Mammalian target of rapamycin in spinal cord neurons mediates hypersensitivity induced by peripheral inflammation.
about
Ribosomal Protein S6 Phosphorylation in the Nervous System: From Regulation to FunctionAMPK: An emerging target for modification of injury-induced pain plasticity.mTOR, a new potential target for chronic pain and opioid-induced tolerance and hyperalgesiaTranslational control of nociception via 4E-binding protein 1Adenosine Monophosphate-activated Protein Kinase (AMPK) Activators For the Prevention, Treatment and Potential Reversal of Pathological Pain.In vivo gene knockdown in rat dorsal root ganglia mediated by self-complementary adeno-associated virus serotype 5 following intrathecal delivery.mTORC1 inhibition induces pain via IRS-1-dependent feedback activation of ERK.Spinal phosphinositide 3-kinase-Akt-mammalian target of rapamycin signaling cascades in inflammation-induced hyperalgesiamTOR kinase: a possible pharmacological target in the management of chronic pain.Spatial and cellular characterization of mTORC1 activation after spinal cord injury reveals biphasic increase mainly attributed to microglia/macrophages.Involvement of phosphatase and tensin homolog deleted from chromosome 10 in rodent model of neuropathic pain.Mechanisms and pharmacology of neuropathic pain in multiple sclerosis.Spinal cord NMDA receptor-mediated activation of mammalian target of rapamycin is required for the development and maintenance of bone cancer-induced pain hypersensitivities in rats.Activation of the Mammalian Target of Rapamycin in the Rostral Ventromedial Medulla Contributes to the Maintenance of Nerve Injury-Induced Neuropathic Pain in RatmTOR and its downstream pathway are activated in the dorsal root ganglion and spinal cord after peripheral inflammation, but not after nerve injuryNeuropathic pain in animal models of nervous system autoimmune diseases.Peripheral administration of translation inhibitors reverses increased hyperalgesia in a model of chronic pain in the rat.Ras and rheb signaling in survival and cell deathIntact subepidermal nerve fibers mediate mechanical hypersensitivity via the activation of protein kinase C gamma in spared nerve injury.Activation of mammalian target of rapamycin mediates rat pain-related responses induced by BmK I, a sodium channel-specific modulator.mTOR Inhibition: From Aging to Autism and Beyond.Inhibition of Mammalian Target of Rapamycin (mTOR) Signaling in the Insular Cortex Alleviates Neuropathic Pain after Peripheral Nerve Injury.Axonal protein synthesis and the regulation of primary afferent functionDrug repositioning: playing dirty to kill pain.Targeting adenosine monophosphate-activated protein kinase (AMPK) in preclinical models reveals a potential mechanism for the treatment of neuropathic pain.Localized experimental bone metastasis drives osteolysis and sensory hypersensitivity at distant non-tumor-bearing sites.Targeting AMPK for the Alleviation of Pathological Pain.Descending serotonergic facilitation mediated by spinal 5-HT3 receptors engages spinal rapamycin-sensitive pathways in the rat.Spinal protein kinase M ζ underlies the maintenance mechanism of persistent nociceptive sensitizationTranslational Control Mechanisms in Persistent Pain.Sinomenine attenuates chronic inflammatory pain in mice.Glucocorticoid mediates water avoidance stress-sensitized colon-bladder cross-talk via RSK2/PSD-95/NR2B in rats.Effects of intrathecal injection of rapamycin on pain threshold and spinal cord glial activation in rats with neuropathic pain.Rapamycin ameliorates neuropathic pain by activating autophagy and inhibiting interleukin-1β in the rat spinal cord.Diosgenin Glucoside Protects against Spinal Cord Injury by Regulating Autophagy and Alleviating ApoptosiseIF4E-Dependent Translational Control: A Central Mechanism for Regulation of Pain Plasticity
P2860
Q26770762-43606BED-7536-4942-8D6C-0ED248466752Q27021903-55A1E264-9949-4354-8F77-3C08C668B30EQ28081540-4579ED34-DED2-44E9-8B9B-F38611A9FB9BQ28551851-232C4045-212D-4DB1-B22E-BF1F5AB28A60Q30383901-39B68B56-0C31-4FEC-B00B-762EE5E87546Q34189539-7776AA19-E54D-40FE-A23C-15F319A3497CQ34340709-8C0E009E-289C-4A87-8D66-E40010DFBE9CQ34989480-95B8C0F9-24F9-4400-AA54-B3CB90E68F66Q35035474-6027B634-B9A3-41DB-B5A3-2AB1C6F13311Q35140800-249A9F5B-CAC7-4128-A69C-495330FD0E3EQ35276335-8FEB4901-E910-45AF-9DEE-2EBBBD100159Q35731415-BBC5FA34-C978-4F77-BED7-15460FA34654Q35874744-AB867318-1C57-4F33-BE9B-ECBE380EE4EBQ36386694-1EB080C6-C859-4C9A-A662-543B4B744178Q36843926-C22392EE-E814-4302-9405-A8EEA9F7C71AQ36869524-37ACB90F-4B3D-427D-91BD-E565B9D9C791Q36979846-6EEF4859-5FC5-4198-8388-2324CA1EEFFBQ37061781-317971BF-4F0D-47F8-8900-23E65693C9D0Q37115771-B031091F-5AA8-4CC8-9845-2FC54328804CQ37348245-A0118B4B-78AF-4F2A-B40F-E69F15284A57Q37385781-34FB13D5-D3E3-4AF0-ADB3-94B876427657Q37712584-098BD9DA-6850-4672-81DE-D7BF5A01FB51Q38148485-D4CAD60F-FD27-4706-B487-78008A772D5AQ38170322-0DCB369F-AC2D-4796-9B74-85DB097DCAB7Q38624930-F2EA22BB-715D-45CB-AC61-6BAC76AEBB45Q38849607-DA5961D2-1510-4E93-8D73-0B5D7947E524Q38998418-6523318E-C0D0-4D68-907C-42A89679194EQ42059691-A86F4D07-6188-4125-8406-02FE0A28BE80Q42729374-4928B948-046D-4E62-A35B-C7D9DD412AC8Q47220043-EA1F667E-BDA1-4579-AE4D-3611AB41D41BQ48536936-D7D77861-CBD6-4F9A-97E1-E81E3CA90BC7Q51298669-33679575-94C1-4FBB-AE73-8BF2054DD22AQ52941274-1CFE9AEF-4E73-4180-8854-8A2B63A9EE7DQ53675240-09DE3765-91C4-4C9F-9501-35A437E99375Q58802838-EFAD7707-23F7-4D1B-BA22-A7E55DB55CE2Q59136306-E28FFBD3-7786-497E-B47C-55CA2679368C
P2860
Mammalian target of rapamycin in spinal cord neurons mediates hypersensitivity induced by peripheral inflammation.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
Mammalian target of rapamycin ...... ed by peripheral inflammation.
@en
type
label
Mammalian target of rapamycin ...... ed by peripheral inflammation.
@en
prefLabel
Mammalian target of rapamycin ...... ed by peripheral inflammation.
@en
P2093
P2860
P1433
P1476
Mammalian target of rapamycin ...... ced by peripheral inflammation
@en
P2093
E Norsted Gregory
J A Gregory
J Steinauer
P2860
P304
P356
10.1016/J.NEUROSCIENCE.2010.05.067
P407
P577
2010-06-09T00:00:00Z