor potential vanilloid receptors.Electroacupuncture reduces apoptotic index and inhibits p38 mitogen-activated protein kinase signaling pathway in the hippocampus of rats with cerebral ischemia/reperfusion injury.Overexpression of Exosomal Cardioprotective miRNAs Mitigates Hypoxia-Induced H9c2 Cells Apoptosis.Protein Kinases as Drug Development Targets for Heart Disease TherapySirtuin1-p53, forkhead box O3a, p38 and post-infarct cardiac remodeling in the spontaneously diabetic Goto-Kakizaki ratActivation of the AMP-activated protein kinase-p38 MAP kinase pathway mediates apoptosis induced by conjugated linoleic acid in p53-mutant mouse mammary tumor cells.Sulfaphenazole protects heart against ischemia-reperfusion injury and cardiac dysfunction by overexpression of iNOS, leading to enhancement of nitric oxide bioavailability and tissue oxygenation.Thioredoxin-1 gene therapy enhances angiogenic signaling and reduces ventricular remodeling in infarcted myocardium of diabetic ratsTrimetazidine, administered at the onset of reperfusion, ameliorates myocardial dysfunction and injury by activation of p38 mitogen-activated protein kinase and Akt signaling.Inhibition of apoptosis by progesterone in cardiomyocytes.Inhibition of p38 mitogen-activated protein kinase provides neuroprotection in cerebral focal ischemia.cGMP-dependent protein kinases and cGMP phosphodiesterases in nitric oxide and cGMP action.Crosstalk between mitogen-activated protein kinases and mitochondria in cardiac diseases: therapeutic perspectivesProtection of the ischaemic heart: investigations into the phenomenon of ischaemic preconditioning.p38alpha mitogen-activated protein kinase plays a critical role in cardiomyocyte survival but not in cardiac hypertrophic growth in response to pressure overload.Cardioprotective role of P38 MAPK during myocardial infarction via parallel activation of α-crystallin B and Nrf2.14-3-3 proteins block apoptosis and differentially regulate MAPK cascadesRole of mitogen- and stress-activated kinases in ischemic injury.Activation of Mst1 causes dilated cardiomyopathy by stimulating apoptosis without compensatory ventricular myocyte hypertrophy.Anti-apoptotic effect of benidipine, a long-lasting vasodilating calcium antagonist, in ischaemic/reperfused myocardial cells.Differential roles of cardiac and leukocyte derived macrophage migration inhibitory factor in inflammatory responses and cardiac remodelling post myocardial infarction.A p38MAPK/MK2 signaling pathway leading to redox stress, cell death and ischemia/reperfusion injury.Effect of p38 MAP kinases on contractility and ischemic injury in intact heart.
P2860
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P2860
Inhibition of p38 mitogen-activated protein kinase decreases cardiomyocyte apoptosis and improves cardiac function after myocardial ischemia and reperfusion.
description
1999 nî lūn-bûn
@nan
1999 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Inhibition of p38 mitogen-acti ...... dial ischemia and reperfusion.
@ast
Inhibition of p38 mitogen-acti ...... dial ischemia and reperfusion.
@en
type
label
Inhibition of p38 mitogen-acti ...... dial ischemia and reperfusion.
@ast
Inhibition of p38 mitogen-acti ...... dial ischemia and reperfusion.
@en
prefLabel
Inhibition of p38 mitogen-acti ...... dial ischemia and reperfusion.
@ast
Inhibition of p38 mitogen-acti ...... dial ischemia and reperfusion.
@en
P2093
P1433
P1476
Inhibition of p38 mitogen-acti ...... dial ischemia and reperfusion.
@en
P2093
C S Louden
G Z Feuerstein
T A Christopher
P304
P356
10.1161/01.CIR.99.13.1685
P407
P577
1999-04-01T00:00:00Z