Transcription of human papillomavirus type 16 early genes in a cervical cancer and a cancer-derived cell line and identification of the E7 protein.
about
Inactivation of p53 rescues the maintenance of high risk HPV DNA genomes deficient in expression of E6Papillomavirus genome structure, expression, and post-transcriptional regulationThe region of the HPV E7 oncoprotein homologous to adenovirus E1a and Sv40 large T antigen contains separate domains for Rb binding and casein kinase II phosphorylationRole of the PDZ domain-binding motif of the oncoprotein E6 in the pathogenesis of human papillomavirus type 31Purification and characterization of human papillomavirus type 16 E7 protein with preferential binding capacity to the underphosphorylated form of retinoblastoma gene productActivation of p53 in cervical carcinoma cells by small molecules.Binding of high-risk human papillomavirus E6 oncoproteins to the human homologue of the Drosophila discs large tumor suppressor proteinDifferential localization of HPV16 E6 splice products with E6-associated protein.Human Papillomavirus (HPV)-associated Oral Cancers and Treatment StrategiesFunctional Roles of E6 and E7 Oncoproteins in HPV-Induced Malignancies at Diverse Anatomical SitesHPV16 E6 and E7 proteins cooperate to immortalize human foreskin keratinocytesThe regulatory element in the 3'-untranslated region of human papillomavirus 16 inhibits expression by binding CUG-binding protein 1The current status of therapeutic HPV vaccineTopography of early HPV 16 transcription in high-grade genital precancers.Pathobiology of papillomavirus-related cervical diseases: prospects for immunodiagnosis.Multiple-integrations of HPV16 genome and altered transcription of viral oncogenes and cellular genes are associated with the development of cervical cancerNonradioactive RNA in situ hybridization detection of human papillomavirus 16-E7 transcripts in squamous cell carcinomas of the uterine cervix using confocal laser scan microscopy.Interaction of the human papillomavirus type 16 E6 oncoprotein with wild-type and mutant human p53 proteinsHuman papillomavirus type 16 E7 binds to E2F1 and activates E2F1-driven transcription in a retinoblastoma protein-independent manner.Human papillomavirus-16 E7 interacts with glutathione S-transferase P1 and enhances its role in cell survivalContinued expression of HPV-16 E7 protein is required for maintenance of the transformed phenotype of cells co-transformed by HPV-16 plus EJ-ras.Complex formation of human papillomavirus E7 proteins with the retinoblastoma tumor suppressor gene product.Multiple ASF/SF2 sites in the human papillomavirus type 16 (HPV-16) E4-coding region promote splicing to the most commonly used 3'-splice site on the HPV-16 genome.Association of Human Papilloma Virus 16 Infection and p53 Polymorphism among Tobacco using Oral Leukoplakia Patients: A Clinicopathologic and Genotypic Study.Depletion of the cdk inhibitor p16INK4a differentially affects proliferation of established cervical carcinoma cells.Human papillomavirus type 16 alters human epithelial cell differentiation in vitro.Human papillomavirus type 16 E6 increases the degradation rate of p53 in human keratinocytesOverexpression of C/EBPbeta represses human papillomavirus type 18 upstream regulatory region activity in HeLa cells by interfering with the binding of TATA-binding protein.Transactivation by the E2 protein of oncogenic human papillomavirus type 31 is not essential for early and late viral functions.The role of the E6-p53 interaction in the molecular pathogenesis of HPV.Papillomavirus capsid protein expression level depends on the match between codon usage and tRNA availability.A synthetic peptide defines a serologic IgA response to a human papillomavirus-encoded nuclear antigen expressed in virus-carrying cervical neoplasia.Molecular mechanism of carcinogenesis by human papillomavirus-16.Identification of early proteins of the human papilloma viruses type 16 (HPV 16) and type 18 (HPV 18) in cervical carcinoma cells.Identification of the HPV-16 E6 protein from transformed mouse cells and human cervical carcinoma cell lines.Human papillomavirus type 16 DNA cooperates with activated ras in transforming primary cells.The upstream regulatory region of the human papilloma virus-16 contains an E2 protein-independent enhancer which is specific for cervical carcinoma cells and regulated by glucocorticoid hormones.Transcriptional regulation of the human papillomavirus-16 E6-E7 promoter by a keratinocyte-dependent enhancer, and by viral E2 trans-activator and repressor gene products: implications for cervical carcinogenesis.The E6 and E7 genes of human papillomavirus type 6 have weak immortalizing activity in human epithelial cells.Structure-function analysis of the human papillomavirus type 16 E7 oncoprotein.
P2860
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P2860
Transcription of human papillomavirus type 16 early genes in a cervical cancer and a cancer-derived cell line and identification of the E7 protein.
description
1986 nî lūn-bûn
@nan
1986年の論文
@ja
1986年論文
@yue
1986年論文
@zh-hant
1986年論文
@zh-hk
1986年論文
@zh-mo
1986年論文
@zh-tw
1986年论文
@wuu
1986年论文
@zh
1986年论文
@zh-cn
name
Transcription of human papillo ...... ntification of the E7 protein.
@ast
Transcription of human papillo ...... ntification of the E7 protein.
@en
type
label
Transcription of human papillo ...... ntification of the E7 protein.
@ast
Transcription of human papillo ...... ntification of the E7 protein.
@en
prefLabel
Transcription of human papillo ...... ntification of the E7 protein.
@ast
Transcription of human papillo ...... ntification of the E7 protein.
@en
P2860
P356
P1476
Transcription of human papillo ...... ntification of the E7 protein.
@en
P2093
F O Wettstein
P2860
P304
P356
10.1073/PNAS.83.13.4680
P407
P577
1986-07-01T00:00:00Z