Activation of RAGE induces elevated O2- generation by mononuclear phagocytes in diabetes - Wikidata - awgldk - TriplyDB

Activation of RAGE induces elevated O2- generation by mononuclear phagocytes in diabetes

Activation of RAGE induces elevated O2- generation by mononuclear phagocytes in diabetes

http://www.wikidata.org/entity/Q36870610

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Diabetes mellitus related bone metabolism and periodontal disease Diabetes-induced oxidative stress is mediated by Ca2+-independent phospholipase A2 in neutrophils.Ursolic acid protects diabetic mice against monocyte dysfunction and accelerated atherosclerosis.Annato extract and β-carotene modulate the production of reactive oxygen species/nitric oxide in neutrophils from diabetic rats.Oral inflammatory diseases and systemic inflammation: role of the macrophage.Burkholderia pseudomallei triggers altered inflammatory profiles in a whole-blood model of type 2 diabetes-melioidosis comorbidity Phosphorylation of pleckstrin increases proinflammatory cytokine secretion by mononuclear phagocytes in diabetes mellitus.Markers of Antioxidant Defense in Patients with Type 2 Diabetes Tuning three-dimensional collagen matrix stiffness independently of collagen concentration modulates endothelial cell behavior Reduced Mycobacterium tuberculosis association with monocytes from diabetes patients that have poor glucose control IPLA2 mRNA expression by human neutrophils in type 2 diabetes and chronic periodontitis Na+/H+ exchanger 1 inhibition reverses manifestation of peripheral diabetic neuropathy in type 1 diabetic rats.Potential of the angiotensin receptor blockers (ARBs) telmisartan, irbesartan, and candesartan for inhibiting the HMGB1/RAGE axis in prevention and acute treatment of stroke.Endogenous damage-associated molecular pattern molecules at the crossroads of inflammation and cancer.Controlling the mechanical properties of three-dimensional matrices via non-enzymatic collagen glycation Priming of neutrophil oxidative burst in diabetes requires preassembly of the NADPH oxidase.Ulinastatin preconditioning attenuates inflammatory reaction of hepatic ischemia reperfusion injury in rats via high mobility group box 1(HMGB1) inhibition Quintessential risk factors: their role in promoting cognitive dysfunction and Alzheimer's disease.Mechanisms of distal axonal degeneration in peripheral neuropathies.2016 ATVB Plenary Lecture: Receptor for Advanced Glycation Endproducts and Implications for the Pathogenesis an Treatment of Cardiometabolic Disorders: Spotlight on the Macrophage.Beneficial effects of a novel RAGE inhibitor on early diabetic retinopathy and tactile allodynia.Overexpression of Bcl-2 in vascular endothelium inhibits the microvascular lesions of diabetic retinopathy.Erythropoietin attenuates advanced glycation endproducts-induced toxicity of Schwann cells in vitro.Scavenging of reactive oxygen species by astaxanthin inhibits epithelial-mesenchymal transition in high glucose-stimulated mesothelial cells.STR/ort mice, a model for spontaneous osteoarthritis, exhibit elevated levels of both local and systemic inflammatory markers.Abnormal cell responses and role of TNF-α in impaired diabetic wound healing.RAGE mediates the inactivation of nAChRs in sympathetic neurons under high glucose conditions.Linking senile dementia to type 2 diabetes: role of oxidative stress markers, C-reactive protein and tumor necrosis factor-α.

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Activation of RAGE induces elevated O2- generation by mononuclear phagocytes in diabetes

http://www.wikidata.org/entity/Q36870610

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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2006年论文

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2006年论文

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name

Activation of RAGE induces elevated O2- generation by mononuclear phagocytes in diabetes

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type

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Activation of RAGE induces elevated O2- generation by mononuclear phagocytes in diabetes

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Activation of RAGE induces elevated O2- generation by mononuclear phagocytes in diabetes

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Journal of Leukocyte Biology

P1476

Activation of RAGE induces elevated O2- generation by mononuclear phagocytes in diabetes

@en

P2093

Alan Malabanan

http://www.w3.org/2001/XMLSchema#string

Alpdogan Kantarci

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Hatice Hasturk

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Thomas E Van Dyke

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Yong Ding

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P2860

RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

S100A1 and S100B expression and target proteins in type I diabetes

Establishment and characterization of a human acute monocytic leukemia cell line (THP-1)

N(epsilon)-(carboxymethyl)lysine adducts of proteins are ligands for receptor for advanced glycation end products that activate cell signaling pathways and modulate gene expression

Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage

Activation of the respiratory burst enzyme in human polymorphonuclear leukocytes by chemoattractants and other soluble stimuli. Evidence that the same oxidase is activated by different transductional mechanisms.

Advanced protein glycosylation induces transendothelial human monocyte chemotaxis and secretion of platelet-derived growth factor: role in vascular disease of diabetes and aging.

Advanced glycosylation end products in tissue and the biochemical basis of diabetic complications.

Cellular response to oxidative stress: signaling for suicide and survival.

Pathogenesis of beta(2)-microglobulin amyloidosis: role of monocytes/macrophages.

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10.1189/JLB.0406262

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2

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81

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Philip C Trackman

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2006-11-09T00:00:00Z

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6701043

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17095613

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2533418

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