about
A THEMIS:SHP1 complex promotes T-cell survivalThe lymphoid lineage-specific actin-uncapping protein Rltpr is essential for costimulation via CD28 and the development of regulatory T cellsNegative regulation of natural killer cell function by EAT-2, a SAP-related adaptorQuantitative proteomics analysis of signalosome dynamics in primary T cells identifies the surface receptor CD6 as a Lat adaptor-independent TCR signaling hubCo-recruitment analysis of the CBL and CBLB signalosomes in primary T cells identifies CD5 as a key regulator of TCR-induced ubiquitylationRevisiting the Timing of Action of the PAG Adaptor Using Quantitative Proteomics Analysis of Primary T CellsBinding of SAP SH2 domain to FynT SH3 domain reveals a novel mechanism of receptor signalling in immune regulation.The tumor necrosis factor alpha-induced protein 3 (TNFAIP3, A20) imposes a brake on antitumor activity of CD8 T cellsMolecular dissection of 2B4 signaling: implications for signal transduction by SLAM-related receptors.In the absence of its cytosolic domain, the CD28 molecule still contributes to T cell activation.CD6 modulates thymocyte selection and peripheral T cell homeostasisDual T cell- and B cell-intrinsic deficiency in humans with biallelic RLTPR mutations.The scaffolding function of the RLTPR protein explains its essential role for CD28 co-stimulation in mouse and human T cells.Lymphoproliferative disorders involving T helper effector cells with defective LAT signalosomes.EAT-2, a SAP-like adaptor, controls NK cell activation through phospholipase Cγ, Ca++, and Erk, leading to granule polarization.Precise Temporal Profiling of Signaling Complexes in Primary Cells Using SWATH Mass Spectrometry.LAT signaling pathology: an "autoimmune" condition without T cell self-reactivity.Computational modeling of the main signaling pathways involved in mast cell activation.Integrative biology of T cell activation.A hematopoietic cell-driven mechanism involving SLAMF6 receptor, SAP adaptors and SHP-1 phosphatase regulates NK cell education.Proteomic analysis of the SH2 domain-containing leukocyte protein of 76 kDa (SLP76) interactome in resting and activated primary mast cells [corrected].Loss of the LAT adaptor converts antigen-responsive T cells into pathogenic effectors that function independently of the T cell receptor.Proteomic Analysis of the SH2 Domain-containing Leukocyte Protein of 76 kDa (SLP76) Interactome in Resting and Activated Primary Mast Cells.The proline-rich sequence of CD3epsilon controls T cell antigen receptor expression on and signaling potency in preselection CD4+CD8+ thymocytes.Control of CD8 T cell proliferation and terminal differentiation by active STAT5 and CDKN2A/CDKN2B.EAT-2 : un nouveau régulateur de la fonction des cellules NKCD5, an Undercover Regulator of TCR SignalingFit αβ T-cell receptor suppresses leukemogenesis of Pten-deficient thymocytesThe costimulatory molecule CD226 signals through VAV1 to amplify TCR signals and promote IL-17 production by CD4+ T cellsQuantitative interactomics in primary T cells unveils TCR signal diversification extent and dynamicsQuantitative Interactomics in Primary T Cells Provides a Rationale for Concomitant PD-1 and BTLA Coinhibitor Blockade in Cancer Immunotherapy
P50
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P50
description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Romain Roncagalli
@ast
Romain Roncagalli
@en
Romain Roncagalli
@es
Romain Roncagalli
@nl
Romain Roncagalli
@sl
type
label
Romain Roncagalli
@ast
Romain Roncagalli
@en
Romain Roncagalli
@es
Romain Roncagalli
@nl
Romain Roncagalli
@sl
prefLabel
Romain Roncagalli
@ast
Romain Roncagalli
@en
Romain Roncagalli
@es
Romain Roncagalli
@nl
Romain Roncagalli
@sl
P106
P21
P31
P496
0000-0001-7554-0552