Contribution of insulin and Akt1 signaling to endothelial nitric oxide synthase in the regulation of endothelial function and blood pressure - Wikidata - awgldk - TriplyDB

Contribution of insulin and Akt1 signaling to endothelial nitric oxide synthase in the regulation of endothelial function and blood pressure

Contribution of insulin and Akt1 signaling to endothelial nitric oxide synthase in the regulation of endothelial function and blood pressure

http://www.wikidata.org/entity/Q42132463

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Insulin resistance, hyperglycemia, and atherosclerosis Stability and function of adult vasculature is sustained by Akt/Jagged1 signalling axis in endothelium.Subcellular and cellular locations of nitric oxide synthase isoforms as determinants of health and disease Na+-sensitive elevation in blood pressure is ENaC independent in diet-induced obesity and insulin resistance Ceacam1 deletion causes vascular alterations in large vessels Serine phosphorylation sites on IRS2 activated by angiotensin II and protein kinase C to induce selective insulin resistance in endothelial cells Regulation of obesity and insulin resistance by nitric oxide Insulin Signaling and Heart Failure.Modification of high saturated fat diet with n-3 polyunsaturated fat improves glucose intolerance and vascular dysfunction Angiotensin II stimulates thick ascending limb NO production via AT(2) receptors and Akt1-dependent nitric-oxide synthase 3 (NOS3) activation Mouse models of the metabolic syndrome.Physical activity maintains aortic endothelium-dependent relaxation in the obese type 2 diabetic OLETF rat Partial deletion of eNOS gene causes hyperinsulinemic state, unbalance of cardiac insulin signaling pathways and coronary dysfunction independently of high fat diet.Serum from calorie-restricted rats activates vascular cell eNOS through enhanced insulin signaling mediated by adiponectin.Regulation of nitric oxide production in health and disease.Metabolomic analyses for atherosclerosis, diabetes, and obesity.Lipotoxicity contributes to endothelial dysfunction: a focus on the contribution from ceramide.Loss of insulin signaling in vascular endothelial cells accelerates atherosclerosis in apolipoprotein E null mice Knockout of insulin receptors in cardiomyocytes attenuates coronary arterial dysfunction induced by pressure overload Role of Endothelial Cells in Myocardial Ischemia-Reperfusion Injury.Urokinase-type plasminogen activator (uPA) induces pulmonary microvascular endothelial permeability through low density lipoprotein receptor-related protein (LRP)-dependent activation of endothelial nitric-oxide synthase.FM19G11 reverses endothelial dysfunction in rat and human arteries through stimulation of the PI3K/Akt/eNOS pathway, independently of mTOR/HIF-1α activation.Antiobesogenic role of endothelial nitric oxide synthase.Fasting-induced reductions in cardiovascular and metabolic variables occur sooner in obese versus lean mice.Ceramide mediates vascular dysfunction in diet-induced obesity by PP2A-mediated dephosphorylation of the eNOS-Akt complex Ceramide-Initiated Protein Phosphatase 2A Activation Contributes to Arterial Dysfunction In Vivo.Ceramide synthase 5 mediates lipid-induced autophagy and hypertrophy in cardiomyocytes.Mechanisms of lipotoxicity in the cardiovascular system.MicroRNA-181b Improves Glucose Homeostasis and Insulin Sensitivity by Regulating Endothelial Function in White Adipose Tissue.Impact of age on the vasodilatory function of human skeletal muscle feed arteries SIRT3 Deficiency Induces Endothelial Insulin Resistance and Blunts Endothelial-Dependent Vasorelaxation in Mice and Human with Obesity.Mechanisms and consequences of endothelial nitric oxide synthase dysfunction in hypertension Insulin resistance: metabolic mechanisms and consequences in the heart.Opportunity "nox": a novel approach to preventing endothelial dysfunction in the context of insulin resistance.Sphingolipid metabolism and obesity-induced inflammation.Exercise training improves vascular mitochondrial function.A low-carbohydrate/high-fat diet reduces blood pressure in spontaneously hypertensive rats without deleterious changes in insulin resistance.Hypertension and decreased aortic compliance due to reduced elastin amounts do not increase atherosclerotic plaque accumulation in Ldlr-/- mice.Arterial insulin resistance in Yucatan micropigs with diet-induced obesity and metabolic syndrome.Clinical Update: Cardiovascular Disease in Diabetes Mellitus: Atherosclerotic Cardiovascular Disease and Heart Failure in Type 2 Diabetes Mellitus - Mechanisms, Management, and Clinical Considerations

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Contribution of insulin and Akt1 signaling to endothelial nitric oxide synthase in the regulation of endothelial function and blood pressure

http://www.wikidata.org/entity/Q42132463

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2009 nî lūn-bûn

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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2009年论文

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name

Contribution of insulin and Ak ...... al function and blood pressure

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Contribution of insulin and Ak ...... al function and blood pressure

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Contribution of insulin and Ak ...... al function and blood pressure

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Contribution of insulin and Ak ...... al function and blood pressure

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Contribution of insulin and Ak ...... al function and blood pressure

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Contribution of insulin and Ak ...... al function and blood pressure

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CIRCRESAHA.108.189316

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Circulation Research

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Contribution of insulin and Ak ...... al function and blood pressure

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Christian Riehle

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Deborah Jones

http://www.w3.org/2001/XMLSchema#string

Derrick Gale

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Elaine Hillas

http://www.w3.org/2001/XMLSchema#string

J David Symons

http://www.w3.org/2001/XMLSchema#string

Jason Tanner

http://www.w3.org/2001/XMLSchema#string

Lloyd J Wilson

http://www.w3.org/2001/XMLSchema#string

Milda Palionyte

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Robert C Cooksey

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Shawna L McMillin

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P2860

Regulation of endothelium-derived nitric oxide production by the protein kinase Akt

Activation of nitric oxide synthase in endothelial cells by Akt-dependent phosphorylation

Growth retardation and increased apoptosis in mice with homozygous disruption of the Akt1 gene

Characterization of selective resistance to insulin signaling in the vasculature of obese Zucker (fa/fa) rats.

Insulin resistance reduces arterial prostacyclin synthase and eNOS activities by increasing endothelial fatty acid oxidation.

Post-translational control of endothelial nitric oxide synthase: why isn't calcium/calmodulin enough?

The role of endothelial insulin signaling in the regulation of vascular tone and insulin resistance.

Pathophysiological implications of insulin resistance on vascular endothelial function.

eNOS at a glance.

Transgenic rescue of insulin receptor-deficient mice

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1085-1094

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scholarly article

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10.1161/CIRCRESAHA.108.189316

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9

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104

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Morris J. Birnbaum

Quan-Jiang Zhang

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2009-04-02T00:00:00Z

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19342603

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2936913

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