Hepatocyte growth factor--scatter factor: mitogen, motogen, and met.
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The zinc-finger protein slug causes desmosome dissociation, an initial and necessary step for growth factor-induced epithelial-mesenchymal transitionExtracellular proteolytic cleavage by urokinase is required for activation of hepatocyte growth factor/scatter factorRON is a heterodimeric tyrosine kinase receptor activated by the HGF homologue MSPEssential role of Gab1 for signaling by the c-Met receptor in vivoA point mutation in the MET oncogene abrogates metastasis without affecting transformation.Disassociation of met-mediated biological responses in vivo: the natural hepatocyte growth factor/scatter factor splice variant NK2 antagonizes growth but facilitates metastasis.Distinct recruitment and function of Gab1 and Gab2 in Met receptor-mediated epithelial morphogenesis.HGF/c-met/Stat3 signaling during skin tumor cell invasion: indications for a positive feedback loop.Proliferation and invasion: plasticity in tumor cells.Oncogenic mutants of RON and MET receptor tyrosine kinases cause activation of the beta-catenin pathway.Mutation of juxtamembrane tyrosine residue 1001 suppresses loss-of-function mutations of the met receptor in epithelial cells.The HGF receptor c-Met is overexpressed in esophageal adenocarcinoma.Role of chemically induced cell proliferation in carcinogenesis and its use in health risk assessmentReversal of ischemic cardiomyopathy with Sca-1+ stem cells modified with multiple growth factorsMet receptor signaling is required for sensory nerve development and HGF promotes axonal growth and survival of sensory neurons.Hepatocyte growth factor prevents acute renal failure and accelerates renal regeneration in mice.A comparative evaluation of hepatocyte growth factor levels in gingival crevicular fluid and saliva and its correlation with clinical parameters in patients with and without chronic periodontitis: A clinico-biochemical study.Invasiveness and metastasis of NIH 3T3 cells induced by Met-hepatocyte growth factor/scatter factor autocrine stimulation.Coexpression of hepatocyte growth factor and receptor (Met) in human breast carcinoma.Hereditary and sporadic papillary renal carcinomas with c-met mutations share a distinct morphological phenotype.Diverse tumorigenesis associated with aberrant development in mice overexpressing hepatocyte growth factor/scatter factorYangZheng XiaoJi exerts anti-tumour growth effects by antagonising the effects of HGF and its receptor, cMET, in human lung cancer cells.Transfer of motogenic and invasive response to scatter factor/hepatocyte growth factor by transfection of human MET protooncogene.Epidermal growth factor receptor plays a significant role in hepatocyte growth factor mediated biological responses in mammary epithelial cellsHepatocyte growth factor levels in bone marrow plasma of patients with leukaemia and its gene expression in leukaemic blast cells.Heparin induces dimerization and confers proliferative activity onto the hepatocyte growth factor antagonists NK1 and NK2.The RET-glial cell-derived neurotrophic factor (GDNF) pathway stimulates migration and chemoattraction of epithelial cells.N-cadherin promotes motility in human breast cancer cells regardless of their E-cadherin expression.Oligomerization-dependent regulation of motility and morphogenesis by the collagen XVIII NC1/endostatin domaintpr-met oncogene product induces maturation-producing factor activation in Xenopus oocytes.Hepatocyte growth factor/Met gene transfer in cardiac stem cells--potential for cardiac repair.Growth factor induced proliferation, migration, and lumen formation of rat endometrial epithelial cells in vitro.Transgenic expression of tpr-met oncogene leads to development of mammary hyperplasia and tumors.Hepatocyte growth factor is a coupling factor for osteoclasts and osteoblasts in vitro.Scatter factor/hepatocyte growth factor as a regulator of skeletal muscle and neural crest development.Enter the matrix: shape, signal and superhighway.Distinct actions and cooperative roles of ROCK and mDia in Rho small G protein-induced reorganization of the actin cytoskeleton in Madin-Darby canine kidney cellsRho and Rab small G proteins coordinately reorganize stress fibers and focal adhesions in MDCK cellsAllelic mutations in noncoding genomic sequences construct novel transcription factor binding sites that promote gene overexpression.Activation of NF-kappaB is essential for hepatocyte growth factor-mediated proliferation and tubulogenesis.
P2860
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P2860
Hepatocyte growth factor--scatter factor: mitogen, motogen, and met.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on June 1991
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Hepatocyte growth factor--scatter factor: mitogen, motogen, and met.
@en
Hepatocyte growth factor--scatter factor: mitogen, motogen, and met.
@nl
type
label
Hepatocyte growth factor--scatter factor: mitogen, motogen, and met.
@en
Hepatocyte growth factor--scatter factor: mitogen, motogen, and met.
@nl
prefLabel
Hepatocyte growth factor--scatter factor: mitogen, motogen, and met.
@en
Hepatocyte growth factor--scatter factor: mitogen, motogen, and met.
@nl
P1476
Hepatocyte growth factor--scatter factor: mitogen, motogen, and met.
@en
P2093
P304
P577
1991-06-01T00:00:00Z