Cell-specific actions of HIV-Tat and morphine on opioid receptor expression in glia. - Wikidata - awgldk - TriplyDB

Cell-specific actions of HIV-Tat and morphine on opioid receptor expression in glia.

Cell-specific actions of HIV-Tat and morphine on opioid receptor expression in glia.

http://www.wikidata.org/entity/Q42127774

about

Opioids and Viral Infections: A Double-Edged Sword Interactions of Opioids and HIV Infection in the Pathogenesis of Chronic Pain Interaction between Tat and Drugs of Abuse during HIV-1 Infection and Central Nervous System Disease Opioid-induced central immune signaling: implications for opioid analgesia Morphine enhances microglial migration through modulation of P2X4 receptor signaling Mu-opioid receptor and delta-opioid receptor differentially regulate microglial inflammatory response to control proopiomelanocortin neuronal apoptosis in the hypothalamus: effects of neonatal alcohol.Regional heterogeneity and diversity in cytokine and chemokine production by astroglia: differential responses to HIV-1 Tat, gp120, and morphine revealed by multiplex analysis Spinal microglial motility is independent of neuronal activity and plasticity in adult mice.Delta-opioid receptor analgesia is independent of microglial activation in a rat model of neuropathic pain Inhibition of microglial P2X4 receptors attenuates morphine tolerance, Iba1, GFAP and mu opioid receptor protein expression while enhancing perivascular microglial ED2.Interactive comorbidity between opioid drug abuse and HIV-1 Tat: chronic exposure augments spine loss and sublethal dendritic pathology in striatal neurons Interactions of HIV and drugs of abuse: the importance of glia, neural progenitors, and host genetic factors.Exploring the neuroimmunopharmacology of opioids: an integrative review of mechanisms of central immune signaling and their implications for opioid analgesia.Sensitization of enteric neurons to morphine by HIV-1 Tat protein.Morphine potentiates neurodegenerative effects of HIV-1 Tat through actions at μ-opioid receptor-expressing glia.Chronic SIV and morphine treatment increases heat shock protein 5 expression at the synapse.Opiate addiction therapies and HIV-1 Tat: interactive effects on glial [Ca²⁺]i, oxyradical and neuroinflammatory chemokine production and correlative neurotoxicity.Saturated long-chain fatty acids activate inflammatory signaling in astrocytes.Morphine and gp120 toxic interactions in striatal neurons are dependent on HIV-1 strain.Morphine efficacy is altered in conditional HIV-1 Tat transgenic mice.Opiate drug use and the pathophysiology of neuroAIDS.Regulation and Functional Implications of Opioid Receptor Splicing in Opioid Pharmacology and HIV Pathogenesis Mechanism(s) involved in opioid drug abuse modulation of HAND Opioid drug abuse and modulation of immune function: consequences in the susceptibility to opportunistic infections.Effects of Morphine on Behavioral Task Performance in SIV-Infected Rhesus Macaques.Differential expression and HIV-1 regulation of μ-opioid receptor splice variants across human central nervous system cell types.Differential expression of the alternatively spliced OPRM1 isoform μ-opioid receptor-1K in HIV-infected individuals.Loss of μ opioid receptor signaling in nociceptors, but not microglia, abrogates morphine tolerance without disrupting analgesia.Viral and cellular factors underlying neuropathogenesis in HIV associated neurocognitive disorders (HAND)Functions of the chemokine receptor CXCR4 in the central nervous system and its regulation by μ-opioid receptors.HIV, Tat and dopamine transmission.Glial and Neuroimmune Mechanisms as Critical Modulators of Drug Use and Abuse.GSK3β-activation is a point of convergence for HIV-1 and opiate-mediated interactive neurotoxicity The protective effect of hydromorphone to ischemia in rat glial cells Morphine and HIV-Tat increase microglial-free radical production and oxidative stress: possible role in cytokine regulation.CCL5/RANTES gene deletion attenuates opioid-induced increases in glial CCL2/MCP-1 immunoreactivity and activation in HIV-1 Tat-exposed mice.Connexin-purinergic signaling in enteric glia mediates the prolonged effect of morphine on constipation.

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P2860

Cell-specific actions of HIV-Tat and morphine on opioid receptor expression in glia.

http://www.wikidata.org/entity/Q42127774

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Cell-specific actions of HIV-Tat and morphine on opioid receptor expression in glia.

@en

Cell-specific actions of HIV-Tat and morphine on opioid receptor expression in glia.

@nl

type

label

Cell-specific actions of HIV-Tat and morphine on opioid receptor expression in glia.

@en

Cell-specific actions of HIV-Tat and morphine on opioid receptor expression in glia.

@nl

prefLabel

Cell-specific actions of HIV-Tat and morphine on opioid receptor expression in glia.

@en

Cell-specific actions of HIV-Tat and morphine on opioid receptor expression in glia.

@nl

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Cell-specific actions of HIV-Tat and morphine on opioid receptor expression in glia

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Annadora J Bruce-Keller

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Filomena O Dimayuga

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Jadwiga Turchan-Cholewo

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Kurt F Hauser

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Pamela E Knapp

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Qunxing Ding

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P2860

HIV-1 Tat protein mimicry of chemokines

Estrogen protects against the synergistic toxicity by HIV proteins, methamphetamine and cocaine

Neuropathology and neurodegeneration in human immunodeficiency virus infection. Pathogenesis of HIV-induced lesions of the brain, correlations with HIV-associated disorders and modifications according to treatments

The Tat protein of HIV-1 induces tumor necrosis factor-alpha production. Implications for HIV-1-associated neurological diseases

HIV-1 Tat protein exits from cells via a leaderless secretory pathway and binds to extracellular matrix-associated heparan sulfate proteoglycans through its basic region

Human immunodeficiency virus type 1 tat activates non-N-methyl-D-aspartate excitatory amino acid receptors and causes neurotoxicity

Morphine promotes the growth of HIV-1 in human peripheral blood mononuclear cell cocultures

Expression of the mu opioid receptor in the human immunodeficiency virus type 1 transgenic rat model

Mu-opioid receptor desensitization by beta-arrestin-2 determines morphine tolerance but not dependence

Alteration of intracellular traffic by monensin; mechanism, specificity and relationship to toxicity

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