Two-compartment tumor metabolism: autophagy in the tumor microenvironment and oxidative mitochondrial metabolism (OXPHOS) in cancer cells
about
Cilia in autophagy and cancerDistinct Biochemical Pools of Golgi Phosphoprotein 3 in the Human Breast Cancer Cell Lines MCF7 and MDA-MB-231The multiple cellular functions of the oncoprotein Golgi phosphoprotein 3The footprint of the ageing stroma in older patients with breast cancer.The nutritional phenome of EMT-induced cancer stem-like cellsDeregulation of the EGFR/PI3K/PTEN/Akt/mTORC1 pathway in breast cancer: possibilities for therapeutic intervention.Down-regulation of stromal caveolin-1 expression in esophageal squamous cell carcinoma: a potent predictor of lymph node metastases, early tumor recurrence, and poor prognosis.Mechanisms of environmental chemicals that enable the cancer hallmark of evasion of growth suppression.Gli1 mediates lung cancer cell proliferation and Sonic Hedgehog-dependent mesenchymal cell activation.The ketogenic diet and hyperbaric oxygen therapy prolong survival in mice with systemic metastatic cancer.Loss of Sirt1 promotes prostatic intraepithelial neoplasia, reduces mitophagy, and delays PARK2 translocation to mitochondriaMitochondrial dysfunction promotes breast cancer cell migration and invasion through HIF1α accumulation via increased production of reactive oxygen speciesGOLPH3 links the Golgi, DNA damage, and cancer.Metabolic phenotypes in pancreatic cancer.Mitochondrial mass, a new metabolic biomarker for stem-like cancer cells: Understanding WNT/FGF-driven anabolic signaling.Gene expression signatures of breast cancer stem and progenitor cells do not exhibit features of Warburg metabolism.Cigarette Smoking Condensate Disrupts Endoplasmic Reticulum-Golgi Network Homeostasis Through GOLPH3 Expression in Normal Lung Epithelial Cells.Mitochondrial fission induces glycolytic reprogramming in cancer-associated myofibroblasts, driving stromal lactate production, and early tumor growth.Mitochondrial biogenesis in epithelial cancer cells promotes breast cancer tumor growth and confers autophagy resistance.Mitochondria "fuel" breast cancer metabolism: fifteen markers of mitochondrial biogenesis label epithelial cancer cells, but are excluded from adjacent stromal cellsMitochondrial dysfunction in breast cancer cells prevents tumor growth: understanding chemoprevention with metformin.Impaired mitochondrial metabolism and mammary carcinogenesisCigarette smoke metabolically promotes cancer, via autophagy and premature aging in the host stromal microenvironment.Li Fraumeni syndrome, cancer and senescence: a new hypothesis.Compartment-specific activation of PPARγ governs breast cancer tumor growth, via metabolic reprogramming and symbiosis.Metabolic control analysis of respiration in human cancer tissue.Oncogenes and inflammation rewire host energy metabolism in the tumor microenvironment: RAS and NFκB target stromal MCT4.Phosphorylated AKT inhibits the apoptosis induced by DRAM-mediated mitophagy in hepatocellular carcinoma by preventing the translocation of DRAM to mitochondria.GOLPH3 overexpression correlates with poor response to neoadjuvant therapy and prognosis in locally advanced rectal cancerStudy of GOLPH3: a potential stress-inducible protein from Golgi apparatus.Autophagy and cancer metabolism.Targeting tumor microenvironment: crossing tumor interstitial fluid by multifunctional nanomedicinesMetabostemness: a new cancer hallmark.Metabolic restructuring and cell fate conversion.The Intricate Interplay between Mechanisms Underlying Aging and Cancer.Mitochondrial Respiration in Human Colorectal and Breast Cancer Clinical Material Is Regulated Differently.Golgi phosphoprotein3 overexpression is associated with poor survival in patients with solid tumors: a meta-analysis.Berberine Regulated Lipid Metabolism in the Presence of C75, Compound C, and TOFA in Breast Cancer Cell Line MCF-7.Golgi-Related Proteins GOLPH2 (GP73/GOLM1) and GOLPH3 (GOPP1/MIDAS) in Cutaneous Melanoma: Patterns of Expression and Prognostic Significance.Vitamin C and Doxycycline: A synthetic lethal combination therapy targeting metabolic flexibility in cancer stem cells (CSCs).
P2860
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P2860
Two-compartment tumor metabolism: autophagy in the tumor microenvironment and oxidative mitochondrial metabolism (OXPHOS) in cancer cells
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Two-compartment tumor metaboli ...... olism (OXPHOS) in cancer cells
@ast
Two-compartment tumor metaboli ...... olism (OXPHOS) in cancer cells
@en
type
label
Two-compartment tumor metaboli ...... olism (OXPHOS) in cancer cells
@ast
Two-compartment tumor metaboli ...... olism (OXPHOS) in cancer cells
@en
prefLabel
Two-compartment tumor metaboli ...... olism (OXPHOS) in cancer cells
@ast
Two-compartment tumor metaboli ...... olism (OXPHOS) in cancer cells
@en
P2093
P2860
P50
P356
P1433
P1476
Two-compartment tumor metaboli ...... olism (OXPHOS) in cancer cells
@en
P2093
Ahmed F Salem
Anthony Howell
Mazhar Salim Al-Zoubi
Richard G Pestell
Ubaldo E Martinez-Outschoorn
P2860
P304
P356
10.4161/CC.20920
P577
2012-07-01T00:00:00Z