Checkpoint adaptation precedes spontaneous and damage-induced genomic instability in yeast.
about
Cellular functions of human RPA1. Multiple roles of domains in replication, repair, and checkpointsInvolvement of nucleotide excision and mismatch repair mechanisms in double strand break repairPolo-like kinase 1, on the rise from cell cycle regulation to prostate cancer developmentRole of RAD52 epistasis group genes in homologous recombination and double-strand break repairTelomere uncapping in progenitor cells with critical telomere shortening is coupled to S-phase progression in vivoBubR1- and Polo-coated DNA tethers facilitate poleward segregation of acentric chromatids.Mre11-Rad50 promotes rapid repair of DNA damage in the polyploid archaeon Haloferax volcanii by restraining homologous recombinationElevated levels of the polo kinase Cdc5 override the Mec1/ATR checkpoint in budding yeast by acting at different steps of the signaling pathway.CDC5 inhibits the hyperphosphorylation of the checkpoint kinase Rad53, leading to checkpoint adaptationSgs1 and exo1 redundantly inhibit break-induced replication and de novo telomere addition at broken chromosome ends.RAD51-dependent break-induced replication differs in kinetics and checkpoint responses from RAD51-mediated gene conversionRegulatory functional territory of PLK-1 and their substrates beyond mitosis.Chk2 and p53 are haploinsufficient with dependent and independent functions to eliminate cells after telomere loss.Cycles of chromosome instability are associated with a fragile site and are increased by defects in DNA replication and checkpoint controls in yeast.Rescue from replication stress during mitosis.Spontaneous chromosome loss in Saccharomyces cerevisiae is suppressed by DNA damage checkpoint functionsA novel selection system for chromosome translocations in Saccharomyces cerevisiae.Positive and negative roles of homologous recombination in the maintenance of genome stability in Saccharomyces cerevisiae.Reciprocal translocations in Saccharomyces cerevisiae formed by nonhomologous end joining.Two checkpoint complexes are independently recruited to sites of DNA damage in vivo.The DNA damage response and checkpoint adaptation in Saccharomyces cerevisiae: distinct roles for the replication protein A2 (Rfa2) N-terminus.Chromosomes with delayed replication timing lead to checkpoint activation, delayed recruitment of Aurora B and chromosome instability.The chromokinesin Klp3a and microtubules facilitate acentric chromosome segregation.Mammalian chromosomes contain cis-acting elements that control replication timing, mitotic condensation, and stability of entire chromosomes.RAD51-dependent break-induced replication in yeast.Cell cycle regulation of DNA replication.A novel Tel1/ATM N-terminal motif, TAN, is essential for telomere length maintenance and a DNA damage response.Deacetylase Rpd3 facilitates checkpoint adaptation by preventing Rad53 overactivation.LTE1 promotes exit from mitosis by multiple mechanisms.Rad53 downregulates mitotic gene transcription by inhibiting the transcriptional activator Ndd1.Understanding the limitations of radiation-induced cell cycle checkpoints.When genome integrity and cell cycle decisions collide: roles of polo kinases in cellular adaptation to DNA damage.Reduced kinase activity of polo kinase Cdc5 affects chromosome stability and DNA damage response in S. cerevisiae.Genome reversion process of endopolyploidy confers chromosome instability on the descendent diploid cells.Involvement of RAD9-dependent damage checkpoint control in arrest of cell cycle, induction of cell death, and chromosome instability caused by defects in origin recognition complex in Saccharomyces cerevisiae.Protective bystander effects simulated with the state-vector model.Virulence and karyotype analyses of rad52 mutants of Candida albicans: regeneration of a truncated chromosome of a reintegrant strain (rad52/RAD52) in the host.Recovery from DNA damage-induced G2 arrest requires actin-binding protein filamin-A/actin-binding protein 280.Functionally distinct isoforms of Cik1 are differentially regulated by APC/C-mediated proteolysis.Large inverted repeats in the vicinity of a single double-strand break strongly affect repair in yeast diploids lacking Rad51.
P2860
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P2860
Checkpoint adaptation precedes spontaneous and damage-induced genomic instability in yeast.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
@zh
2001年论文
@zh-cn
name
Checkpoint adaptation precedes ...... genomic instability in yeast.
@en
Checkpoint adaptation precedes ...... genomic instability in yeast.
@nl
type
label
Checkpoint adaptation precedes ...... genomic instability in yeast.
@en
Checkpoint adaptation precedes ...... genomic instability in yeast.
@nl
prefLabel
Checkpoint adaptation precedes ...... genomic instability in yeast.
@en
Checkpoint adaptation precedes ...... genomic instability in yeast.
@nl
P2860
P1476
Checkpoint adaptation precedes ...... genomic instability in yeast.
@en
P2093
D J Galgoczy
D P Toczyski
P2860
P304
P356
10.1128/MCB.21.5.1710-1718.2001
P407
P577
2001-03-01T00:00:00Z