A paradoxical reduction in susceptibility to colonic injury upon targeted transgenic ablation of goblet cells
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Aberrant mucin assembly in mice causes endoplasmic reticulum stress and spontaneous inflammation resembling ulcerative colitisTrefoil peptide expression and goblet cell number in rat intestine: effects of KGF and fasting-refeedingAbsence of bacterially induced RELMbeta reduces injury in the dextran sodium sulfate model of colitis.Functional and morphological changes of the gut barrier during the restitution process after hemorrhagic shockCombined NADPH oxidase 1 and interleukin 10 deficiency induces chronic endoplasmic reticulum stress and causes ulcerative colitis-like disease in miceThe zinc-finger transcription factor Klf4 is required for terminal differentiation of goblet cells in the colon.Altered intestinal epithelial homeostasis in mice with intestine-specific deletion of the Krüppel-like factor 4 gene.Getting a grip on things: how do communities of bacterial symbionts become established in our intestine?Colitis-associated variant of TLR2 causes impaired mucosal repair because of TFF3 deficiency.The Endoplasmic Reticulum Stress Sensor IRE1α in Intestinal Epithelial Cells Is Essential for Protecting against Colitis.Transforming growth factor-beta mediates intestinal healing and susceptibility to injury in vitro and in vivo through epithelial cells.Impaired enterocyte proliferation in aquaporin-3 deficiency in mouse models of colitis.Actin reorganization as the molecular basis for the regulation of apoptosis in gastrointestinal epithelial cells.Increased susceptibility to colitis and colorectal tumors in mice lacking core 3-derived O-glycans.Alteration of ocular surface mucins in MUC5AC-DTA transgenic miceGuanylate cyclase C deficiency causes severe inflammation in a murine model of spontaneous colitisEndoplasmic reticulum stress in the intestinal epithelium and inflammatory bowel disease.Endoplasmic reticulum stress and intestinal inflammation.Endoplasmic Reticulum Stress and Oxidative Stress: A Vicious Nexus Implicated in Bowel Disease PathophysiologyExpression of interleukin-9 leads to Th2 cytokine-dominated responses and fatal enteropathy in mice with chronic Schistosoma mansoni infectionsTargeted ablation of glucose-dependent insulinotropic polypeptide-producing cells in transgenic mice reduces obesity and insulin resistance induced by a high fat diet.Disruption of Paneth and goblet cell homeostasis and increased endoplasmic reticulum stress in Agr2-/- mice.Murine guanylate cyclase C regulates colonic injury and inflammation.Resistin-like molecule beta regulates intestinal mucous secretion and curtails TNBS-induced colitis in mice.
P2860
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P2860
A paradoxical reduction in susceptibility to colonic injury upon targeted transgenic ablation of goblet cells
description
1999 nî lūn-bûn
@nan
1999 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
A paradoxical reduction in sus ...... genic ablation of goblet cells
@ast
A paradoxical reduction in sus ...... genic ablation of goblet cells
@en
type
label
A paradoxical reduction in sus ...... genic ablation of goblet cells
@ast
A paradoxical reduction in sus ...... genic ablation of goblet cells
@en
prefLabel
A paradoxical reduction in sus ...... genic ablation of goblet cells
@ast
A paradoxical reduction in sus ...... genic ablation of goblet cells
@en
P2093
P2860
P356
P1476
A paradoxical reduction in sus ...... genic ablation of goblet cells
@en
P2093
P2860
P304
P356
10.1172/JCI6211
P407
P577
1999-12-01T00:00:00Z