Defects in a cell cycle checkpoint may be responsible for the genomic instability of cancer cells.
about
The human G2 checkpoint control protein hRAD9 is a nuclear phosphoprotein that forms complexes with hRAD1 and hHUS1Interaction of p53 with the human Rad51 proteinECA39, a conserved gene regulated by c-Myc in mice, is involved in G1/S cell cycle regulation in yeastA human homologue of the Schizosaccharomyces pombe rad1+ checkpoint gene encodes an exonucleaseIdentification of a human homologue of the Schizosaccharomyces pombe rad17+ checkpoint geneHEC, a novel nuclear protein rich in leucine heptad repeats specifically involved in mitosisInhibition of p53 DNA binding by human papillomavirus E6 proteinsCharacterization of a novel 350-kilodalton nuclear phosphoprotein that is specifically involved in mitotic-phase progressionIdentification of an overlapping binding domain on Cdc20 for Mad2 and anaphase-promoting complex: model for spindle checkpoint regulationWidely dispersed p53 mutation in respiratory epithelium. A novel mechanism for field carcinogenesiscDNA cloning and gene mapping of a candidate human cell cycle checkpoint proteinAdaptive mutation: the uses of adversityHuman papillomavirus 16 E6 oncoprotein binds to interferon regulatory factor-3 and inhibits its transcriptional activityA DNA damage and stress inducible G protein-coupled receptor blocks cells in G2/MCytoplasmic p53 polypeptide is associated with ribosomesTransforming growth factor beta stabilizes p15INK4B protein, increases p15INK4B-cdk4 complexes, and inhibits cyclin D1-cdk4 association in human mammary epithelial cellsTransforming growth factor-beta and breast cancer: Cell cycle arrest by transforming growth factor-beta and its disruption in cancerHuman papilloma viruses and cervical tumours: mapping of integration sites and analysis of adjacent cellular sequencesPossible attenuation of the G2 DNA damage cell cycle checkpoint in HeLa cells by extremely low frequency (ELF) electromagnetic fieldsDifferential role of RB in response to UV and IR damage.Deregulation of p27 by oncogenic signaling and its prognostic significance in breast cancerMyosins as fundamental components during tumorigenesis: diverse and indispensableStem cells, senescence, neosis and self-renewal in cancer.Crystal structure of the tetramerization domain of the p53 tumor suppressor at 1.7 angstromsNFBD1, a novel nuclear protein with signature motifs of FHA and BRCT, and an internal 41-amino acid repeat sequence, is an early participant in DNA damage responseATR functions as a gene dosage-dependent tumor suppressor on a mismatch repair-deficient backgroundCHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulumIdentification of the Lipodepsipeptide MDN-0066, a Novel Inhibitor of VHL/HIF Pathway Produced by a New Pseudomonas SpeciesPersistent nicotine treatment potentiates amplification of the dihydrofolate reductase gene in rat lung epithelial cells as a consequence of Ras activationGrowth retardation, polyploidy, and multinucleation induced by Clast3, a novel cell cycle-regulated proteinCyclin G is a transcriptional target of the p53 tumor suppressor proteinA PLAC8-containing protein from an endomycorrhizal fungus confers cadmium resistance to yeast cells by interacting with Mlh3pAPC/C--the master controller of origin licensing?Aneuploidy vs. gene mutation hypothesis of cancer: recent study claims mutation but is found to support aneuploidy.Autocrine human growth hormone (hGH) regulation of human mammary carcinoma cell gene expression. Identification of CHOP as a mediator of hGH-stimulated human mammary carcinoma cell survival.Prognostic value of p53 mutations, bax, and spontaneous apoptosis in maxillary sinus squamous cell carcinoma.Physical and functional interactions between nucleotide excision repair and DNA damage checkpoint.NSAIDs modulate clonal evolution in Barrett's esophagus.Reversible inactivation of the transcriptional function of P53 protein by farnesylation.Nuclear hBD-1 accumulation in malignant salivary gland tumours
P2860
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P2860
Defects in a cell cycle checkpoint may be responsible for the genomic instability of cancer cells.
description
1992 nî lūn-bûn
@nan
1992 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1992 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
name
Defects in a cell cycle checkp ...... c instability of cancer cells.
@ast
Defects in a cell cycle checkp ...... c instability of cancer cells.
@en
type
label
Defects in a cell cycle checkp ...... c instability of cancer cells.
@ast
Defects in a cell cycle checkp ...... c instability of cancer cells.
@en
prefLabel
Defects in a cell cycle checkp ...... c instability of cancer cells.
@ast
Defects in a cell cycle checkp ...... c instability of cancer cells.
@en
P1433
P1476
Defects in a cell cycle checkp ...... c instability of cancer cells.
@en
P2093
Hartwell L
P304
P356
10.1016/0092-8674(92)90586-2
P407
P577
1992-11-01T00:00:00Z