Abnormalities in cell proliferation and apico-basal cell polarity are separable in Drosophila lgl mutant clones in the developing eye. - Wikidata - awgldk - TriplyDB

Abnormalities in cell proliferation and apico-basal cell polarity are separable in Drosophila lgl mutant clones in the developing eye.

Abnormalities in cell proliferation and apico-basal cell polarity are separable in Drosophila lgl mutant clones in the developing eye.

http://www.wikidata.org/entity/Q34283595

about

Involvement of Lgl and Mahjong/VprBP in cell competition Cell competition: how to eliminate your neighbours The lethal giant larvae tumour suppressor mutation requires dMyc oncoprotein to promote clonal malignancy.An apicobasal gradient of Rac activity determines protrusion form and position Drosophila as a model for context-dependent tumorigenesis.Loss of the Drosophila cell polarity regulator Scribbled promotes epithelial tissue overgrowth and cooperation with oncogenic Ras-Raf through impaired Hippo pathway signaling Hugl1 and Hugl2 in mammary epithelial cells: polarity, proliferation, and differentiation.Hippo signaling: growth control and beyond.Drosophila Myc: A master regulator of cellular performance.VprBP (DCAF1): a promiscuous substrate recognition subunit that incorporates into both RING-family CRL4 and HECT-family EDD/UBR5 E3 ubiquitin ligases Identification of novel Ras-cooperating oncogenes in Drosophila melanogaster: a RhoGEF/Rho-family/JNK pathway is a central driver of tumorigenesis.Loss of Llgl1 in retinal neuroepithelia reveals links between apical domain size, Notch activity and neurogenesis.Cooperation of the BTB-Zinc finger protein, Abrupt, with cytoskeletal regulators in Drosophila epithelial tumorigenesis.Tumor suppressor protein Lgl mediates G1 cell cycle arrest at high cell density by forming an Lgl-VprBP-DDB1 complex.Regulation of Notch signaling and endocytosis by the Lgl neoplastic tumor suppressor.Cell Competition Drives the Growth of Intestinal Adenomas in Drosophila.Mechanisms of cell competition: themes and variations.ALFY-Controlled DVL3 Autophagy Regulates Wnt Signaling, Determining Human Brain Size.Epithelial neoplasia in Drosophila entails switch to primitive cell states Cell wars: regulation of cell survival and proliferation by cell competition Control of tumourigenesis by the Scribble/Dlg/Lgl polarity module.Tissue repair through cell competition and compensatory cellular hypertrophy in postmitotic epithelia.Cell competition and its implications for development and cancer.Drosophila lethal giant larvae neoplastic mutant as a genetic tool for cancer modeling.Troponin-I enhances and is required for oncogenic overgrowth Dissecting social cell biology and tumors using Drosophila genetics.Lgl/aPKC and Crb regulate the Salvador/Warts/Hippo pathway The flower code and cancer development.Interface between normal and transformed epithelial cells: a road to a novel type of cancer prevention and treatment.The Hippo pathway in organ size control, tissue regeneration and stem cell self-renewal.Drosophila cancer models.Hippo signaling in Drosophila: recent advances and insights Apicobasal polarity and cell proliferation during development.The Scribble-Dlg-Lgl polarity module in development and cancer: from flies to man.Hippo gains weight: added insights and complexity to pathway control.Lethal (2) giant larvae: an indispensable regulator of cell polarity and cancer development.Oncogenic Ras diverts a host TNF tumor suppressor activity into tumor promoter.Epithelial self-defense against cancer.Loss of Scribble causes cell competition in mammalian cells.A Kinome RNAi Screen in Drosophila Identifies Novel Genes Interacting with Lgl, aPKC, and Crb Cell Polarity Genes in Epithelial Tissues.

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P2860

Abnormalities in cell proliferation and apico-basal cell polarity are separable in Drosophila lgl mutant clones in the developing eye.

http://www.wikidata.org/entity/Q34283595

description

2007 nî lūn-bûn

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2007 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2007 թվականի օգոստոսին հրատարակված գիտական հոդված

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2007年の論文

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Abnormalities in cell prolifer ...... clones in the developing eye.

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Abnormalities in cell prolifer ...... clones in the developing eye.

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Abnormalities in cell prolifer ...... clones in the developing eye.

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Abnormalities in cell prolifer ...... clones in the developing eye.

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Abnormalities in cell prolifer ...... clones in the developing eye.

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Abnormalities in cell prolifer ...... clones in the developing eye.

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Abnormalities in cell prolifer ...... clones in the developing eye.

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J.YDBIO.2007.08.025

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Developmental Biology

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Abnormalities in cell prolifer ...... t clones in the developing eye

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P2093

Anthony M Brumby

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Julie Secombe

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Nancy Amin

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Nicola A Grzeschik

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P2860

Human homolog of Drosophila tumor suppressor Scribble negatively regulates cell-cycle progression from G1 to S phase by localizing at the basolateral membrane in epithelial cells

Dlg, Scribble and Lgl in cell polarity, cell proliferation and cancer

Drosophila Crumbs is a positional cue in photoreceptor adherens junctions and rhabdomeres

The tumour-suppressor Scribble dictates cell polarity during directed epithelial migration: regulation of Rho GTPase recruitment to the leading edge

Requirement of PDZ-containing proteins for cell cycle regulation and differentiation in the mouse lens epithelium

Loss of cell polarity causes severe brain dysplasia in Lgl1 knockout mice

Analysis of genetic mosaics in developing and adult Drosophila tissues

Organizing a functional junctional complex requires specific domains of the Drosophila MAGUK Discs large.

Dlg protein is required for junction structure, cell polarity, and proliferation control in Drosophila epithelia

aPKCzeta cortical loading is associated with Lgl cytoplasmic release and tumor growth in Drosophila and human epithelia.

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106-123

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10.1016/J.YDBIO.2007.08.025

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1

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Helena E. Richardson

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5970921

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2974846

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