Decreased energy metabolism extends life span in Caenorhabditis elegans without reducing oxidative damage.
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A nuclear role for the respiratory enzyme CLK-1 in regulating mitochondrial stress responses and longevityHuman conditions of insulin-like growth factor-I (IGF-I) deficiencyA mitochondrial superoxide signal triggers increased longevity in Caenorhabditis elegansMitochondrial ROS and the Effectors of the Intrinsic Apoptotic Pathway in Aging Cells: The Discerning Killers!Xenohormetic and anti-aging activity of secoiridoid polyphenols present in extra virgin olive oil: a new family of gerosuppressant agentsBacteria, yeast, worms, and flies: exploiting simple model organisms to investigate human mitochondrial diseasesAntioxidant vitamins and mineral supplementation, life span expansion and cancer incidence: a critical commentary.Mitochondrial oxidative stress alters a pathway in Caenorhabditis elegans strongly resembling that of bile acid biosynthesis and secretion in vertebrates.Evaluation of environmental safety concentrations of DMSA Coated Fe2O3-NPs using different assay systems in nematode Caenorhabditis elegans.Regulation of life span by mitochondrial respiration: the HIF-1 and ROS connection.The evolving role of radiation therapy in the management of malignant melanomaMitochondrial and cytoplasmic ROS have opposing effects on lifespan.Future of radiation therapy for malignant melanoma in an era of newer, more effective biological agents.Hormesis does not make sense except in the light of TOR-driven aging.Molecular damage in cancer: an argument for mTOR-driven agingAdenylyl Cyclase Type 5 Deficiency Protects Against Diet-Induced Obesity and Insulin ResistanceSuperoxide dismutase is dispensable for normal animal lifespanCell cycle arrest is not yet senescence, which is not just cell cycle arrest: terminology for TOR-driven aging.Hypoxia and gerosuppression: the mTOR saga continuesCarbon Sources for Yeast Growth as a Precondition of Hydrogen Peroxide Induced Hormetic Phenotype.Fructose-Induced Carbonyl/Oxidative Stress in S. cerevisiae: Involvement of TOR.The SFT-1 and OXA-1 respiratory chain complex assembly factors influence lifespan by distinct mechanisms in C. elegans.Is metabolic rate a universal 'pacemaker' for biological processes?Mitochondrial responsibility in ageing process: innocent, suspect or guilty.The impact of mitochondrial oxidative stress on bile acid-like molecules in C. elegans provides a new perspective on human metabolic diseases.Long-term monitoring of Ca2+ dynamics in C. elegans pharynx: an in vivo energy balance sensor.A Caenorhabditis elegans Genome-Scale Metabolic Network Model.Dietary restriction decreases coenzyme Q and ubiquinol potentially via changes in gene expression in the model organism C. elegans.Aging causes decreased resistance to multiple stresses and a failure to activate specific stress response pathwaysLifespan Extension Induced by Caffeine in Caenorhabditis elegans is Partially Dependent on Adenosine Signaling.Different Mechanisms of Longevity in Long-Lived Mouse and Caenorhabditis elegans Mutants Revealed by Statistical Analysis of Mortality Rates.Specific microRNAs regulate heat stress responses in Caenorhabditis elegans.Effects of Long-Term Cultivation on Medium with Alpha-Ketoglutarate Supplementation on Metabolic Processes of Saccharomyces cerevisiae.Adaptive Evolution under Extreme Genetic Drift in Oxidatively Stressed Caenorhabditis elegans.Measuring Oxidative Stress in Caenorhabditis elegans: Paraquat and Juglone Sensitivity Assays.Breaking the Ceiling of Human Maximal Lifespan.Activation of DAF-16/FOXO by reactive oxygen species contributes to longevity in long-lived mitochondrial mutants in Caenorhabditis elegans.Quantification of in vivo oxidative damage in Caenorhabditis elegans during aging by endogenous F3-isoprostane measurement.The Good, the Bad, and the Ugly of ROS: New Insights on Aging and Aging-Related Diseases from Eukaryotic and Prokaryotic Model Organisms.Genetic inhibition of an ATP synthase subunit extends lifespan in C. elegans
P2860
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P2860
Decreased energy metabolism extends life span in Caenorhabditis elegans without reducing oxidative damage.
description
2010 nî lūn-bûn
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2010年の論文
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2010年学术文章
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2010年学术文章
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2010年学术文章
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2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
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name
Decreased energy metabolism ex ...... out reducing oxidative damage.
@en
type
label
Decreased energy metabolism ex ...... out reducing oxidative damage.
@en
prefLabel
Decreased energy metabolism ex ...... out reducing oxidative damage.
@en
P2093
P2860
P1433
P1476
Decreased energy metabolism ex ...... out reducing oxidative damage.
@en
P2093
Claire Bénard
Darius Camp
Jeremy Michael Van Raamsdonk
Siegfried Hekimi
P2860
P304
P356
10.1534/GENETICS.110.115378
P407
P577
2010-04-09T00:00:00Z