Multiple low-dose streptozotocin-induced hyperglycemia and insulitis in C57BL mice: influence of inbred background, sex, and thymus
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Immunotoxicology - regulatory and risk assessment conceptsCan exposure to environmental chemicals increase the risk of diabetes type 1 development?Diabetes mellitus reduces the function and expression of ATP-dependent K⁺ channels in cardiac mitochondriaMechanisms of estradiol-induced insulin secretion by the G protein-coupled estrogen receptor GPR30/GPER in pancreatic beta-cellsActivation of AKT by O-linked N-acetylglucosamine induces vascular calcification in diabetes mellitusThe antidiabetic effect of MSCs is not impaired by insulin prophylaxis and is not improved by a second dose of cells.Early hepatic insulin resistance precedes the onset of diabetes in obese C57BLKS-db/db mice.Genistein induces pancreatic beta-cell proliferation through activation of multiple signaling pathways and prevents insulin-deficient diabetes in miceInterleukin-1β produced in response to islet autoantigen presentation differentiates T-helper 17 cells at the expense of regulatory T-cells: Implications for the timing of tolerizing immunotherapy.Disruption of hepatocyte growth factor/c-Met signaling enhances pancreatic beta-cell death and accelerates the onset of diabetesMultiple low-dose streptozotocin-induced diabetes in the mouse. Evidence for stimulation of a cytotoxic cellular immune response against an insulin-producing beta cell line.High pancreatic n-3 fatty acids prevent STZ-induced diabetes in fat-1 mice: inflammatory pathway inhibitionTransfer of hematopoietic stem cells encoding autoantigen prevents autoimmune diabetes.CD18 deficiency protects against multiple low-dose streptozotocin-induced diabetes.Genistein ameliorates hyperglycemia in a mouse model of nongenetic type 2 diabetes.Alteration of the cardiac sympathetic innervation is modulated by duration of diabetes in female ratsThe streptozotocin-induced diabetic nude mouse model: differences between animals from different sources.Calcium insufficiency accelerates type 1 diabetes in vitamin D receptor-deficient nonobese diabetic (NOD) mice.Early-onset diabetic E1-DN mice develop albuminuria and glomerular injury typical of diabetic nephropathy.A humoral immune defect distinguishes the response to Staphylococcus aureus infections in mice with obesity and type 2 diabetes from that in mice with type 1 diabetesMagnetic resonance imaging of the pancreas in streptozotocin-induced diabetic rats: Gadofluorine P and Gd-DOTA.Autoimmunity and environmental factors in the pathogenesis of primary biliary cirrhosis.The Effects of Acute Blood Loss for Diagnostic Bloodwork and Fluid Replacement in Clinically Ill Mice.Nonhuman primate models for islet transplantation in type 1 diabetes research.Expression profiling pre-diabetic mice to uncover drugs with clinical application to type 1 diabetes.Association of Bactericidal Dysfunction of Paneth Cells in Streptozocin-Induced Diabetic Mice with Insulin DeficiencyOsteoprotegerin and Denosumab Stimulate Human Beta Cell Proliferation through Inhibition of the Receptor Activator of NF-κB Ligand Pathway.In Vivo Evaluation of the Visual Pathway in Streptozotocin-Induced Diabetes by Diffusion Tensor MRI and Contrast Enhanced MRI.Human umbilical cord matrix-derived stem cells exert trophic effects on β-cell survival in diabetic rats and isolated islets.Diabetes induced in male transgenic mice by expression of human H-ras oncoprotein in pancreatic beta cellsAdvances in murine models of diabetic nephropathy.Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset.Streptozotocin is equally diabetogenic whether administered to fed or fasted miceExperimental diabetic neuropathy: impairment of slow transport with changes in axon cross-sectional area.Single dose streptozotocin-induced diabetes: considerations for study design in islet transplantation models.Mouse models of diabetic neuropathy.Rodent animal models: from mild to advanced stages of diabetic nephropathy.T-cell vaccination leads to suppression of intrapancreatic Th17 cells through Stat3-mediated RORγt inhibition in autoimmune diabetes.β-Cell Specific Overexpression of GPR39 Protects against Streptozotocin-Induced HyperglycemiaA mitochondrial therapeutic reverses visual decline in mouse models of diabetes
P2860
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P2860
Multiple low-dose streptozotocin-induced hyperglycemia and insulitis in C57BL mice: influence of inbred background, sex, and thymus
description
1982 nî lūn-bûn
@nan
1982年の論文
@ja
1982年学术文章
@wuu
1982年学术文章
@zh-cn
1982年学术文章
@zh-hans
1982年学术文章
@zh-my
1982年学术文章
@zh-sg
1982年學術文章
@yue
1982年學術文章
@zh
1982年學術文章
@zh-hant
name
Multiple low-dose streptozotoc ...... ed background, sex, and thymus
@ast
Multiple low-dose streptozotoc ...... ed background, sex, and thymus
@en
type
label
Multiple low-dose streptozotoc ...... ed background, sex, and thymus
@ast
Multiple low-dose streptozotoc ...... ed background, sex, and thymus
@en
prefLabel
Multiple low-dose streptozotoc ...... ed background, sex, and thymus
@ast
Multiple low-dose streptozotoc ...... ed background, sex, and thymus
@en
P2860
P356
P1476
Multiple low-dose streptozotoc ...... ed background, sex, and thymus
@en
P2093
E H Leiter
P2860
P304
P356
10.1073/PNAS.79.2.630
P407
P577
1982-01-01T00:00:00Z