BRCA1 may modulate neuronal cell cycle re-entry in Alzheimer disease
about
The excitotoxin quinolinic acid induces tau phosphorylation in human neuronsRepair of oxidative DNA damage, cell-cycle regulation and neuronal death may influence the clinical manifestation of Alzheimer's disease.Induction of neuro-protective/regenerative genes in stem cells infiltrating post-ischemic brain tissue.Differential network analyses of Alzheimer's disease identify early events in Alzheimer's disease pathologyPathological implications of cell cycle re-entry in Alzheimer disease.Evidence for induction of the ornithine transcarbamylase expression in Alzheimer's disease.Cell cycle checkpoint abnormalities during dementia: A plausible association with the loss of protection against oxidative stress in Alzheimer's disease [corrected].Beta-amyloid increases the expression level of ATBF1 responsible for death in cultured cortical neurons.BRCA1 silencing is associated with failure of DNA repairing in retinal neurocytesInhibition of Polo-like kinase 1 reduces beta-amyloid-induced neuronal cell death in Alzheimer's disease.Brca1 is expressed in human microglia and is dysregulated in human and animal model of ALS.Oxidative stress response and Nrf2 signaling in aging.DNA repair factor BRCA1 depletion occurs in Alzheimer brains and impairs cognitive function in mice.Retinoblastoma protein phosphorylation at multiple sites is associated with neurofibrillary pathology in Alzheimer disease.HIV-infected macrophages mediate neuronal apoptosis through mitochondrial glutaminaseThe neuronal expression of MYC causes a neurodegenerative phenotype in a novel transgenic mouse.Global hypermethylation in fetal cortex of Down syndrome due to DNMT3L overexpression.Tumorigenesis and neurodegeneration: two sides of the same coin?Cell cycle deregulation in the neurons of Alzheimer's disease.Cell cycle proteins in brain in mild cognitive impairment: insights into progression to Alzheimer disease.Caffeine modulates tau phosphorylation and affects Akt signaling in postmitotic neurons.Loss of the chromatin regulator MRG15 limits neural stem/progenitor cell proliferation via increased expression of the p21 Cdk inhibitor.Neuron-specific methylome analysis reveals epigenetic regulation and tau-related dysfunction of BRCA1 in Alzheimer's disease.Distinct chronology of neuronal cell cycle re-entry and tau pathology in the 3xTg-AD mouse model and Alzheimer's disease patients.U1 snRNP Alteration and Neuronal Cell Cycle Reentry in Alzheimer Disease.Brca1 Is Upregulated by 5-Aza-CdR and Promotes DNA Repair and Cell Survival, and Inhibits Neurite Outgrowth in Rat Retinal Neurons.Repositioning Microtubule Stabilizing Drugs for Brain Disorders
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BRCA1 may modulate neuronal cell cycle re-entry in Alzheimer disease
description
2007 nî lūn-bûn
@nan
2007年の論文
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2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
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2007年论文
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2007年论文
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2007年论文
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name
BRCA1 may modulate neuronal cell cycle re-entry in Alzheimer disease
@ast
BRCA1 may modulate neuronal cell cycle re-entry in Alzheimer disease
@en
type
label
BRCA1 may modulate neuronal cell cycle re-entry in Alzheimer disease
@ast
BRCA1 may modulate neuronal cell cycle re-entry in Alzheimer disease
@en
prefLabel
BRCA1 may modulate neuronal cell cycle re-entry in Alzheimer disease
@ast
BRCA1 may modulate neuronal cell cycle re-entry in Alzheimer disease
@en
P2093
P2860
P50
P356
P1476
BRCA1 may modulate neuronal cell cycle re-entry in Alzheimer disease
@en
P2093
André Delacourte
Arun K Raina
Hyoung-gon Lee
Olga Aprelikova
Teresa A Evans
P2860
P304
P356
10.7150/IJMS.4.140
P407
P577
2007-05-12T00:00:00Z