The human papillomavirus type 18 (HPV18) E2 gene product is a repressor of the HPV18 regulatory region in human keratinocytes.
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Brd4-independent transcriptional repression function of the papillomavirus e2 proteinsBrd4 links chromatin targeting to HPV transcriptional silencingThe 3' region of human papillomavirus type 16 early mRNAs decrease expression.Human Papillomavirus (HPV)-associated Oral Cancers and Treatment StrategiesHuman papillomavirus induced transformation in cervical and head and neck cancersThe Cellular DNA Helicase ChlR1 Regulates Chromatin and Nuclear Matrix Attachment of the Human Papillomavirus 16 E2 Protein and High-Copy-Number Viral Genome Establishment.The human papillomavirus type 16 L1 protein directly interacts with E2 and enhances E2-dependent replication and transcription activation.Abrogation of the Brd4-positive transcription elongation factor B complex by papillomavirus E2 protein contributes to viral oncogene repressionPapillomavirus interaction with cellular chromatin.Genome-wide siRNA screen identifies SMCX, EP400, and Brd4 as E2-dependent regulators of human papillomavirus oncogene expressionTransactivation-competent bovine papillomavirus E2 protein is specifically required for efficient repression of human papillomavirus oncogene expression and for acute growth inhibition of cervical carcinoma cell linesTransactivation by the E2 protein of oncogenic human papillomavirus type 31 is not essential for early and late viral functions.Repression of the integrated papillomavirus E6/E7 promoter is required for growth suppression of cervical cancer cells.Productive replication of adeno-associated virus can occur in human papillomavirus type 16 (HPV-16) episome-containing keratinocytes and is augmented by the HPV-16 E2 protein.Mechanisms of human papillomavirus E2-mediated repression of viral oncogene expression and cervical cancer cell growth inhibition.The bovine papillomavirus constitutive enhancer is essential for viral transformation, DNA replication, and the maintenance of latencyRapid and sensitive detection of physical status of human papillomavirus type 16 DNA by quantitative real-time PCR.Analysis by multiplex PCR of the physical status of human papillomavirus type 16 DNA in cervical cancersE2F-Rb complexes assemble and inhibit cdc25A transcription in cervical carcinoma cells following repression of human papillomavirus oncogene expressionGenetic analysis of cis regulatory elements within the 5' region of the human papillomavirus type 31 upstream regulatory region during different stages of the viral life cycleA transactivator function of cottontail rabbit papillomavirus e2 is essential for tumor induction in rabbits.Human papillomavirus type 16 E2 protein has no effect on transcription from episomal viral DNA.Human papillomavirus type 16 status in cervical carcinoma cell DNA assayed by multiplex PCR.Control of HPV 18 DNA replication by cellular and viral transcription factors.Functional implications of mitochondrial reactive oxygen species generated by oncogenic virusesDuring negative regulation of the human papillomavirus-16 E6 promoter, the viral E2 protein can displace Sp1 from a proximal promoter element.Brd4 is required for e2-mediated transcriptional activation but not genome partitioning of all papillomaviruses.Small molecule inhibitors of human papillomavirus protein - protein interactions.Replication and partitioning of papillomavirus genomes.Transcriptome signature of irreversible senescence in human papillomavirus-positive cervical cancer cellsRecent Insights into the Control of Human Papillomavirus (HPV) Genome Stability, Loss, and Degradation.CCCTC-binding factor recruitment to the early region of the human papillomavirus 18 genome regulates viral oncogene expression.Repression of human papillomavirus oncogenes in HeLa cervical carcinoma cells causes the orderly reactivation of dormant tumor suppressor pathways.A switch region determines the cell type-specific positive or negative action of YY1 on the activity of the human papillomavirus type 18 promoter.Suppression of cellular proliferation by the papillomavirus E2 protein.Enhanced transcriptional activation by E2 proteins from the oncogenic human papillomavirusesCCAAT displacement protein, a regulator of differentiation-specific gene expression, binds a negative regulatory element within the 5' end of the human papillomavirus type 6 long control region.Transcriptional activation of human papillomavirus 16 by nuclear factor I, AP1, steroid receptors and a possibly novel transcription factor, PVF: a model for the composition of genital papillomavirus enhancers.Mechanisms of human papillomavirus-induced oncogenesis.Extinction of the HPV18 upstream regulatory region in cervical carcinoma cells after fusion with non-tumorigenic human keratinocytes under non-selective conditions
P2860
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P2860
The human papillomavirus type 18 (HPV18) E2 gene product is a repressor of the HPV18 regulatory region in human keratinocytes.
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
1989年论文
@zh
1989年论文
@zh-cn
name
The human papillomavirus type ...... region in human keratinocytes.
@en
type
label
The human papillomavirus type ...... region in human keratinocytes.
@en
prefLabel
The human papillomavirus type ...... region in human keratinocytes.
@en
P2093
P2860
P1433
P1476
The human papillomavirus type ...... region in human keratinocytes
@en
P2093
P2860
P304
P407
P577
1989-10-01T00:00:00Z