Cyclic AMP inhibits the proliferation of thyroid carcinoma cell lines through regulation of CDK4 phosphorylation.
about
CDK4 T172 phosphorylation is central in a CDK7-dependent bidirectional CDK4/CDK2 interplay mediated by p21 phosphorylation at the restriction pointcAMP-dependent activation of mammalian target of rapamycin (mTOR) in thyroid cells. Implication in mitogenesis and activation of CDK4.Mechanisms underlying dual effects of serotonin during development of Helisoma trivolvis (Mollusca)Proliferation and survival molecules implicated in the inhibition of BRAF pathway in thyroid cancer cells harbouring different genetic mutations.Switch in signaling control of mTORC1 activity after oncoprotein expression in thyroid cancer cell lines.mRNA expression in papillary and anaplastic thyroid carcinoma: molecular anatomy of a killing switchLow-power laser irradiation promotes the proliferation and osteogenic differentiation of human periodontal ligament cells via cyclic adenosine monophosphateThe CDK4/CDK6 inhibitor PD0332991 paradoxically stabilizes activated cyclin D3-CDK4/6 complexes.Cellular chloride and bicarbonate retention alters intracellular pH regulation in Cftr KO crypt epithelium.The CDK Network: Linking Cycles of Cell Division and Gene Expression.A Cdk7-Cdk4 T-loop phosphorylation cascade promotes G1 progressionDifferential regulation of cyclin-dependent kinase 4 (CDK4) and CDK6, evidence that CDK4 might not be activated by CDK7, and design of a CDK6 activating mutation.Increased mitogen-activated protein kinase kinase/extracellularly regulated kinase activity in human endometrial stromal fibroblasts of women with endometriosis reduces 3',5'-cyclic adenosine 5'-monophosphate inhibition of cyclin D1.Could radiotherapy effectiveness be enhanced by electromagnetic field treatment?Morphoproteomics demonstrates activation of mammalian target of rapamycin pathway in papillary thyroid carcinomas with nuclear translocation of MTOR in aggressive histological variants.cAMP inhibits mammalian target of rapamycin complex-1 and -2 (mTORC1 and 2) by promoting complex dissociation and inhibiting mTOR kinase activity.A genome-wide RNAi screen identifies novel targets of neratinib resistance leading to identification of potential drug resistant genetic markers.JNKs function as CDK4-activating kinases by phosphorylating CDK4 and p21.CDK4 phosphorylation status and a linked gene expression profile predict sensitivity to palbociclib.Commentary: Thyrotropin Stimulates Differentiation Not Proliferation of Normal Human Thyrocytes in Culture.Clenbuterol Induces Cell Cycle Arrest in C2C12 Myoblasts by Delaying p27 Degradation through β-arrestin 2 Signaling.
P2860
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P2860
Cyclic AMP inhibits the proliferation of thyroid carcinoma cell lines through regulation of CDK4 phosphorylation.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Cyclic AMP inhibits the prolif ...... ation of CDK4 phosphorylation.
@en
Cyclic AMP inhibits the prolif ...... ation of CDK4 phosphorylation.
@nl
type
label
Cyclic AMP inhibits the prolif ...... ation of CDK4 phosphorylation.
@en
Cyclic AMP inhibits the prolif ...... ation of CDK4 phosphorylation.
@nl
prefLabel
Cyclic AMP inhibits the prolif ...... ation of CDK4 phosphorylation.
@en
Cyclic AMP inhibits the prolif ...... ation of CDK4 phosphorylation.
@nl
P2093
P2860
P356
P1476
Cyclic AMP inhibits the prolif ...... ation of CDK4 phosphorylation.
@en
P2093
Ana Sofia Rocha
Jacques E Dumont
Katia Coulonval
Pierre P Roger
Sabine Paternot
P2860
P304
P356
10.1091/MBC.E08-06-0617
P50
P577
2008-09-17T00:00:00Z