The jun and fos protein families are both required for cell cycle progression in fibroblasts.
about
RhoA: a therapeutic target for chronic myeloid leukemiaCritical role of c-Jun overexpression in liver metastasis of human breast cancer xenograft modelIdentification of a murine TEF-1-related gene expressed after mitogenic stimulation of quiescent fibroblasts and during myogenic differentiationBMK1/ERK5 regulates serum-induced early gene expression through transcription factor MEF2Cc-Jun regulates cell cycle progression and apoptosis by distinct mechanismsMultiple mitogen-activated protein kinase signaling pathways connect the cot oncoprotein to the c-jun promoter and to cellular transformationDominant negative c-jun inhibits activation of the cyclin D1 and cyclin E kinase complexesTargeted disruption of the c-fos gene demonstrates c-fos-dependent and -independent pathways for gene expression stimulated by growth factors or oncogenesControl of cell cycle progression by c-Jun is p53 dependentOncogenic transformation by ras and fos is mediated by c-Jun N-terminal phosphorylationMEKK2 associates with the adapter protein Lad/RIBP and regulates the MEK5-BMK1/ERK5 pathwayStrain differences in vaginal responses to the xenoestrogen bisphenol ACell fate determination factor DACH1 inhibits c-Jun-induced contact-independent growthCyclin A is a c-Jun target gene and is necessary for c-Jun-induced anchorage-independent growth in RAT1a cellsEffect of deoxyribozymes targeting c-Jun on solid tumor growth and angiogenesis in rodentsInduction of c-jun and TGF-beta 1 in Fischer 344 rats during amiodarone-induced pulmonary fibrosisThe Mos/MAP kinase pathway stabilizes c-Fos by phosphorylation and augments its transforming activity in NIH 3T3 cellsAP-1 in cell proliferation and survivalHuman SWI-SNF component BRG1 represses transcription of the c-fos geneGenome-wide analysis of the endothelial transcriptome under short-term chronic hypoxia.c-Fos overexpression increases the proliferation of human hepatocytes by stabilizing nuclear Cyclin D1Hepatocyte expression of serum response factor is essential for liver function, hepatocyte proliferation and survival, and postnatal body growth in miceEffects of ethanol on calcium homeostasis in the nervous system: implications for astrocytes.A network of mitogen-activated protein kinases links G protein-coupled receptors to the c-jun promoter: a role for c-Jun NH2-terminal kinase, p38s, and extracellular signal-regulated kinase 5.Cot, a novel kinase of histone H3, induces cellular transformation through up-regulation of c-fos transcriptional activity.Fos family members induce cell cycle entry by activating cyclin D1.Fra-1 induces morphological transformation and increases in vitro invasiveness and motility of epithelioid adenocarcinoma cells.Emerging roles of ATF2 and the dynamic AP1 network in cancer.Identification of c-fos-responsive elements downstream of TAR in the long terminal repeat of human immunodeficiency virus type-1.Proliferative signaling initiated in ACTH receptors.Adenovirus E1A negatively and positively modulates transcription of AP-1 dependent genes by dimer-specific regulation of the DNA binding and transactivation activities of Jun.Regulation of eicosanoid production and mitogenesis in rat intestinal epithelial cells by transforming growth factor-alpha and phorbol ester.The mammalian Jun proteins: redundancy and specificity.Epstein-Barr virus latent membrane protein 2A regulates c-Jun protein through extracellular signal-regulated kinase.Regulation of transcription factor activity during cellular aging.Integrin binding and cell spreading on extracellular matrix act at different points in the cell cycle to promote hepatocyte growth.Integrin alpha 5 beta 1 expression negatively regulates cell growth: reversal by attachment to fibronectinCell cycle-dependent variations in c-Jun and JunB phosphorylation: a role in the control of cyclin D1 expression.MEK kinase 2 and the adaptor protein Lad regulate extracellular signal-regulated kinase 5 activation by epidermal growth factor via SrcPropolis augments apoptosis induced by butyrate via targeting cell survival pathways.
P2860
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P2860
The jun and fos protein families are both required for cell cycle progression in fibroblasts.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
The jun and fos protein famili ...... le progression in fibroblasts.
@en
type
label
The jun and fos protein famili ...... le progression in fibroblasts.
@en
prefLabel
The jun and fos protein famili ...... le progression in fibroblasts.
@en
P2860
P356
P1476
The jun and fos protein famili ...... le progression in fibroblasts.
@en
P2093
P2860
P304
P356
10.1128/MCB.11.9.4466
P407
P577
1991-09-01T00:00:00Z