Transgenic mice expressing soluble tumor necrosis factor-receptor are protected against bone loss caused by estrogen deficiency.
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Pathogenesis of osteoporosis: concepts, conflicts, and prospectsOsteoimmunology and the influence of pro-inflammatory cytokines on osteoclastsChildhood obesity, bone development, and cardiometabolic risk factorsImmune regulation of osteoclast function in postmenopausal osteoporosis: a critical interdisciplinary perspectiveEnhanced trabecular-bone calcium deposition in female rats with a high physiological dose of prolactin diminishes after ovariectomyEstrogen decreases osteoclast formation by down-regulating receptor activator of NF-kappa B ligand (RANKL)-induced JNK activation.Postneonatal Mortality and Liver Changes in Cloned Pigs Associated with Human Tumor Necrosis Factor Receptor I-Fc and Human Heme Oxygenase-1 Overexpression.Bone phenotypes in response to gonadotropin misexpression: the role for gonadotropins in postmenopausal osteoporosisEstrogen's bone-protective effects may involve differential IL-1 receptor regulation in human osteoclast-like cells.Tumor necrosis factor alpha stimulates osteoclast differentiation by a mechanism independent of the ODF/RANKL-RANK interactionUp-regulation of TNF-producing T cells in the bone marrow: a key mechanism by which estrogen deficiency induces bone loss in vivo.Physiology of bone loss.The effect of calcium and vitamin D supplementation on osteoporotic rabbit bones studied by vibrational spectroscopy.T cells: critical bone regulators in health and disease.Female reproductive system and bone.The immune system and bone.Bone mineral density in rheumatoid arthritis patients 1 year after adalimumab therapy: arrest of bone loss.Ovariectomy disregulates osteoblast and osteoclast formation through the T-cell receptor CD40 ligand.Protein intake and bone growth.Estrogen deficiency and bone loss: an inflammatory tale.Soluble cytokine receptors in biological therapy.Bone strength and its determinants.Effects of obesity on bone metabolism.M-CSF neutralization and egr-1 deficiency prevent ovariectomy-induced bone loss.A review on current osteoporosis research: with special focus on disuse bone loss.Absence of platelet-activating factor receptor protects mice from osteoporosis following ovariectomy.The multiple facets of premature aging in rheumatoid arthritis.A Bone Anabolic Effect of RANKL in a Murine Model of Osteoporosis Mediated Through FoxP3+ CD8 T Cells.T cells and post menopausal osteoporosis in murine modelsOxidative stress causes bone loss in estrogen-deficient mice through enhanced bone marrow dendritic cell activationEstrogen and the skeletonReceptor activator of nuclear factor κB ligand (RANKL) protein expression by B lymphocytes contributes to ovariectomy-induced bone lossA 1-year case-control study in patients with rheumatoid arthritis indicates prevention of loss of bone mineral density in both responders and nonresponders to infliximab.Osteopontin-deficient mice are resistant to ovariectomy-induced bone resorption.Bone remodeling in post-menopausal osteoporosis.Activated NF-κB in bone marrow mesenchymal stem cells from systemic lupus erythematosus patients inhibits osteogenic differentiation through downregulating Smad signaling.Disruption of claudin-18 diminishes ovariectomy-induced bone loss in miceEstrogen decreases TNF gene expression by blocking JNK activity and the resulting production of c-Jun and JunD.Estradiol repression of tumor necrosis factor-alpha transcription requires estrogen receptor activation function-2 and is enhanced by coactivators.IL-17 mediates estrogen-deficient osteoporosis in an Act1-dependent manner.
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Transgenic mice expressing soluble tumor necrosis factor-receptor are protected against bone loss caused by estrogen deficiency.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on April 1997
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Transgenic mice expressing sol ...... caused by estrogen deficiency.
@en
Transgenic mice expressing sol ...... caused by estrogen deficiency.
@nl
type
label
Transgenic mice expressing sol ...... caused by estrogen deficiency.
@en
Transgenic mice expressing sol ...... caused by estrogen deficiency.
@nl
prefLabel
Transgenic mice expressing sol ...... caused by estrogen deficiency.
@en
Transgenic mice expressing sol ...... caused by estrogen deficiency.
@nl
P2093
P2860
P356
P1476
Transgenic mice expressing sol ...... caused by estrogen deficiency.
@en
P2093
Bonjour JP
Vassalli P
P2860
P304
P356
10.1172/JCI119333
P407
P577
1997-04-01T00:00:00Z