Aberrant V(D)J recombination in ataxia telangiectasia mutated-deficient lymphocytes is dependent on nonhomologous DNA end joining.
about
Lymphoid tumours and breast cancer in ataxia telangiectasia; substantial protective effect of residual ATM kinase activity against childhood tumoursAtaxia telangiectasia: a reviewATM limits incorrect end utilization during non-homologous end joining of multiple chromosome breaksMolecular allelokaryotyping of T-cell prolymphocytic leukemia cells with high density single nucleotide polymorphism arrays identifies novel common genomic lesions and acquired uniparental disomyDNA double-strand break signaling and human disorders.Correct end use during end joining of multiple chromosomal double strand breaks is influenced by repair protein RAD50, DNA-dependent protein kinase DNA-PKcs, and transcription context.Redundant and nonredundant functions of ATM and H2AX in αβ T-lineage lymphocytes.Faithful after break-up: suppression of chromosomal translocations.Restoration of ATM Expression in DNA-PKcs-Deficient Cells Inhibits Signal End Joining.MRN complex function in the repair of chromosomal Rag-mediated DNA double-strand breaksAccessibility of chromosomal recombination breaks in nuclei of wild-type and DNA-PKcs-deficient cells.53BP1 is limiting for NHEJ repair in ATM-deficient model systems that are subjected to oncogenic stress or radiation.V(D)J Recombination: Mechanism, Errors, and Fidelity.Repair of chromosomal RAG-mediated DNA breaks by mutant RAG proteins lacking phosphatidylinositol 3-like kinase consensus phosphorylation sites.Breaks invisible to the DNA damage response machinery accumulate in ATM-deficient cells.Tia1 dependent regulation of mRNA subcellular location and translation controls p53 expression in B cells.A hypomorphic Artemis human disease allele causes aberrant chromosomal rearrangements and tumorigenesis.Presence of ATM protein and residual kinase activity correlates with the phenotype in ataxia-telangiectasia: a genotype-phenotype study.ATM and p53 are essential in the cell-cycle containment of DNA breaks during V(D)J recombination in vivo.
P2860
Q24597674-4239F278-51C7-485A-8571-0DA9487F1836Q28079581-18E198C3-190C-4327-B62E-EEC80AC5C7ACQ28476006-7C84290C-9C84-4609-A1B0-746784CAD672Q33417186-6E9F232A-6971-468E-8AB6-CA8247CEE9D2Q34360500-780248BF-53C4-468D-A722-5040435D464FQ35604928-68F0F5F0-38FB-46A8-9C1E-3C12363E3720Q36106641-93B3C325-9A0A-41D0-99C5-5914D4A4098DQ36410982-E9DAE0FC-5CE7-43A0-96C5-A6DDF85E8080Q36706287-4DFEBD75-7214-4C38-8D01-226EE420C24EQ37234327-6ACC0147-8399-41A4-BDF9-E7D770156B83Q37243923-923393D1-5E93-4524-B472-53C774D447CAQ37684399-4A200B42-2DBF-45F6-8AE7-2FEFAFE53FFCQ38535955-DC10595A-00AA-447C-9205-92C0A4366090Q39509534-2BAB8B03-4083-4690-807A-CC9A2F53C68CQ39849267-22B45BA4-EF78-4AEA-AED3-3BC9A6F667B3Q41126970-8E9A20AC-D33D-4C74-B47A-D4A27B4A91CAQ42146685-AD5B61E3-383F-4089-9F5C-17A5BD41EDDFQ43809547-CF825976-D35E-4A46-9102-9BBA23BAE490Q53359826-E66FBB23-D4CD-47B1-98B3-ABD6C76B92B9
P2860
Aberrant V(D)J recombination in ataxia telangiectasia mutated-deficient lymphocytes is dependent on nonhomologous DNA end joining.
description
2008 nî lūn-bûn
@nan
2008 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Aberrant V(D)J recombination i ...... nonhomologous DNA end joining.
@ast
Aberrant V(D)J recombination i ...... nonhomologous DNA end joining.
@en
type
label
Aberrant V(D)J recombination i ...... nonhomologous DNA end joining.
@ast
Aberrant V(D)J recombination i ...... nonhomologous DNA end joining.
@en
prefLabel
Aberrant V(D)J recombination i ...... nonhomologous DNA end joining.
@ast
Aberrant V(D)J recombination i ...... nonhomologous DNA end joining.
@en
P2093
P2860
P1476
Aberrant V(D)J recombination i ...... nonhomologous DNA end joining
@en
P2093
Andrea L Bredemeyer
Barry P Sleckman
Craig H Bassing
Laura M Walker
P2860
P304
P356
10.4049/JIMMUNOL.181.4.2620
P407
P577
2008-08-01T00:00:00Z