Pleiotropic IFN-dependent and -independent effects of IRF5 on the pathogenesis of experimental lupus
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Breakdown of Immune Tolerance in Systemic Lupus Erythematosus by Dendritic CellsInterferon regulatory factor-5 deficiency ameliorates disease severity in the MRL/lpr mouse model of lupus in the absence of a mutation in DOCK2.Serum autoantibodies in pristane induced lupus are regulated by neutrophil gelatinase associated lipocalinAn essential role of caspase 1 in the induction of murine lupus and its associated vascular damageRequirements for innate immune pathways in environmentally induced autoimmunity.Mechanisms of tumor necrosis factor α antagonist-induced lupus in a murine model.IRF5 deficiency ameliorates lupus but promotes atherosclerosis and metabolic dysfunction in a mouse model of lupus-associated atherosclerosisIRF5 controls both acute and chronic inflammation.Pristane-Accelerated Autoimmune Disease in (SWR X NZB) F1 Mice Leads to Prominent Tubulointerstitial Inflammation and Human Lupus Nephritis-Like Fibrosis.Monocytes from Irf5-/- mice have an intrinsic defect in their response to pristane-induced lupus.Maintenance of autoantibody production in pristane-induced murine lupus.C1q Modulates the Response to TLR7 Stimulation by Pristane-Primed Macrophages: Implications for Pristane-Induced Lupus.Maintenance of anti-Sm/RNP autoantibody production by plasma cells residing in ectopic lymphoid tissue and bone marrow memory B cells.Phenotype and function of B cells and dendritic cells from interferon regulatory factor 5-deficient mice with and without a mutation in DOCK2.The scavenger receptor SCARF1 mediates the clearance of apoptotic cells and prevents autoimmunity.Interferon regulatory factor 5 and autoimmune lupus.Epigenetics and the IRFs: a complex interplay in the control of immunity and autoimmunity.Toll-like receptor 7-stimulated tumor necrosis factor α causes bone marrow damage in systemic lupus erythematosus.IRF5 is elevated in childhood-onset SLE and regulated by histone acetyltransferase and histone deacetylase inhibitors.CD38 promotes pristane-induced chronic inflammation and increases susceptibility to experimental lupus by an apoptosis-driven and TRPM2-dependent mechanism.Immunosuppressive CD11b+Ly6Chi monocytes in pristane-induced lupus mouse model.
P2860
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P2860
Pleiotropic IFN-dependent and -independent effects of IRF5 on the pathogenesis of experimental lupus
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
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2012年论文
@zh-cn
name
Pleiotropic IFN-dependent and ...... ogenesis of experimental lupus
@ast
Pleiotropic IFN-dependent and ...... ogenesis of experimental lupus
@en
type
label
Pleiotropic IFN-dependent and ...... ogenesis of experimental lupus
@ast
Pleiotropic IFN-dependent and ...... ogenesis of experimental lupus
@en
prefLabel
Pleiotropic IFN-dependent and ...... ogenesis of experimental lupus
@ast
Pleiotropic IFN-dependent and ...... ogenesis of experimental lupus
@en
P2093
P2860
P356
P1476
Pleiotropic IFN-dependent and ...... ogenesis of experimental lupus
@en
P2093
Clayton E Mathews
Dina C Nacionales
Haoyang Zhuang
Jason Weinstein
Li-Jun Yang
Lyle L Moldawer
Minoru Satoh
P2860
P304
P356
10.4049/JIMMUNOL.1103113
P407
P577
2012-03-14T00:00:00Z