Estradiol repression of tumor necrosis factor-alpha transcription requires estrogen receptor activation function-2 and is enhanced by coactivators.
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Estradiol and selective estrogen receptor modulators differentially regulate target genes with estrogen receptors alpha and betaLiquiritigenin is a plant-derived highly selective estrogen receptor beta agonistHistone deacetylase 7 and FoxA1 in estrogen-mediated repression of RPRMERbeta Binds N-CoR in the Presence of Estrogens via an LXXLL-like Motif in the N-CoR C-terminusMinireview: nuclear receptor coregulators of the p160 family: insights into inflammation and metabolismGender differences in murine pulmonary responses elicited by cellulose nanocrystalsEstrogenic plant extracts reverse weight gain and fat accumulation without causing mammary gland or uterine proliferationEstrogen receptor-β regulates human tryptophan hydroxylase-2 through an estrogen response element in the 5' untranslated regionThe ligand-mediated nuclear mobility and interaction with estrogen-responsive elements of estrogen receptors are subtype specific.The clinical significance of PR, ER, NF- κ B, and TNF- α in breast cancer17β-Estradiol Ameliorates Tight Junction Disruption via Repression of MMP Transcription.Benefit of physical fitness against inflammation in obesity: role of beta adrenergic receptors.Effects of estradiol on lipopolysaccharide and Pam3Cys stimulation of CCL20/macrophage inflammatory protein 3 alpha and tumor necrosis factor alpha production by uterine epithelial cells in cultureEstradiol suppresses NF-kappa B activation through coordinated regulation of let-7a and miR-125b in primary human macrophages.Estrogen receptor beta-selective transcriptional activity and recruitment of coregulators by phytoestrogens.Factor recruitment and TIF2/GRIP1 corepressor activity at a collagenase-3 response element that mediates regulation by phorbol esters and hormones.Positive cross-talk between estrogen receptor and NF-kappaB in breast cancer.Alternate surfaces of transcriptional coregulator GRIP1 function in different glucocorticoid receptor activation and repression contexts.Estradiol rapidly inhibits soluble guanylyl cyclase expression in rat uterus.The nuclear receptor transcriptional coregulator RIP140.Markers of inflammation are heritable and associated with subcutaneous and ectopic skeletal muscle adiposity in African ancestry families.Estradiol and progesterone strongly inhibit the innate immune response of mononuclear cells in newborns.Breaking the cycle: An insight into the role of ERα in eukaryotic cell cycles.Timing of estrogen therapy after ovariectomy dictates the efficacy of its neuroprotective and antiinflammatory actions.Interactions between the immune system and bone.Estrogenic modulation of inflammation-related genes in male rats following volume overloadEstrogen deficiency induces bone loss by increasing T cell proliferation and lifespan through IFN-gamma-induced class II transactivator.The role of T lymphocytes in bone metabolism.Expression of oestrogen receptor-beta in oestrogen receptor-alpha negative human breast tumours.Estrogen receptor beta isoform-specific induction of transforming growth factor beta-inducible early gene-1 in human osteoblast cells: an essential role for the activation function 1 domainMarkers of increased cardiovascular risk in postmenopausal women: focus on oxidized-LDL and HDL subpopulations.Repression of ESR1 through actions of estrogen receptor alpha and Sin3A at the proximal promoter.MF101: a multi-component botanical selective estrogen receptor beta modulator for the treatment of menopausal vasomotor symptoms.Evidence that estrogen receptor beta enhances MMP-13 promoter activity in HIG-82 cells and that this enhancement can be influenced by ligands and involves specific promoter sites.The type I interferon signaling pathway is a target for glucocorticoid inhibition.Metyrapone restores the febrile response to Escherichia coli LPS in pregnant rats.The functional interaction between HMGA1 and the estrogen receptor requires either the N- or the C-terminal domain of the receptor.The histone deacetylase inhibitor trichostatin A sensitizes estrogen receptor alpha-negative breast cancer cells to tamoxifen.Effects of phorbol dibutyrate on cell proliferation, apoptosis, and tumor necrosis factor-alpha expression in human endometrial adenocarcinoma cells.Estradiol decreases IGF-1 and IGF-1 receptor expression in rat aortic smooth muscle cells. Mechanisms for its atheroprotective effects.
P2860
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P2860
Estradiol repression of tumor necrosis factor-alpha transcription requires estrogen receptor activation function-2 and is enhanced by coactivators.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
1999年论文
@zh
1999年论文
@zh-cn
name
Estradiol repression of tumor ...... d is enhanced by coactivators.
@en
type
label
Estradiol repression of tumor ...... d is enhanced by coactivators.
@en
prefLabel
Estradiol repression of tumor ...... d is enhanced by coactivators.
@en
P2093
P2860
P356
P1476
Estradiol repression of tumor ...... d is enhanced by coactivators.
@en
P2093
D C Leitman
J A Gustafsson
J D Baxter
P J Kushner
R C Ribeiro
P2860
P304
15161-15166
P356
10.1073/PNAS.96.26.15161
P407
P577
1999-12-01T00:00:00Z