Selective replication of E1B55K-deleted adenoviruses depends on enhanced E1A expression in cancer cells. - Wikidata - awgldk - TriplyDB

Selective replication of E1B55K-deleted adenoviruses depends on enhanced E1A expression in cancer cells.

Selective replication of E1B55K-deleted adenoviruses depends on enhanced E1A expression in cancer cells.

http://www.wikidata.org/entity/Q40341364

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Molecular basis for viral selective replication in cancer cells: activation of CDK2 by adenovirus-induced cyclin E Combinatory cytotoxic effects produced by E1B-55kDa-deleted adenoviruses and chemotherapeutic agents are dependent on the agents in esophageal carcinoma.Virotherapy targeting cyclin E overexpression in tumors with adenovirus-enhanced cancer-selective promoter.Oncolytic adenovirus targeting cyclin E overexpression repressed tumor growth in syngeneic immunocompetent mice.Etoposide enhances antitumor efficacy of MDR1-driven oncolytic adenovirus through autoupregulation of the MDR1 promoter activity.Combination of autophagy inducer rapamycin and oncolytic adenovirus improves antitumor effect in cancer cells.Adenovirus with DNA Packaging Gene Mutations Increased Virus Release.Adenovirus E1B55K region is required to enhance cyclin E expression for efficient viral DNA replication.Development of an Oncolytic Adenovirus with Enhanced Spread Ability through Repeated UV Irradiation and Cancer Selection

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Selective replication of E1B55K-deleted adenoviruses depends on enhanced E1A expression in cancer cells.

http://www.wikidata.org/entity/Q40341364

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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name

Selective replication of E1B55 ...... 1A expression in cancer cells.

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Selective replication of E1B55 ...... 1A expression in cancer cells.

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Selective replication of E1B55 ...... 1A expression in cancer cells.

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Cancer Gene Therapy

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Selective replication of E1B55 ...... E1A expression in cancer cells

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McMasters KM

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Rao XM

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Snodgrass CL

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Zhou HS

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P2860

Efficient adenoviral-mediated ornithine transcarbamylase expression in deficient mouse and human hepatocytes.

Dual cancer-specific targeting strategy cures primary and distant breast carcinomas in nude mice.

ONYX-015: mechanisms of action and clinical potential of a replication-selective adenovirus.

Adenovirus-5 E1A: paradox and paradigm.

Does the antitumor adenovirus ONYX-015/dl1520 selectively target cells defective in the p53 pathway?

p53 functions as a cell cycle control protein in osteosarcomas

Replication-selective virotherapy for cancer: Biological principles, risk management and future directions.

An adenovirus mutant that replicates selectively in p53-deficient human tumor cells.

Adenovirus proteins from both E1B reading frames are required for transformation of rodent cells by viral infection and DNA transfection.

Replication-selective viruses for cancer therapy.

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10.1038/SJ.CGT.7700923

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6

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1002494177

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