Cholesterol and breast cancer pathophysiology. - Wikidata - awgldk - TriplyDB

Cholesterol and breast cancer pathophysiology.

Cholesterol and breast cancer pathophysiology.

http://www.wikidata.org/entity/Q34715261

about

Lipid-sensors, enigmatic-orphan and orphan nuclear receptors as therapeutic targets in breast-cancer Identification of CETP as a molecular target for estrogen positive breast cancer cell death by cholesterol depleting agents Identifying gene-gene interactions that are highly associated with four quantitative lipid traits across multiple cohorts.Targeting Intracellular Cholesterol is a Novel Therapeutic Strategy for Cancer Treatment.Serum Lipids and Breast Cancer Risk: A Meta-Analysis of Prospective Cohort Studies.Cholestenoic acid, an endogenous cholesterol metabolite, is a potent γ-secretase modulator.Transcriptomic analysis of pancreatic cancer cells in response to metformin and aspirin: an implication of synergy.ABL kinases promote breast cancer osteolytic metastasis by modulating tumor-bone interactions through TAZ and STAT5 signaling.Cardiovascular Risk and Level of Statin Use Among Women With Breast Cancer in a Cardio-Oncology Clinic Selective Reversible Inhibition of Autophagy in Hypoxic Breast Cancer Cells Promotes Pulmonary Metastasis.The Contribution of Cholesterol and Its Metabolites to the Pathophysiology of Breast Cancer Actinidia chinensis Planch root extract inhibits cholesterol metabolism in hepatocellular carcinoma through upregulation of PCSK9.The link between lipids, statins and cancer: is there a role for cardio-oncology?Circulating Lipoproteins: A Trojan Horse Guiding Squalenoylated Drugs to LDL-Accumulating Cancer Cells.Impact of Statin Use on Outcomes in Triple Negative Breast Cancer The cholesterol metabolite 27 hydroxycholesterol facilitates breast cancer metastasis through its actions on immune cells.Drug-induced reactive oxygen species (ROS) rely on cell membrane properties to exert anticancer effects.Dietary Factors and Female Breast Cancer Risk: A Prospective Cohort Study.The significance of cholesterol and its metabolite, 27-hydroxycholesterol in breast cancer.27-Hydroxycholesterol, an endogenous selective estrogen receptor modulator.Preliminary evaluation of serum total cholesterol concentrations in dogs with osteosarcoma.The Divergent Function of Androgen Receptor in Breast Cancer; Analysis of Steroid Mediators and Tumor Intracrinology Metabolic Syndrome and the Risk of Breast Cancer and Subtypes by Race, Menopause and BMI A Mendelian randomization study of the effects of blood lipids on breast cancer risk

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P2860

Cholesterol and breast cancer pathophysiology.

http://www.wikidata.org/entity/Q34715261

description

2014 nî lūn-bûn

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2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2014 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Cholesterol and breast cancer pathophysiology.

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Cholesterol and breast cancer pathophysiology.

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Cholesterol and breast cancer pathophysiology.

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Cholesterol and breast cancer pathophysiology.

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Cholesterol and breast cancer pathophysiology.

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Cholesterol and breast cancer pathophysiology.

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Cholesterol and breast cancer pathophysiology.

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Cholesterol and breast cancer pathophysiology.

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Cholesterol and breast cancer pathophysiology.

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J.TEM.2014.10.001

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Trends in Endocrinology and Metabolism

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Cholesterol and breast cancer pathophysiology.

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Ching-yi Chang

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Donald P McDonnell

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P2860

LXR regulates cholesterol uptake through Idol-dependent ubiquitination of the LDL receptor

Sterol-regulated transport of SREBPs from endoplasmic reticulum to Golgi: oxysterols block transport by binding to Insig

Feedback regulation of cholesterol uptake by the LXR-IDOL-LDLR axis

Cholestenoic Acid Is a Naturally Occurring Ligand for Liver X Receptor α

Structure of the human gene encoding sterol regulatory element binding protein-1 (SREBF1) and localization of SREBF1 and SREBF2 to chromosomes 17p11.2 and 22q13

The oxysterol-CXCR2 axis plays a key role in the recruitment of tumor-promoting neutrophils

Transcriptomic classification of genetically engineered mouse models of breast cancer identifies human subtype counterparts.

Mammary tumor growth and pulmonary metastasis are enhanced in a hyperlipidemic mouse model.

The cholesterol metabolite 25-hydroxycholesterol activates estrogen receptor α-mediated signaling in cancer cells and in cardiomyocytes

Hypercholesterolemia induces angiogenesis and accelerates growth of breast tumors in vivo.

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649-655

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scholarly article

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10.1016/J.TEM.2014.10.001

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12

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2014-11-04T00:00:00Z

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pathophysiology

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