The hyper-gene conversion hpr5-1 mutation of Saccharomyces cerevisiae is an allele of the SRS2/RADH gene
about
RTEL1 maintains genomic stability by suppressing homologous recombinationDisruption of mechanisms that prevent rereplication triggers a DNA damage responseRequirement for three novel protein complexes in the absence of the Sgs1 DNA helicase in Saccharomyces cerevisiaeMeiotic versus mitotic recombination: two different routes for double-strand break repair: the different functions of meiotic versus mitotic DSB repair are reflected in different pathway usage and different outcomesSrs2 and RecQ homologs cooperate in mei-3-mediated homologous recombination repair of Neurospora crassa.Mechanisms and regulation of mitotic recombination in Saccharomyces cerevisiaeModulation of Saccharomyces cerevisiae DNA double-strand break repair by SRS2 and RAD51.RDH54, a RAD54 homologue in Saccharomyces cerevisiae, is required for mitotic diploid-specific recombination and repair and for meiosisNej1 recruits the Srs2 helicase to DNA double-strand breaks and supports repair by a single-strand annealing-like mechanism.DNA bending facilitates the error-free DNA damage tolerance pathway and upholds genome integrity.Remodeling of the Rad51 DNA strand-exchange protein by the Srs2 helicase.Requirement for the SRS2 DNA helicase gene in non-homologous end joining in yeast.The Saccharomyces cerevisiae RAD9, RAD17, RAD24 and MEC3 genes are required for tolerating irreparable, ultraviolet-induced DNA damage.Choices have consequences: the nexus between DNA repair pathways and genomic instability in cancerThe yeast type I topoisomerase Top3 interacts with Sgs1, a DNA helicase homolog: a potential eukaryotic reverse gyraseFrom yeast to mammals: recent advances in genetic control of homologous recombinationRad52 sumoylation prevents the toxicity of unproductive Rad51 filaments independently of the anti-recombinase Srs2DNA repair mechanisms and the bypass of DNA damage in Saccharomyces cerevisiaeSaccharomyces cerevisiae Rrm3p DNA helicase promotes genome integrity by preventing replication fork stalling: viability of rrm3 cells requires the intra-S-phase checkpoint and fork restart activitiesSrs2 and Sgs1-Top3 suppress crossovers during double-strand break repair in yeastCloning and characterisation of the S. pombe rad15 gene, a homologue to the S. cerevisiae RAD3 and human ERCC2 genes.Stripped-down DNA repair in a highly reduced parasite.Effects of mutations in SGS1 and in genes functionally related to SGS1 on inverted repeat-stimulated spontaneous unequal sister-chromatid exchange in yeastPost-replication repair suppresses duplication-mediated genome instabilitySrs2: the "Odd-Job Man" in DNA repair.Both RAD5-dependent and independent pathways are involved in DNA damage-associated sister chromatid exchange in budding yeast.Suppression of a new allele of the yeast RAD52 gene by overexpression of RAD51, mutations in srs2 and ccr4, or mating-type heterozygosity.Unwinding of synthetic replication and recombination substrates by Srs2.The relative roles in vivo of Saccharomyces cerevisiae Pol eta, Pol zeta, Rev1 protein and Pol32 in the bypass and mutation induction of an abasic site, T-T (6-4) photoadduct and T-T cis-syn cyclobutane dimerThe short life span of Saccharomyces cerevisiae sgs1 and srs2 mutants is a composite of normal aging processes and mitotic arrest due to defective recombinationSrs2 overexpression reveals a helicase-independent role at replication forks that requires diverse cell functionsAlleles of the homologous recombination gene, RAD59, identify multiple responses to disrupted DNA replication in Saccharomyces cerevisiaeThe yeast Shu complex utilizes homologous recombination machinery for error-free lesion bypass via physical interaction with a Rad51 paralogue.Epistasis analysis between homologous recombination genes in Saccharomyces cerevisiae identifies multiple repair pathways for Sgs1, Mus81-Mms4 and RNase H2Multiple regulation of Rad51-mediated homologous recombination by fission yeast Fbh1.Evolutionary and functional analysis of the invariant SWIM domain in the conserved Shu2/SWS1 protein family from Saccharomyces cerevisiae to Homo sapiens.Regulation of DNA pairing in homologous recombination.Postreplication repair inhibits CAG.CTG repeat expansions in Saccharomyces cerevisiae.The human F-Box DNA helicase FBH1 faces Saccharomyces cerevisiae Srs2 and postreplication repair pathway roles.Mrc1 is required for sister chromatid cohesion to aid in recombination repair of spontaneous damage.
P2860
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P2860
The hyper-gene conversion hpr5-1 mutation of Saccharomyces cerevisiae is an allele of the SRS2/RADH gene
description
1991 nî lūn-bûn
@nan
1991 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1991 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
name
The hyper-gene conversion hpr5 ...... n allele of the SRS2/RADH gene
@ast
The hyper-gene conversion hpr5 ...... n allele of the SRS2/RADH gene
@en
type
label
The hyper-gene conversion hpr5 ...... n allele of the SRS2/RADH gene
@ast
The hyper-gene conversion hpr5 ...... n allele of the SRS2/RADH gene
@en
prefLabel
The hyper-gene conversion hpr5 ...... n allele of the SRS2/RADH gene
@ast
The hyper-gene conversion hpr5 ...... n allele of the SRS2/RADH gene
@en
P2093
P2860
P1433
P1476
The hyper-gene conversion hpr5 ...... n allele of the SRS2/RADH gene
@en
P2093
P2860
P407
P577
1991-01-01T00:00:00Z