p53 and the hypoxia-induced apoptosis of cultured neonatal rat cardiac myocytes.
about
miR-30 regulates mitochondrial fission through targeting p53 and the dynamin-related protein-1 pathwayApoptosis in myocardial ischaemia and infarctionBRCA1 is an essential regulator of heart function and survival following myocardial infarctionHypoxic stress suppresses RNA polymerase III recruitment and tRNA gene transcription in cardiomyocytes.Mitochondria and mitophagy: the yin and yang of cell death controlA Small Molecule Binding to the Coactivator CREB-Binding Protein Blocks Apoptosis in CardiomyocytesTranscription factor Foxo3a prevents apoptosis by regulating calcium through the apoptosis repressor with caspase recruitment domainNucleolin/C23 mediates the antiapoptotic effect of heat shock protein 70 during oxidative stressp53 initiates apoptosis by transcriptionally targeting the antiapoptotic protein ARCInvolvement of GADD153 and cardiac ankyrin repeat protein in hypoxia-induced apoptosis of H9c2 cellsInvolvement of GADD153 and cardiac ankyrin repeat protein in cardiac ischemia-reperfusion injuryp53 and TIGAR regulate cardiac myocyte energy homeostasis under hypoxic stress.Glucose uptake and glycolysis reduce hypoxia-induced apoptosis in cultured neonatal rat cardiac myocytes.Modulation of ceramide content and lack of apoptosis in the chronically hypoxic neonatal rat heart.Heat shock pretreatment inhibited the release of Smac/DIABLO from mitochondria and apoptosis induced by hydrogen peroxide in cardiomyocytes and C2C12 myogenic cells.Mitofusin 1 is negatively regulated by microRNA 140 in cardiomyocyte apoptosis.Protein Kinase Signaling at the Crossroads of Myocyte Life and Death in Ischemic Heart Disease.Hypoxia-activated apoptosis of cardiac myocytes requires reoxygenation or a pH shift and is independent of p53.Myocardial knockdown of mRNA-stabilizing protein HuR attenuates post-MI inflammatory response and left ventricular dysfunction in IL-10-null mice.Lysine methyltransferase Smyd2 suppresses p53-dependent cardiomyocyte apoptosis.Hypoxia, angiotensin-II, and norepinephrine mediated apoptosis is stimulus specific in canine failed cardiomyocytes: a role for p38 MAPK, Fas-L and cyclin D1.Nogo-A knockdown inhibits hypoxia/reoxygenation-induced activation of mitochondrial-dependent apoptosis in cardiomyocytes.E2F1 suppresses cardiac neovascularization by down-regulating VEGF and PlGF expression.Senescent cardiac fibroblast is critical for cardiac fibrosis after myocardial infarction.Protective effects of non-mitogenic human acidic fibroblast growth factor on hydrogen peroxide-induced damage to cardiomyocytes in vitroReciprocal influence of the p53 and the hypoxic pathwaysNanocurcumin Prevents Hypoxia Induced Stress in Primary Human Ventricular Cardiomyocytes by Maintaining Mitochondrial Homeostasis.Inhibition of p53 function prevents renin-angiotensin system activation and stretch-mediated myocyte apoptosisBRCA2 protein deficiency exaggerates doxorubicin-induced cardiomyocyte apoptosis and cardiac failurePrenatal hypoxia and cardiac programming.Vascular endothelial growth factor prevents apoptosis and preserves contractile function in hypertrophied infant heart.miR-23a binds to p53 and enhances its association with miR-128 promoter.It Takes 15 to Tango: Making Sense of the Many Ubiquitin Ligases of p53.Analysis of Tyrosine Kinase Inhibitor-Mediated Decline in Contractile Force in Rat Engineered Heart TissueFAT10 protects cardiac myocytes against apoptosis.Cardiac Stem Cell Secretome Protects Cardiomyocytes from Hypoxic Injury Partly via Monocyte Chemotactic Protein-1-Dependent Mechanism.A standardized extract of Ginkgo biloba suppresses doxorubicin-induced oxidative stress and p53-mediated mitochondrial apoptosis in rat testes.Selective inhibition of the master regulator transcription factor Egr-1 with catalytic oligonucleotides reduces myocardial injury and improves left ventricular systolic function in a preclinical model of myocardial infarctionThe role of Bcl-2 family member BNIP3 in cell death and disease: NIPping at the heels of cell death.Adenoviral delivery of E2F-1 directs cell cycle reentry and p53-independent apoptosis in postmitotic adult myocardium in vivo.
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p53 and the hypoxia-induced apoptosis of cultured neonatal rat cardiac myocytes.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on June 1997
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
p53 and the hypoxia-induced apoptosis of cultured neonatal rat cardiac myocytes.
@en
p53 and the hypoxia-induced apoptosis of cultured neonatal rat cardiac myocytes.
@nl
type
label
p53 and the hypoxia-induced apoptosis of cultured neonatal rat cardiac myocytes.
@en
p53 and the hypoxia-induced apoptosis of cultured neonatal rat cardiac myocytes.
@nl
prefLabel
p53 and the hypoxia-induced apoptosis of cultured neonatal rat cardiac myocytes.
@en
p53 and the hypoxia-induced apoptosis of cultured neonatal rat cardiac myocytes.
@nl
P2093
P2860
P356
P1476
p53 and the hypoxia-induced apoptosis of cultured neonatal rat cardiac myocytes.
@en
P2093
Cirielli C
Hipolito ML
Lakatta EG
Lundberg MS
P2860
P304
P356
10.1172/JCI119452
P407
P577
1997-06-01T00:00:00Z