Interplay of viral and cellular proteins along the long control region of human papillomavirus type 18.
about
Expression of the papillomavirus E2 protein in HeLa cells leads to apoptosisCharacterization of junD: a new member of the jun proto-oncogene familyHeparin (GAG-hed) inhibits LCR activity of human papillomavirus type 18 by decreasing AP1 binding.Novel binding sites for regulatory factors in the human papillomavirus type 18 enhancer and promoter identified by in vivo footprintingTransactivation-competent bovine papillomavirus E2 protein is specifically required for efficient repression of human papillomavirus oncogene expression and for acute growth inhibition of cervical carcinoma cell linesMechanisms of human papillomavirus E2-mediated repression of viral oncogene expression and cervical cancer cell growth inhibition.The bovine papillomavirus constitutive enhancer is essential for viral transformation, DNA replication, and the maintenance of latencyRetinoic acid-mediated repression of human papillomavirus 18 transcription and different ligand regulation of the retinoic acid receptor beta gene in non-tumorigenic and tumorigenic HeLa hybrid cellsThe upstream regulatory region of human papillomavirus type 31 is insensitive to glucocorticoid induction.Human papillomavirus type 16 E2 protein has no effect on transcription from episomal viral DNA.Characterization of the methylation patterns in human papillomavirus type 16 viral DNA in head and neck cancers.CpG methylation of human papillomavirus type 16 DNA in cervical cancer cell lines and in clinical specimens: genomic hypomethylation correlates with carcinogenic progressionClusters of nuclear factor I binding sites identify enhancers of several papillomaviruses but alone are not sufficient for enhancer function.The effects of DNA methylation and histone deacetylase inhibitors on human papillomavirus early gene expression in cervical cancer, an in vitro and clinical studyDelineation of human papillomavirus type 18 enhancer binding proteins: the intracellular distribution of a novel octamer binding protein p92 is cell cycle regulated.A switch region determines the cell type-specific positive or negative action of YY1 on the activity of the human papillomavirus type 18 promoter.The product of the adenovirus intermediate gene IX is a transcriptional activator.Transcription activities of human papillomavirus type 11 E6 promoter-proximal elements in raft and submerged cultures of foreskin keratinocytes.Activation of human papillomavirus type 18 E6-E7 oncogene expression by transcription factor Sp1.Propagation of infectious human papillomavirus type 16 by using an adenovirus and Cre/LoxP mechanism.Inhibition of cervical carcinoma cell line proliferation by the introduction of a bovine papillomavirus regulatory geneIdentification and characterization of novel promoters in the genome of human papillomavirus type 18.Cell-type-specific activity of the human papillomavirus type 18 upstream regulatory region in transgenic mice and its modulation by tetradecanoyl phorbol acetate and glucocorticoids.Epidermal growth factor (EGF) elicits down-regulation of human papillomavirus type 16 (HPV-16) E6/E7 mRNA at the transcriptional level in an EGF-stimulated human keratinocyte cell line: functional role of EGF-responsive silencer in the HPV-16 long cA constitutive enhancer in the bovine papillomavirus upstream regulatory region shares genetic elements with the viral P1 promoterTwo AP1 sites binding JunB are essential for human papillomavirus type 18 transcription in keratinocytesTransient replication of human papillomavirus DNAs.Cooperative activation of transcription by bovine papillomavirus type 1 E2 can occur over a large distanceA member of the activator protein 1 family found in keratinocytes but not in fibroblasts required for transcription from a human papillomavirus type 18 promoter.A bovine papillomavirus constitutive enhancer is negatively regulated by the E2 repressor through competitive binding for a cellular factorMutational analysis of cis elements involved in E2 modulation of human papillomavirus type 16 P97 and type 18 P105 promoters.The enhancer of human papillomavirus type 16: binding sites for the ubiquitous transcription factors oct-1, NFA, TEF-2, NF1, and AP-1 participate in epithelial cell-specific transcription.Progesterone and glucocorticoid response elements occur in the long control regions of several human papillomaviruses involved in anogenital neoplasiaThe human papillomavirus type 18 (HPV18) E2 gene product is a repressor of the HPV18 regulatory region in human keratinocytes.NFI-Ski interactions mediate transforming growth factor beta modulation of human papillomavirus type 16 early gene expression.NFI is an essential positive transcription factor for human papillomavirus type 16 early gene expressionA keratinocyte-specific transcription factor, KRF-1, interacts with AP-1 to activate expression of human papillomavirus type 18 in squamous epithelial cells.Transcriptional control of human papillomavirus (HPV) oncogene expression: composition of the HPV type 18 upstream regulatory region.HMG-I(Y) and the CBP/p300 coactivator are essential for human papillomavirus type 18 enhanceosome transcriptional activity.The transcription factors TBX2 and TBX3 interact with human papillomavirus 16 (HPV16) L2 and repress the long control region of HPVs
P2860
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P2860
Interplay of viral and cellular proteins along the long control region of human papillomavirus type 18.
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年論文
@yue
1988年論文
@zh-hant
1988年論文
@zh-hk
1988年論文
@zh-mo
1988年論文
@zh-tw
1988年论文
@wuu
1988年论文
@zh
1988年论文
@zh-cn
name
Interplay of viral and cellula ...... human papillomavirus type 18.
@en
type
label
Interplay of viral and cellula ...... human papillomavirus type 18.
@en
prefLabel
Interplay of viral and cellula ...... human papillomavirus type 18.
@en
P2093
P2860
P1433
P1476
Interplay of viral and cellula ...... human papillomavirus type 18.
@en
P2093
Garcia-Carranca A
P2860
P304
P407
P577
1988-11-01T00:00:00Z