Comparative oncogenomics identifies breast tumors enriched in functional tumor-initiating cells.
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Expression profiling of cancerous and normal breast tissues identifies microRNAs that are differentially expressed in serum from patients with (metastatic) breast cancer and healthy volunteersTargeting serous epithelial ovarian cancer with designer zinc finger transcription factorsThe mammary stem cell hierarchy: a looking glass into heterogeneous breast cancer landscapesBreast cancer epithelial-to-mesenchymal transition: examining the functional consequences of plasticityIdentifying and targeting tumor-initiating cells in the treatment of breast cancerEmerging role of cell polarity proteins in breast cancer progression and metastasisMicroRNAs: regulators of cancer metastasis and epithelial-mesenchymal transition (EMT)Obesity, metabolic dysregulation, and cancer: a growing concern and an inflammatory (and microenvironmental) issueThe miR-200 and miR-221/222 microRNA families: opposing effects on epithelial identityPy2T murine breast cancer cells, a versatile model of TGFβ-induced EMT in vitro and in vivoDerivation of myoepithelial progenitor cells from bipotent mammary stem/progenitor cellsWnt-responsive cancer stem cells are located close to distorted blood vessels and not in hypoxic regions in a p53-null mouse model of human breast cancer.Prolactin cooperates with loss of p53 to promote claudin-low mammary carcinomasRe-expression of miR-200c suppresses proliferation, colony formation and in vivo tumor growth of murine claudin-low mammary tumor cells.Specificity protein 1 (Sp1) maintains basal epithelial expression of the miR-200 family: implications for epithelial-mesenchymal transitionTranscriptomic classification of genetically engineered mouse models of breast cancer identifies human subtype counterparts.Comparison of RNA-Seq by poly (A) capture, ribosomal RNA depletion, and DNA microarray for expression profiling.A genomic analysis of mouse models of breast cancer reveals molecular features of mouse models and relationships to human breast cancer.Biocompatible nanopolymers: the next generation of breast cancer treatment?A mouse model for triple-negative breast cancer tumor-initiating cells (TNBC-TICs) exhibits similar aggressive phenotype to the human diseaseEMT inducers catalyze malignant transformation of mammary epithelial cells and drive tumorigenesis towards claudin-low tumors in transgenic mice.Claudin-low breast cancers: clinical, pathological, molecular and prognostic characterizationMouse p53-deficient cancer models as platforms for obtaining genomic predictors of human cancer clinical outcomesMicroRNA-200 family modulation in distinct breast cancer phenotypes.Genome-wide profiles of CtBP link metabolism with genome stability and epithelial reprogramming in breast cancer.Distinct luminal-type mammary carcinomas arise from orthotopic Trp53-null mammary transplantation of juvenile versus adult mice.A genome wide meta-analysis study for identification of common variation associated with breast cancer prognosis.C-Terminal Binding Protein: A Molecular Link between Metabolic Imbalance and Epigenetic Regulation in Breast Cancer.Combined deletion of Pten and p53 in mammary epithelium accelerates triple-negative breast cancer with dependency on eEF2KProfiling the tyrosine phosphoproteome of different mouse mammary tumour models reveals distinct, model-specific signalling networks and conserved oncogenic pathways.The osteogenic niche promotes early-stage bone colonization of disseminated breast cancer cells.BCL11A is a triple-negative breast cancer gene with critical functions in stem and progenitor cells.Fibromatosis-like metaplastic carcinoma of the breast has a claudin-low immunohistochemical phenotype.DNA damage and breast cancerMouse models of estrogen receptor-positive breast cancerCytokeratin 5/6 fingerprinting in HER2-positive tumors identifies a poor prognosis and trastuzumab-resistant basal-HER2 subtype of breast cancer.Poly(2-oxazoline) based micelles with high capacity for 3rd generation taxoids: preparation, in vitro and in vivo evaluation.Progress in breast cancer research.Cross-species DNA copy number analyses identifies multiple 1q21-q23 subtype-specific driver genes for breast cancerc-Jun N-terminal kinase 2 prevents luminal cell commitment in normal mammary glands and tumors by inhibiting p53/Notch1 and breast cancer gene 1 expression.
P2860
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P2860
Comparative oncogenomics identifies breast tumors enriched in functional tumor-initiating cells.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Comparative oncogenomics ident ...... tional tumor-initiating cells.
@ast
Comparative oncogenomics ident ...... tional tumor-initiating cells.
@en
type
label
Comparative oncogenomics ident ...... tional tumor-initiating cells.
@ast
Comparative oncogenomics ident ...... tional tumor-initiating cells.
@en
prefLabel
Comparative oncogenomics ident ...... tional tumor-initiating cells.
@ast
Comparative oncogenomics ident ...... tional tumor-initiating cells.
@en
P2093
P2860
P356
P1476
Comparative oncogenomics ident ...... tional tumor-initiating cells.
@en
P2093
Daniel Medina
David Darr
David Edwards
George Murrow
Jana Knezevic
Jason I Herschkowitz
Jeffrey M Rosen
Jerry Usary
Melissa A Troester
P2860
P304
P356
10.1073/PNAS.1018862108
P407
P577
2011-06-01T00:00:00Z