Maintaining PGC-1α expression following pressure overload-induced cardiac hypertrophy preserves angiogenesis but not contractile or mitochondrial function.
about
Insulin Signaling and Heart Failure.Cardiomyocyte-specific deficiency of ketone body metabolism promotes accelerated pathological remodeling.Metabolic efficiency promotes protection from pressure overload in hearts expressing slow skeletal troponin I.Enhanced cardiac Akt/protein kinase B signaling contributes to pathological cardiac hypertrophy in part by impairing mitochondrial function via transcriptional repression of mitochondrion-targeted nuclear genes.c-Myc Alters Substrate Utilization and O-GlcNAc Protein Posttranslational Modifications without Altering Cardiac Function during Early Aortic ConstrictionCelecoxib prevents pressure overload-induced cardiac hypertrophy and dysfunction by inhibiting inflammation, apoptosis and oxidative stress.Triptolide Upregulates Myocardial Forkhead Helix Transcription Factor p3 Expression and Attenuates Cardiac Hypertrophy.miR-182 Modulates Myocardial Hypertrophic Response Induced by Angiogenesis in Heart.Exercise leads to unfavourable cardiac remodelling and enhanced metabolic homeostasis in obese mice with cardiac and skeletal muscle autophagy deficiencyDioxygen and Metabolism; Dangerous Liaisons in Cardiac Function and Disease.Peroxisome proliferator-activated receptor-γ coactivator 1 α1 induces a cardiac excitation-contraction coupling phenotype without metabolic remodelling.Moderate Modulation of Cardiac PGC-1α Expression Partially Affects Age-Associated Transcriptional Remodeling of the Heart.
P2860
Q30377910-B278FC5A-59E0-473D-9393-AB7D0E802868Q34404106-32251D9D-0367-415D-8835-7BC5DB86C62CQ35002176-CA93EEA0-02E6-463D-A42B-5E03A65E12B2Q35069561-A0C198A6-8582-46A9-A301-123E1131697CQ35743572-3B27112B-36D4-4372-B233-BC9BDA633807Q36476890-5FAADC2A-C1AA-4AFE-8A50-C71B50A0DD12Q37452461-F6FB0F82-DBF9-4EBF-8AF8-BE04566EC35BQ38542567-917E722E-5895-44AA-AE98-8F84B2FA559BQ41368114-54CE91DC-BC64-4EE7-BC4B-5D6B1860CB77Q49247394-FED328E4-889F-452A-9998-9F44887137EEQ51417816-B34D03FE-4E10-4D19-B598-838DF89D1CBEQ55124445-27138AF8-0A2C-4891-9EA8-C85A670E9013
P2860
Maintaining PGC-1α expression following pressure overload-induced cardiac hypertrophy preserves angiogenesis but not contractile or mitochondrial function.
description
2014 nî lūn-bûn
@nan
2014 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Maintaining PGC-1α expression ...... ile or mitochondrial function.
@ast
Maintaining PGC-1α expression ...... ile or mitochondrial function.
@en
type
label
Maintaining PGC-1α expression ...... ile or mitochondrial function.
@ast
Maintaining PGC-1α expression ...... ile or mitochondrial function.
@en
prefLabel
Maintaining PGC-1α expression ...... ile or mitochondrial function.
@ast
Maintaining PGC-1α expression ...... ile or mitochondrial function.
@en
P2093
P2860
P356
P1433
P1476
Maintaining PGC-1α expression ...... tile or mitochondrial function
@en
P2093
Adam R Wende
Ashley Crum
Christian Riehle
Curtis D Olsen
Douglas Hunter
Renata O Pereira
Tenley Rawlings
Walter F Ward
P2860
P304
P356
10.1096/FJ.14-253823
P407
P50
P577
2014-04-28T00:00:00Z