Non-homologous end joining as an important mutagenic process in cell cycle-arrested cells - Wikidata - awgldk - TriplyDB

Non-homologous end joining as an important mutagenic process in cell cycle-arrested cells

Non-homologous end joining as an important mutagenic process in cell cycle-arrested cells

http://www.wikidata.org/entity/Q39756406

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Making ends meet: repairing breaks in bacterial DNA by non-homologous end-joining Genome Engineering with TALE and CRISPR Systems in Neuroscience Advanced In vivo Use of CRISPR/Cas9 and Anti-sense DNA Inhibition for Gene Manipulation in the Brain Yeasts acquire resistance secondary to antifungal drug treatment by adaptive mutagenesis Mechanism of nonhomologous end-joining in mycobacteria: a low-fidelity repair system driven by Ku, ligase D and ligase C The yeast environmental stress response regulates mutagenesis induced by proteotoxic stress Cryptococcus vaughanmartiniae sp. nov. and Cryptococcus onofrii sp. nov.: two new species isolated from worldwide cold environments.Mutability and importance of a hypermutable cell subpopulation that produces stress-induced mutants in Escherichia coli.The impact of the nucleosome code on protein-coding sequence evolution in yeast Disruption of HPV16-E7 by CRISPR/Cas system induces apoptosis and growth inhibition in HPV16 positive human cervical cancer cells Genomic mutation rates: what high-throughput methods can tell us Roles of E. coli double-strand-break-repair proteins in stress-induced mutation Capture of extranuclear DNA at fission yeast double-strand breaks Adaptive mutation in Saccharomyces cerevisiae.Disruption of human papillomavirus 16 E6 gene by clustered regularly interspaced short palindromic repeat/Cas system in human cervical cancer cells.The influence of recombination on human genetic diversity.A genomic screen revealing the importance of vesicular trafficking pathways in genome maintenance and protection against genotoxic stress in diploid Saccharomyces cerevisiae cells Frameshift mutagenesis: the roles of primer-template misalignment and the nonhomologous end-joining pathway in Saccharomyces cerevisiae.Mutation as a stress response and the regulation of evolvability Longevity mutation in SCH9 prevents recombination errors and premature genomic instability in a Werner/Bloom model system.A simple model system for age-dependent DNA damage and cancer Impaired resection of meiotic double-strand breaks channels repair to nonhomologous end joining in Caenorhabditis elegans.Checkpoint kinase phosphorylation in response to endogenous oxidative DNA damage in repair-deficient stationary-phase Saccharomyces cerevisiae Oncogene homologue Sch9 promotes age-dependent mutations by a superoxide and Rev1/Polzeta-dependent mechanism.Mismatch tolerance by DNA polymerase Pol4 in the course of nonhomologous end joining in Saccharomyces cerevisiae.Non-homologous end joining induced alterations in DNA methylation: A source of permanent epigenetic change.DNA repair and mutations during quiescence in yeast.Studying age-dependent genomic instability using the S. cerevisiae chronological lifespan model.BCR/ABL stimulates WRN to promote survival and genomic instability.Assessing chronological aging in Saccharomyces cerevisiae.Mutagenicity of non-homologous end joining DNA repair in a resistant subset of human chronic lymphocytic leukaemia B cells.Adaptive point mutation and adaptive amplification pathways in the Escherichia coli Lac system: stress responses producing genetic change The role of DNA polymerase alpha in the control of mutagenesis in Saccharomyces cerevisiae cells starved for nutrients.RAD52 is required for RNA-templated recombination repair in post-mitotic neurons.microRNAs and Acute Myeloid Leukemia Chemoresistance: A Mechanistic Overview.Stress-induced cellular adaptive strategies: ancient evolutionarily conserved programs as new anticancer therapeutic targets.The frameshift infidelity of human DNA polymerase lambda. Implications for function.Mutagenic assessment of chemotherapy and Smac mimetic drugs in cells with defective DNA damage response pathways CRISPR-Cas immunity, DNA repair and genome stability

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Non-homologous end joining as an important mutagenic process in cell cycle-arrested cells

http://www.wikidata.org/entity/Q39756406

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年學術文章

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2003年學術文章

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name

Non-homologous end joining as an important mutagenic process in cell cycle-arrested cells

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Non-homologous end joining as ...... in cell cycle-arrested cells.

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Non-homologous end joining as an important mutagenic process in cell cycle-arrested cells

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Non-homologous end joining as ...... in cell cycle-arrested cells.

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Non-homologous end joining as an important mutagenic process in cell cycle-arrested cells

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Non-homologous end joining as ...... in cell cycle-arrested cells.

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The EMBO Journal

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Non-homologous end joining as an important mutagenic process in cell cycle-arrested cells

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Bernd Kneidinger

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Erich Heidenreich

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Rene Novotny

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Ulrike Wintersberger

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Veronika Holzmann

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P2860

Instability and decay of the primary structure of DNA

Mutations of Bacteria from Virus Sensitivity to Virus Resistance

Multiple pathways of recombination induced by double-strand breaks in Saccharomyces cerevisiae

A newly identified DNA ligase of Saccharomyces cerevisiae involved in RAD52-independent repair of DNA double-strand breaks

Adaptive mutation: implications for evolution

Cell cycle and genetic requirements of two pathways of nonhomologous end-joining repair of double-strand breaks in Saccharomyces cerevisiae

The nonhomologous end-joining pathway of DNA repair is required for genomic stability and the suppression of translocations

NEJ1 controls non-homologous end joining in Saccharomyces cerevisiae.

Nej1p, a cell type-specific regulator of nonhomologous end joining in yeast.

Yeast DNA ligase IV mediates non-homologous DNA end joining.

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2274-2283

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10.1093/EMBOJ/CDG203

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9

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22

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12727893

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156075

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