Histopathological analysis of renal cystic epithelia in the Pkd2WS25/- mouse model of ADPKD. - Wikidata - awgldk - TriplyDB

Histopathological analysis of renal cystic epithelia in the Pkd2WS25/- mouse model of ADPKD.

Histopathological analysis of renal cystic epithelia in the Pkd2WS25/- mouse model of ADPKD.

http://www.wikidata.org/entity/Q34212778

about

Pericentrin forms a complex with intraflagellar transport proteins and polycystin-2 and is required for primary cilia assembly Role of PDZK1 in membrane expression of renal brush border ion exchangers.Novel role of ouabain as a cystogenic factor in autosomal dominant polycystic kidney disease Disruption of Myosin 1e promotes podocyte injury Regulation of anion exchanger Slc26a6 by protein kinase C Acceleration of polycystic kidney disease progression in cpk mice carrying a deletion in the homeodomain protein Cux1 Zebrafish mutations affecting cilia motility share similar cystic phenotypes and suggest a mechanism of cyst formation that differs from pkd2 morphants.Hepato-renal pathology in pkd2ws25/- mice, an animal model of autosomal dominant polycystic kidney disease Cyst formation and activation of the extracellular regulated kinase pathway after kidney specific inactivation of Pkd1.The C-terminal tail of the polycystin-1 protein interacts with the Na,K-ATPase alpha-subunit.The fate of bone marrow-derived cells carrying a Polycystic Kidney Disease mutation in the genetically normal kidney Cyclic AMP-mediated cyst expansion Antisense-mediated angiotensinogen inhibition slows polycystic kidney disease in mice with a targeted mutation in Pkd2 Tolvaptan inhibits ERK-dependent cell proliferation, Cl⁻ secretion, and in vitro cyst growth of human ADPKD cells stimulated by vasopressin Altered renal proximal tubular endocytosis and histology in mice lacking myosin-VI.Calmodulin-sensitive adenylyl cyclases mediate AVP-dependent cAMP production and Cl- secretion by human autosomal dominant polycystic kidney cells.Human ADPKD primary cyst epithelial cells with a novel, single codon deletion in the PKD1 gene exhibit defective ciliary polycystin localization and loss of flow-induced Ca2+ signaling.TRPpathies.Tight junction composition is altered in the epithelium of polycystic kidneys.Transcriptome analysis reveals manifold mechanisms of cyst development in ADPKD.Mouse models of polycystic kidney disease induced by defects of ciliary proteins Haploinsufficiency of Pkd2 is associated with increased tubular cell proliferation and interstitial fibrosis in two murine Pkd2 models.The zebrafish Kupffer's vesicle as a model system for the molecular mechanisms by which the lack of Polycystin-2 leads to stimulation of CFTR.Pkd1 regulates immortalized proliferation of renal tubular epithelial cells through p53 induction and JNK activation.Immunolocalization of NHE8 in rat kidney.Cux1 promotes cell proliferation and polycystic kidney disease progression in an ADPKD mouse model.

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Histopathological analysis of renal cystic epithelia in the Pkd2WS25/- mouse model of ADPKD.

http://www.wikidata.org/entity/Q34212778

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2003 nî lūn-bûn

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2003 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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Histopathological analysis of ...... d2WS25/- mouse model of ADPKD.

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Histopathological analysis of ...... d2WS25/- mouse model of ADPKD.

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AJPRENAL.00153.2003

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American Journal of Physiology - Renal Physiology

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Histopathological analysis of ...... d2WS25/- mouse model of ADPKD.

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Eric Delpire

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Karen Earle

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Peter S Aronson

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R Brent Thomson

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Robert Kim

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Stefan Somlo

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SueAnn Mentone

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P2860

Polycystins 1 and 2 mediate mechanosensation in the primary cilium of kidney cells

Cardiac defects and renal failure in mice with targeted mutations in Pkd2

Chlamydomonas IFT88 and its mouse homologue, polycystic kidney disease gene tg737, are required for assembly of cilia and flagella

Water channel expression in human ADPKD kidneys

The Caenorhabditis elegans autosomal dominant polycystic kidney disease gene homologs lov-1 and pkd-2 act in the same pathway

Active (9.6 s) and inactive (21 s) oligomers of NHE3 in microdomains of the renal brush border.

Genetics and pathogenesis of polycystic kidney disease.

In vitro fluid secretion by epithelium from polycystic kidneys.

Apical plasma membrane mispolarization of NaK-ATPase in polycystic kidney disease epithelia is associated with aberrant expression of the beta2 isoform.

Compromised cytoarchitecture and polarized trafficking in autosomal dominant polycystic kidney disease cells.

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