Abrogation of PIK3CA or PIK3R1 reduces proliferation, migration, and invasion in glioblastoma multiforme cells. - Wikidata - awgldk - TriplyDB

Abrogation of PIK3CA or PIK3R1 reduces proliferation, migration, and invasion in glioblastoma multiforme cells.

Abrogation of PIK3CA or PIK3R1 reduces proliferation, migration, and invasion in glioblastoma multiforme cells.

http://www.wikidata.org/entity/Q39446680

about

Computer-based identification of a novel LIMK1/2 inhibitor that synergizes with salirasib to destabilize the actin cytoskeleton Rapalogs in cancer prevention: anti-aging or anticancer?GSK-3 as potential target for therapeutic intervention in cancer Identifying new small molecule anti-invasive compounds for glioma treatment Advances in targeting signal transduction pathways Gene markers of cellular aging in human multipotent stromal cells in culture.Recent advances in the use of PI3K inhibitors for glioblastoma multiforme: current preclinical and clinical development Whole-exome sequencing identifies ATRX mutation as a key molecular determinant in lower-grade glioma.Selective anti-cancer agents as anti-aging drugs PIK3R1 underexpression is an independent prognostic marker in breast cancer.High-throughput chemical screens identify disulfiram as an inhibitor of human glioblastoma stem cells.SplicingCompass: differential splicing detection using RNA-seq data.Suppression of esophageal tumor growth and chemoresistance by directly targeting the PI3K/AKT pathway.Amphiregulin enhances alpha6beta1 integrin expression and cell motility in human chondrosarcoma cells through Ras/Raf/MEK/ERK/AP-1 pathway.Gene mutation profiling of primary glioblastoma through multiple tumor biopsy guided by 1H-magnetic resonance spectroscopy MicroRNA-542-3p Suppresses Tumor Cell Invasion via Targeting AKT Pathway in Human Astrocytoma.Identification of global alteration of translational regulation in glioma in vivo Ectopic NGAL expression can alter sensitivity of breast cancer cells to EGFR, Bcl-2, CaM-K inhibitors and the plant natural product berberine Alternatively spliced protein arginine methyltransferase 1 isoform PRMT1v2 promotes the survival and invasiveness of breast cancer cells.Answering the ultimate question "what is the proximal cause of aging?"Recent discoveries in the cycling, growing and aging of the p53 field Mechanistic or mammalian target of rapamycin (mTOR) may determine robustness in young male mice at the cost of accelerated aging.PI3K p110β isoform synergizes with JNK in the regulation of glioblastoma cell proliferation and migration through Akt and FAK inhibition.PIK3CA mutations in advanced cancers: characteristics and outcomes.Common drugs and treatments for cancer and age-related diseases: revitalizing answers to NCI's provocative questions.PTEN loss represses glioblastoma tumor initiating cell differentiation via inactivation of Lgl1.Mutation and genomic amplification of the PIK3CA proto-oncogene in pituitary adenomas.Major vault protein supports glioblastoma survival and migration by upregulating the EGFR/PI3K signalling axis Focal adhesion complex proteins in epidermis and squamous cell carcinoma.Expression profiling of cervical cancers in Indian women at different stages to identify gene signatures during progression of the disease.Neoplastic cells are a rare component in human glioblastoma microvasculature.Role of PRMTs in cancer: Could minor isoforms be leaving a mark?Targeting therapeutic liabilities engendered by PIK3R1 mutations for cancer treatment.Anticancer Activity of Punica granatum (Pomegranate): A Review.Knockdown of the AKT3 (PKBγ), PI3KCA, and VEGFR2 genes by RNA interference suppresses glioblastoma multiforme T98G cells invasiveness in vitro.LncRNA TSLC1-AS1 is a novel tumor suppressor in glioma.Knockdown of AKT3 (PKBγ) and PI3KCA suppresses cell viability and proliferation and induces the apoptosis of glioblastoma multiforme T98G cells.Medical Imaging Informatics.Similarity in targets with REST points to neural and glioblastoma related miRNAs FOXP3, a novel glioblastoma oncosuppressor, affects proliferation and migration.

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P2860

Abrogation of PIK3CA or PIK3R1 reduces proliferation, migration, and invasion in glioblastoma multiforme cells.

http://www.wikidata.org/entity/Q39446680

description

2011 nî lūn-bûn

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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2011年學術文章

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2011年學術文章

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name

Abrogation of PIK3CA or PIK3R1 ...... glioblastoma multiforme cells.

@en

Abrogation of PIK3CA or PIK3R1 ...... glioblastoma multiforme cells.

@nl

type

label

Abrogation of PIK3CA or PIK3R1 ...... glioblastoma multiforme cells.

@en

Abrogation of PIK3CA or PIK3R1 ...... glioblastoma multiforme cells.

@nl

prefLabel

Abrogation of PIK3CA or PIK3R1 ...... glioblastoma multiforme cells.

@en

Abrogation of PIK3CA or PIK3R1 ...... glioblastoma multiforme cells.

@nl

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Abrogation of PIK3CA or PIK3R1 ...... glioblastoma multiforme cells

@en

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Gary L Gallia

http://www.w3.org/2001/XMLSchema#string

Genevieve L Weber

http://www.w3.org/2001/XMLSchema#string

Gregory J Riggins

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Zev A Binder

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P2860

Glioblastoma angiogenesis and tumor cell invasiveness are differentially regulated by β8 integrin

An integrated genomic analysis of human glioblastoma multiforme

Mechanism of two classes of cancer mutations in the phosphoinositide 3-kinase catalytic subunit

Radiotherapy plus concomitant and adjuvant temozolomide for glioblastoma

High frequency of mutations of the PIK3CA gene in human cancers

FAK integrates growth-factor and integrin signals to promote cell migration

Regulation of the p85/p110 phosphatidylinositol 3'-kinase: stabilization and inhibition of the p110alpha catalytic subunit by the p85 regulatory subunit

Direct positive regulation of PTEN by the p85 subunit of phosphatidylinositol 3-kinase.

N-terminal domains of the class ia phosphoinositide 3-kinase regulatory subunit play a role in cytoskeletal but not mitogenic signaling.

Regulation of the p85/p110alpha phosphatidylinositol 3'-kinase. Distinct roles for the n-terminal and c-terminal SH2 domains.

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833-849

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scholarly article

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10.18632/ONCOTARGET.346

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11

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2

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Marie-Odile Parat

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3260001

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