Risk factors for herpes simplex virus epithelial keratitis recurring during treatment of stromal keratitis or iridocyclitis. Herpetic Eye Disease Study Group.
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Antiviral treatment and other therapeutic interventions for herpes simplex virus epithelial keratitisAntiviral treatment and other therapeutic interventions for herpes simplex virus epithelial keratitisInvolvement of STAT4 in IgG subtype switching and ocular HSV-1 replication in miceExacerbation of corneal scarring in HSV-1 gK-immunized mice correlates with elevation of CD8+CD25+ T cells in corneas of ocularly infected mice.Inhibitors of signal peptide peptidase (SPP) affect HSV-1 infectivity in vitro and in vivo.Overexpression of interleukin-2 by a recombinant herpes simplex virus type 1 attenuates pathogenicity and enhances antiviral immunityMutations within the pathogenic region of herpes simplex virus 1 gK signal sequences alter cell surface expression and neurovirulenceCD8α dendritic cells drive establishment of HSV-1 latencyAdaptive and innate transforming growth factor beta signaling impact herpes simplex virus 1 latency and reactivationProspects for developing an effective vaccine against ocular herpes simplex virus infection.Clinical efficacy of oral ganciclovir for prophylaxis and treatment of recurrent herpes simplex keratitis.Lymphoid-related CD11c+ CD8alpha+ dendritic cells are involved in enhancing herpes simplex virus type 1 latency.Role of dendritic cells in enhancement of herpes simplex virus type 1 latency and reactivation in vaccinated mice.Level of herpes simplex virus type 1 latency correlates with severity of corneal scarring and exhaustion of CD8+ T cells in trigeminal ganglia of latently infected miceA role for the JAK-STAT1 pathway in blocking replication of HSV-1 in dendritic cells and macrophagesInterrelationship of Primary Virus Replication, Level of Latency, and Time to Reactivation in the Trigeminal Ganglia of Latently Infected Mice.Understanding the role of aldose reductase in ocular inflammation.Inclusion of CD80 in HSV targets the recombinant virus to PD-L1 on DCs and allows productive infection and robust immune responses.Coregulatory interactions among CD8α dendritic cells, the latency-associated transcript, and programmed death 1 contribute to higher levels of herpes simplex virus 1 latency.Binding of HSV-1 glycoprotein K (gK) to signal peptide peptidase (SPP) is required for virus infectivityThe role of LAT in increased CD8+ T cell exhaustion in trigeminal ganglia of mice latently infected with herpes simplex virus 1.An M2 Rather than a TH2 Response Contributes to Better Protection against Latency Reactivation following Ocular Infection of Naive Mice with a Recombinant Herpes Simplex Virus 1 Expressing Murine Interleukin-4.
P2860
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P2860
Risk factors for herpes simplex virus epithelial keratitis recurring during treatment of stromal keratitis or iridocyclitis. Herpetic Eye Disease Study Group.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年学术文章
@wuu
1996年学术文章
@zh-cn
1996年学术文章
@zh-hans
1996年学术文章
@zh-my
1996年学术文章
@zh-sg
1996年學術文章
@yue
1996年學術文章
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1996年學術文章
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name
Risk factors for herpes simple ...... petic Eye Disease Study Group.
@en
Risk factors for herpes simple ...... petic Eye Disease Study Group.
@nl
type
label
Risk factors for herpes simple ...... petic Eye Disease Study Group.
@en
Risk factors for herpes simple ...... petic Eye Disease Study Group.
@nl
prefLabel
Risk factors for herpes simple ...... petic Eye Disease Study Group.
@en
Risk factors for herpes simple ...... petic Eye Disease Study Group.
@nl
P2093
P2860
P356
P1476
Risk factors for herpes simple ...... petic Eye Disease Study Group.
@en
P2093
Bacchetti P
Hyndiuk RA
Kaufman HE
Laibson PR
Stulting RD
P2860
P304
P356
10.1136/BJO.80.11.969
P407
P577
1996-11-01T00:00:00Z