Divalent cation competition with [3H]saxitoxin binding to tetrodotoxin-resistant and -sensitive sodium channels. A two-site structural model of ion/toxin interaction.
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Block of sodium channels by divalent mercury: role of specific cysteinyl residues in the P-loop region.Pore residues critical for mu-CTX binding to rat skeletal muscle Na+ channels revealed by cysteine mutagenesis.The saxitoxin/tetrodotoxin binding site on cloned rat brain IIa Na channels is in the transmembrane electric field.Differential effects of sulfhydryl reagents on saxitoxin and tetrodotoxin block of voltage-dependent Na channels.An unexpected role for brain-type sodium channels in coupling of cell surface depolarization to contraction in the heart.Use dependence of tetrodotoxin block of sodium channels: a revival of the trapped-ion mechanism.Permeation of Na+ through open and Zn(2+)-occupied conductance states of cardiac sodium channels modified by batrachotoxin: exploring ion-ion interactions in a multi-ion channelPost-repolarization block of cloned sodium channels by saxitoxin: the contribution of pore-region amino acids.A mutation in the pore of the sodium channel alters gating.Selectivity and permeation in calcium release channel of cardiac muscle: alkali metal ionsParalytic shellfish poisoning in Kodiak, AlaskaUse-dependent block of the voltage-gated Na(+) channel by tetrodotoxin and saxitoxin: effect of pore mutations that change ionic selectivity.Modification of cardiac sodium channels by carboxyl reagents. Trimethyloxonium and water-soluble carbodiimide.Differential interactions of Na+ channel toxins with T-type Ca2+ channels.Sodium channel diversity in the vestibular ganglion: NaV1.5, NaV1.8, and tetrodotoxin-sensitive currents.The tetrodotoxin binding site is within the outer vestibule of the sodium channel.Matrix effects on a cell-based assay used for the detection of paralytic shellfish toxins in bivalve shellfish samples.A structural model of the tetrodotoxin and saxitoxin binding site of the Na+ channel.Mechanisms of cation permeation in cardiac sodium channel: description by dynamic pore model.A "minimal" sodium channel construct consisting of ligated S5-P-S6 segments forms a toxin-activatable ionophore.Cardiac sodium channels expressed in a peripheral neurotumor-derived cell line, RT4-B8.Zinc co-localizes with GABA and glycine in synapses in the lamprey spinal cord.Amphibian peptides that inhibit neuronal nitric oxide synthase. Isolation of lesuerin from the skin secretion of the Australian Stony Creek frog Litoria lesueuri.Cu(II) inhibition of the proton translocation machinery of the influenza A virus M2 protein.
P2860
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P2860
Divalent cation competition with [3H]saxitoxin binding to tetrodotoxin-resistant and -sensitive sodium channels. A two-site structural model of ion/toxin interaction.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
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1993年學術文章
@zh-hant
name
Divalent cation competition wi ...... odel of ion/toxin interaction.
@ast
Divalent cation competition wi ...... odel of ion/toxin interaction.
@en
type
label
Divalent cation competition wi ...... odel of ion/toxin interaction.
@ast
Divalent cation competition wi ...... odel of ion/toxin interaction.
@en
prefLabel
Divalent cation competition wi ...... odel of ion/toxin interaction.
@ast
Divalent cation competition wi ...... odel of ion/toxin interaction.
@en
P2093
P2860
P356
P1476
Divalent cation competition wi ...... odel of ion/toxin interaction.
@en
P2093
P2860
P304
P356
10.1085/JGP.101.2.153
P577
1993-02-01T00:00:00Z