Mcl-1 mediates TWEAK/Fn14-induced non-small cell lung cancer survival and therapeutic response. - Wikidata - awgldk - TriplyDB

Mcl-1 mediates TWEAK/Fn14-induced non-small cell lung cancer survival and therapeutic response.

Mcl-1 mediates TWEAK/Fn14-induced non-small cell lung cancer survival and therapeutic response.

http://www.wikidata.org/entity/Q37707092

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Targeting MCL-1 sensitizes human esophageal squamous cell carcinoma cells to cisplatin-induced apoptosis Targeting mcl-1 for radiosensitization of pancreatic cancers.TWEAK activation of the non-canonical NF-κB signaling pathway differentially regulates melanoma and prostate cancer cell invasion.TWEAK/Fn14 signaling in tumors.The TWEAK Receptor Fn14 Is an Src-Inducible Protein and a Positive Regulator of Src-Driven Cell Invasion.Vorinostat and metformin sensitize EGFR-TKI resistant NSCLC cells via BIM-dependent apoptosis induction.Bcl-2 family in non-small cell lung cancer: its prognostic and therapeutic implications.TWEAK-Fn14 Influences Neurogenesis Status via Modulating NF-κB in Mice with Spinal Cord Injury.Src Promotes Metastasis of Human Non-Small Cell Lung Cancer Cells through Fn14-Mediated NF-κB Signaling.

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Mcl-1 mediates TWEAK/Fn14-induced non-small cell lung cancer survival and therapeutic response.

http://www.wikidata.org/entity/Q37707092

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 27 January 2014

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Mcl-1 mediates TWEAK/Fn14-indu ...... ival and therapeutic response.

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Mcl-1 mediates TWEAK/Fn14-indu ...... ival and therapeutic response.

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type

label

Mcl-1 mediates TWEAK/Fn14-indu ...... ival and therapeutic response.

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Mcl-1 mediates TWEAK/Fn14-indu ...... ival and therapeutic response.

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prefLabel

Mcl-1 mediates TWEAK/Fn14-indu ...... ival and therapeutic response.

@en

Mcl-1 mediates TWEAK/Fn14-indu ...... ival and therapeutic response.

@nl

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1541-7786.MCR-13-0458

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Molecular Cancer Research

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Mcl-1 mediates TWEAK/Fn14-indu ...... vival and therapeutic response

@en

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Chao Sima

http://www.w3.org/2001/XMLSchema#string

Glen J Weiss

http://www.w3.org/2001/XMLSchema#string

Ian T Mathews

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Janine LoBello

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Michael Bittner

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Michael H Cardone

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Nhan L Tran

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Timothy G Whitsett

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William E Pierceall

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P2860

Mcl-1 is critical for survival in a subgroup of non-small-cell lung cancer cell lines

Mechanisms of apoptosis sensitivity and resistance to the BH3 mimetic ABT-737 in acute myeloid leukemia

BH3 profiling identifies three distinct classes of apoptotic blocks to predict response to ABT-737 and conventional chemotherapeutic agents

MCL1, a gene expressed in programmed myeloid cell differentiation, has sequence similarity to BCL2

The human Fn14 receptor gene is up-regulated in migrating glioma cells in vitro and overexpressed in advanced glial tumors

Targeting the Bcl-2 family for cancer therapy

Targeting Mcl-1 for the therapy of cancer

Increased fibroblast growth factor-inducible 14 expression levels promote glioma cell invasion via Rac1 and nuclear factor-kappaB and correlate with poor patient outcome

The tumor necrosis factor-like weak inducer of apoptosis (TWEAK)-fibroblast growth factor-inducible 14 (Fn14) signaling system regulates glioma cell survival via NFkappaB pathway activation and BCL-XL/BCL-W expression

Oncogenic pathway signatures in human cancers as a guide to targeted therapies

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550-559

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10.1158/1541-7786.MCR-13-0458

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2014-01-27T00:00:00Z

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