Inflammasome-dependent caspase-1 activation in cervical epithelial cells stimulates growth of the intracellular pathogen Chlamydia trachomatis.
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Immune Homeostasis in Epithelial Cells: Evidence and Role of Inflammasome Signaling ReviewedInflammasome in intestinal inflammation and cancerInflammasomes and Their Role in Innate Immunity of Sexually Transmitted InfectionsDanger signals, inflammasomes, and the intricate intracellular lives of chlamydiaeNLRP3 inflammasome activation induced by engineered nanomaterialsThe role of inflammasome modulation in virulenceAssessing a potential role of host Pannexin 1 during Chlamydia trachomatis infectionThe anti-tumorigenic mushroom Agaricus blazei Murill enhances IL-1β production and activates the NLRP3 inflammasome in human macrophagesActivation of an NLRP3 inflammasome restricts Mycobacterium kansasii infectionTLR2 and Nod2 mediate resistance or susceptibility to fatal intracellular Ehrlichia infection in murine models of ehrlichiosis.Propofol's effects on phagocytosis, proliferation, nitrate production, and cytokine secretion in pressure-stimulated microglial cells.Aspergillus fumigatus stimulates the NLRP3 inflammasome through a pathway requiring ROS production and the Syk tyrosine kinase.Induction of interferon-stimulated genes by Chlamydia pneumoniae in fibroblasts is mediated by intracellular nucleotide-sensing receptors.Understanding the mechanisms of human tubal ectopic pregnancies: new evidence from knockout mouse modelsCD200Fc reduces LPS-induced IL-1β activation in human cervical cancer cells by modulating TLR4-NF-κB and NLRP3 inflammasome pathway.Alarmins, inflammasomes and immunity.Chlamydial infection of monocytes stimulates IL-1beta secretion through activation of the NLRP3 inflammasomeEndothelial NLRP3 inflammasome activation and arterial neointima formation associated with acid sphingomyelinase during hypercholesterolemiaCaspase-1 dependent IL-1β secretion is critical for host defense in a mouse model of Chlamydia pneumoniae lung infectionTargeting of a chlamydial protease impedes intracellular bacterial growthRac1 regulates the NLRP3 inflammasome which mediates IL-1beta production in Chlamydophila pneumoniae infected human mononuclear cells.Enhancement of reactive oxygen species production and chlamydial infection by the mitochondrial Nod-like family member NLRX1.TLR2/MyD88/NF-κB pathway, reactive oxygen species, potassium efflux activates NLRP3/ASC inflammasome during respiratory syncytial virus infectionPost-translational control of IL-1β via the human papillomavirus type 16 E6 oncoprotein: a novel mechanism of innate immune escape mediated by the E3-ubiquitin ligase E6-AP and p53.Significant role of IL-1 signaling, but limited role of inflammasome activation, in oviduct pathology during Chlamydia muridarum genital infectionThe Chlamydia protease CPAF regulates host and bacterial proteins to maintain pathogen vacuole integrity and promote virulenceInflammation and fibrosis during Chlamydia pneumoniae infection is regulated by IL-1 and the NLRP3/ASC inflammasome.Immunity and vaccines against sexually transmitted Chlamydia trachomatis infectionAggregatibacter actinomycetemcomitans Invasion Induces Interleukin-1β Production Through Reactive Oxygen Species and Cathepsin B.Guanylate binding proteins enable rapid activation of canonical and noncanonical inflammasomes in Chlamydia-infected macrophages.Nod1, but not the ASC inflammasome, contributes to induction of IL-1β secretion in human trophoblasts after sensing of Chlamydia trachomatisDetection of a microbial metabolite by STING regulates inflammasome activation in response to Chlamydia trachomatis infection.Chlamydial intracellular survival strategies.Critical role for interleukin-1beta (IL-1beta) during Chlamydia muridarum genital infection and bacterial replication-independent secretion of IL-1beta in mouse macrophages.Inflammasomes bridge signaling between pathogen identification and the immune response.A Protective Vaccine against Chlamydia Genital Infection Using Vault Nanoparticles without an Added Adjuvant.Sensing the enemy, containing the threat: cell-autonomous immunity to Chlamydia trachomatis.Chlamydial Plasmid-Dependent Pathogenicity.Genetic and molecular basis of inflammasome-mediated disease.Subversion of Cell-Autonomous Host Defense by Chlamydia Infection.
P2860
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P2860
Inflammasome-dependent caspase-1 activation in cervical epithelial cells stimulates growth of the intracellular pathogen Chlamydia trachomatis.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 31 July 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Inflammasome-dependent caspase ...... athogen Chlamydia trachomatis.
@en
Inflammasome-dependent caspase ...... athogen Chlamydia trachomatis.
@nl
type
label
Inflammasome-dependent caspase ...... athogen Chlamydia trachomatis.
@en
Inflammasome-dependent caspase ...... athogen Chlamydia trachomatis.
@nl
prefLabel
Inflammasome-dependent caspase ...... athogen Chlamydia trachomatis.
@en
Inflammasome-dependent caspase ...... athogen Chlamydia trachomatis.
@nl
P2093
P2860
P356
P1476
Inflammasome-dependent caspase ...... pathogen Chlamydia trachomatis
@en
P2093
Ali A Abdul-Sater
Evonne Koo
Georg Häcker
P2860
P304
26789-26796
P356
10.1074/JBC.M109.026823
P407
P577
2009-07-31T00:00:00Z